nb100 Search Results


anti p35  (Novus Biologicals)
90
Novus Biologicals anti p35
Anti P35, supplied by Novus Biologicals, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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nb100  (Novus Biologicals)
92
Novus Biologicals nb100

Nb100, supplied by Novus Biologicals, used in various techniques. Bioz Stars score: 92/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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alexa fluor 647 conjugated troponin i type 3 antibodies  (Novus Biologicals)
93
Novus Biologicals alexa fluor 647 conjugated troponin i type 3 antibodies

Alexa Fluor 647 Conjugated Troponin I Type 3 Antibodies, supplied by Novus Biologicals, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 93 stars, based on 1 article reviews
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nod2 conjugated with pe  (Novus Biologicals)
93
Novus Biologicals nod2 conjugated with pe
Enhanced SaLTA-induced NO production through MDP is mediated by TLR2 and CD14/MyD88-, and <t>NOD2-dependent</t> pathway. (A) RAW 264.7 cells (500 μl of 5 × 10 5 cells/ml) were treated with SaLTA (0 or 3 μg/ml) and/or MDP (0 or 10 μg/ml) for 24 h. The cells were then stained with anti-mouse TLR2 <t>conjugated</t> with PE or CD14 conjugated with FITC antibodies. For intracellular NOD2, the cells were permeabilized and stained with anti-mouse NOD2 conjugated with PE. The stained cells were then subjected to flow cytometry. Upper , a representative TLR2, NOD2, and CD14 expression determined by flow cytometric analysis. Values given in each histogram indicate mean fluorescence intensity (MFI) of 10,000 events of live cells in the treatment group. Lower , the expression of TLR2, NOD2, and CD14 is presented as mean of MFI ± S.D. of three replicates for each group. Asterisk (*) indicates a significant difference between the indicated treatment groups at P < 0.05. NT, non-treatment group; IC, isotype control. (B, C) BMMs (200 μl of 5 × 10 5 cells/ml) from wild-type, (B) TLR2-, MyD88- CD14- or (C) <t>NOD2-deficient</t> mice were treated with SaLTA (0 or 3 μg/ml) and/or MDP (0 or 10 μg/ml) for 24 h. Then, nitrite levels in the supernatants were measured. The data represent mean ± S.D. of three replicates for each group. * indicates a significant difference compared to the control group at P < 0.05.
Nod2 Conjugated With Pe, supplied by Novus Biologicals, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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anti vegf biotin  (R&D Systems)
95
R&D Systems anti vegf biotin
Enhanced SaLTA-induced NO production through MDP is mediated by TLR2 and CD14/MyD88-, and <t>NOD2-dependent</t> pathway. (A) RAW 264.7 cells (500 μl of 5 × 10 5 cells/ml) were treated with SaLTA (0 or 3 μg/ml) and/or MDP (0 or 10 μg/ml) for 24 h. The cells were then stained with anti-mouse TLR2 <t>conjugated</t> with PE or CD14 conjugated with FITC antibodies. For intracellular NOD2, the cells were permeabilized and stained with anti-mouse NOD2 conjugated with PE. The stained cells were then subjected to flow cytometry. Upper , a representative TLR2, NOD2, and CD14 expression determined by flow cytometric analysis. Values given in each histogram indicate mean fluorescence intensity (MFI) of 10,000 events of live cells in the treatment group. Lower , the expression of TLR2, NOD2, and CD14 is presented as mean of MFI ± S.D. of three replicates for each group. Asterisk (*) indicates a significant difference between the indicated treatment groups at P < 0.05. NT, non-treatment group; IC, isotype control. (B, C) BMMs (200 μl of 5 × 10 5 cells/ml) from wild-type, (B) TLR2-, MyD88- CD14- or (C) <t>NOD2-deficient</t> mice were treated with SaLTA (0 or 3 μg/ml) and/or MDP (0 or 10 μg/ml) for 24 h. Then, nitrite levels in the supernatants were measured. The data represent mean ± S.D. of three replicates for each group. * indicates a significant difference compared to the control group at P < 0.05.
Anti Vegf Biotin, supplied by R&D Systems, used in various techniques. Bioz Stars score: 95/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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anti hif1α  (Novus Biologicals)
90
Novus Biologicals anti hif1α
Enhanced SaLTA-induced NO production through MDP is mediated by TLR2 and CD14/MyD88-, and <t>NOD2-dependent</t> pathway. (A) RAW 264.7 cells (500 μl of 5 × 10 5 cells/ml) were treated with SaLTA (0 or 3 μg/ml) and/or MDP (0 or 10 μg/ml) for 24 h. The cells were then stained with anti-mouse TLR2 <t>conjugated</t> with PE or CD14 conjugated with FITC antibodies. For intracellular NOD2, the cells were permeabilized and stained with anti-mouse NOD2 conjugated with PE. The stained cells were then subjected to flow cytometry. Upper , a representative TLR2, NOD2, and CD14 expression determined by flow cytometric analysis. Values given in each histogram indicate mean fluorescence intensity (MFI) of 10,000 events of live cells in the treatment group. Lower , the expression of TLR2, NOD2, and CD14 is presented as mean of MFI ± S.D. of three replicates for each group. Asterisk (*) indicates a significant difference between the indicated treatment groups at P < 0.05. NT, non-treatment group; IC, isotype control. (B, C) BMMs (200 μl of 5 × 10 5 cells/ml) from wild-type, (B) TLR2-, MyD88- CD14- or (C) <t>NOD2-deficient</t> mice were treated with SaLTA (0 or 3 μg/ml) and/or MDP (0 or 10 μg/ml) for 24 h. Then, nitrite levels in the supernatants were measured. The data represent mean ± S.D. of three replicates for each group. * indicates a significant difference compared to the control group at P < 0.05.
Anti Hif1α, supplied by Novus Biologicals, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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anti hif1α - by Bioz Stars, 2026-06
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alexa fluor 647 conjugated cd14  (Novus Biologicals)
90
Novus Biologicals alexa fluor 647 conjugated cd14
Enhanced SaLTA-induced NO production through MDP is mediated by TLR2 and CD14/MyD88-, and <t>NOD2-dependent</t> pathway. (A) RAW 264.7 cells (500 μl of 5 × 10 5 cells/ml) were treated with SaLTA (0 or 3 μg/ml) and/or MDP (0 or 10 μg/ml) for 24 h. The cells were then stained with anti-mouse TLR2 <t>conjugated</t> with PE or CD14 conjugated with FITC antibodies. For intracellular NOD2, the cells were permeabilized and stained with anti-mouse NOD2 conjugated with PE. The stained cells were then subjected to flow cytometry. Upper , a representative TLR2, NOD2, and CD14 expression determined by flow cytometric analysis. Values given in each histogram indicate mean fluorescence intensity (MFI) of 10,000 events of live cells in the treatment group. Lower , the expression of TLR2, NOD2, and CD14 is presented as mean of MFI ± S.D. of three replicates for each group. Asterisk (*) indicates a significant difference between the indicated treatment groups at P < 0.05. NT, non-treatment group; IC, isotype control. (B, C) BMMs (200 μl of 5 × 10 5 cells/ml) from wild-type, (B) TLR2-, MyD88- CD14- or (C) <t>NOD2-deficient</t> mice were treated with SaLTA (0 or 3 μg/ml) and/or MDP (0 or 10 μg/ml) for 24 h. Then, nitrite levels in the supernatants were measured. The data represent mean ± S.D. of three replicates for each group. * indicates a significant difference compared to the control group at P < 0.05.
Alexa Fluor 647 Conjugated Cd14, supplied by Novus Biologicals, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 90 stars, based on 1 article reviews
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caspaseiii  (Novus Biologicals)
95
Novus Biologicals caspaseiii
Enhanced SaLTA-induced NO production through MDP is mediated by TLR2 and CD14/MyD88-, and <t>NOD2-dependent</t> pathway. (A) RAW 264.7 cells (500 μl of 5 × 10 5 cells/ml) were treated with SaLTA (0 or 3 μg/ml) and/or MDP (0 or 10 μg/ml) for 24 h. The cells were then stained with anti-mouse TLR2 <t>conjugated</t> with PE or CD14 conjugated with FITC antibodies. For intracellular NOD2, the cells were permeabilized and stained with anti-mouse NOD2 conjugated with PE. The stained cells were then subjected to flow cytometry. Upper , a representative TLR2, NOD2, and CD14 expression determined by flow cytometric analysis. Values given in each histogram indicate mean fluorescence intensity (MFI) of 10,000 events of live cells in the treatment group. Lower , the expression of TLR2, NOD2, and CD14 is presented as mean of MFI ± S.D. of three replicates for each group. Asterisk (*) indicates a significant difference between the indicated treatment groups at P < 0.05. NT, non-treatment group; IC, isotype control. (B, C) BMMs (200 μl of 5 × 10 5 cells/ml) from wild-type, (B) TLR2-, MyD88- CD14- or (C) <t>NOD2-deficient</t> mice were treated with SaLTA (0 or 3 μg/ml) and/or MDP (0 or 10 μg/ml) for 24 h. Then, nitrite levels in the supernatants were measured. The data represent mean ± S.D. of three replicates for each group. * indicates a significant difference compared to the control group at P < 0.05.
Caspaseiii, supplied by Novus Biologicals, used in various techniques. Bioz Stars score: 95/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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mouse monoclonal ape1 ab  (Novus Biologicals)
94
Novus Biologicals mouse monoclonal ape1 ab
Enhanced SaLTA-induced NO production through MDP is mediated by TLR2 and CD14/MyD88-, and <t>NOD2-dependent</t> pathway. (A) RAW 264.7 cells (500 μl of 5 × 10 5 cells/ml) were treated with SaLTA (0 or 3 μg/ml) and/or MDP (0 or 10 μg/ml) for 24 h. The cells were then stained with anti-mouse TLR2 <t>conjugated</t> with PE or CD14 conjugated with FITC antibodies. For intracellular NOD2, the cells were permeabilized and stained with anti-mouse NOD2 conjugated with PE. The stained cells were then subjected to flow cytometry. Upper , a representative TLR2, NOD2, and CD14 expression determined by flow cytometric analysis. Values given in each histogram indicate mean fluorescence intensity (MFI) of 10,000 events of live cells in the treatment group. Lower , the expression of TLR2, NOD2, and CD14 is presented as mean of MFI ± S.D. of three replicates for each group. Asterisk (*) indicates a significant difference between the indicated treatment groups at P < 0.05. NT, non-treatment group; IC, isotype control. (B, C) BMMs (200 μl of 5 × 10 5 cells/ml) from wild-type, (B) TLR2-, MyD88- CD14- or (C) <t>NOD2-deficient</t> mice were treated with SaLTA (0 or 3 μg/ml) and/or MDP (0 or 10 μg/ml) for 24 h. Then, nitrite levels in the supernatants were measured. The data represent mean ± S.D. of three replicates for each group. * indicates a significant difference compared to the control group at P < 0.05.
Mouse Monoclonal Ape1 Ab, supplied by Novus Biologicals, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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ikk β antibody  (Novus Biologicals)
94
Novus Biologicals ikk β antibody
Enhanced SaLTA-induced NO production through MDP is mediated by TLR2 and CD14/MyD88-, and <t>NOD2-dependent</t> pathway. (A) RAW 264.7 cells (500 μl of 5 × 10 5 cells/ml) were treated with SaLTA (0 or 3 μg/ml) and/or MDP (0 or 10 μg/ml) for 24 h. The cells were then stained with anti-mouse TLR2 <t>conjugated</t> with PE or CD14 conjugated with FITC antibodies. For intracellular NOD2, the cells were permeabilized and stained with anti-mouse NOD2 conjugated with PE. The stained cells were then subjected to flow cytometry. Upper , a representative TLR2, NOD2, and CD14 expression determined by flow cytometric analysis. Values given in each histogram indicate mean fluorescence intensity (MFI) of 10,000 events of live cells in the treatment group. Lower , the expression of TLR2, NOD2, and CD14 is presented as mean of MFI ± S.D. of three replicates for each group. Asterisk (*) indicates a significant difference between the indicated treatment groups at P < 0.05. NT, non-treatment group; IC, isotype control. (B, C) BMMs (200 μl of 5 × 10 5 cells/ml) from wild-type, (B) TLR2-, MyD88- CD14- or (C) <t>NOD2-deficient</t> mice were treated with SaLTA (0 or 3 μg/ml) and/or MDP (0 or 10 μg/ml) for 24 h. Then, nitrite levels in the supernatants were measured. The data represent mean ± S.D. of three replicates for each group. * indicates a significant difference compared to the control group at P < 0.05.
Ikk β Antibody, supplied by Novus Biologicals, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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anti ps1 antibody aps18  (Novus Biologicals)
90
Novus Biologicals anti ps1 antibody aps18
Enhanced SaLTA-induced NO production through MDP is mediated by TLR2 and CD14/MyD88-, and <t>NOD2-dependent</t> pathway. (A) RAW 264.7 cells (500 μl of 5 × 10 5 cells/ml) were treated with SaLTA (0 or 3 μg/ml) and/or MDP (0 or 10 μg/ml) for 24 h. The cells were then stained with anti-mouse TLR2 <t>conjugated</t> with PE or CD14 conjugated with FITC antibodies. For intracellular NOD2, the cells were permeabilized and stained with anti-mouse NOD2 conjugated with PE. The stained cells were then subjected to flow cytometry. Upper , a representative TLR2, NOD2, and CD14 expression determined by flow cytometric analysis. Values given in each histogram indicate mean fluorescence intensity (MFI) of 10,000 events of live cells in the treatment group. Lower , the expression of TLR2, NOD2, and CD14 is presented as mean of MFI ± S.D. of three replicates for each group. Asterisk (*) indicates a significant difference between the indicated treatment groups at P < 0.05. NT, non-treatment group; IC, isotype control. (B, C) BMMs (200 μl of 5 × 10 5 cells/ml) from wild-type, (B) TLR2-, MyD88- CD14- or (C) <t>NOD2-deficient</t> mice were treated with SaLTA (0 or 3 μg/ml) and/or MDP (0 or 10 μg/ml) for 24 h. Then, nitrite levels in the supernatants were measured. The data represent mean ± S.D. of three replicates for each group. * indicates a significant difference compared to the control group at P < 0.05.
Anti Ps1 Antibody Aps18, supplied by Novus Biologicals, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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hif 2α epas1  (Novus Biologicals)
96
Novus Biologicals hif 2α epas1
Enhanced SaLTA-induced NO production through MDP is mediated by TLR2 and CD14/MyD88-, and <t>NOD2-dependent</t> pathway. (A) RAW 264.7 cells (500 μl of 5 × 10 5 cells/ml) were treated with SaLTA (0 or 3 μg/ml) and/or MDP (0 or 10 μg/ml) for 24 h. The cells were then stained with anti-mouse TLR2 <t>conjugated</t> with PE or CD14 conjugated with FITC antibodies. For intracellular NOD2, the cells were permeabilized and stained with anti-mouse NOD2 conjugated with PE. The stained cells were then subjected to flow cytometry. Upper , a representative TLR2, NOD2, and CD14 expression determined by flow cytometric analysis. Values given in each histogram indicate mean fluorescence intensity (MFI) of 10,000 events of live cells in the treatment group. Lower , the expression of TLR2, NOD2, and CD14 is presented as mean of MFI ± S.D. of three replicates for each group. Asterisk (*) indicates a significant difference between the indicated treatment groups at P < 0.05. NT, non-treatment group; IC, isotype control. (B, C) BMMs (200 μl of 5 × 10 5 cells/ml) from wild-type, (B) TLR2-, MyD88- CD14- or (C) <t>NOD2-deficient</t> mice were treated with SaLTA (0 or 3 μg/ml) and/or MDP (0 or 10 μg/ml) for 24 h. Then, nitrite levels in the supernatants were measured. The data represent mean ± S.D. of three replicates for each group. * indicates a significant difference compared to the control group at P < 0.05.
Hif 2α Epas1, supplied by Novus Biologicals, used in various techniques. Bioz Stars score: 96/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Image Search Results


Journal: Cell Reports

Article Title: USP15 Deubiquitinase Safeguards Hematopoiesis and Genome Integrity in Hematopoietic Stem Cells and Leukemia Cells

doi: 10.1016/j.celrep.2020.108533

Figure Lengend Snippet:

Article Snippet: Rabbit Polyclonal Anti-FUS antibody , Novus Biologicals , Cat# NB100-565.

Techniques: Recombinant, Gene Knockout, Transfection, Cell Isolation, RNA Sequencing, Knockdown, Mass Spectrometry, Expressing, Knock-Out, Illumina Sequencing, Multiplexing, CRISPR, Control, shRNA, Sequencing, Quantitative RT-PCR, Plasmid Preparation, Software, Quantitative Proteomics, Irradiation, Imaging

Enhanced SaLTA-induced NO production through MDP is mediated by TLR2 and CD14/MyD88-, and NOD2-dependent pathway. (A) RAW 264.7 cells (500 μl of 5 × 10 5 cells/ml) were treated with SaLTA (0 or 3 μg/ml) and/or MDP (0 or 10 μg/ml) for 24 h. The cells were then stained with anti-mouse TLR2 conjugated with PE or CD14 conjugated with FITC antibodies. For intracellular NOD2, the cells were permeabilized and stained with anti-mouse NOD2 conjugated with PE. The stained cells were then subjected to flow cytometry. Upper , a representative TLR2, NOD2, and CD14 expression determined by flow cytometric analysis. Values given in each histogram indicate mean fluorescence intensity (MFI) of 10,000 events of live cells in the treatment group. Lower , the expression of TLR2, NOD2, and CD14 is presented as mean of MFI ± S.D. of three replicates for each group. Asterisk (*) indicates a significant difference between the indicated treatment groups at P < 0.05. NT, non-treatment group; IC, isotype control. (B, C) BMMs (200 μl of 5 × 10 5 cells/ml) from wild-type, (B) TLR2-, MyD88- CD14- or (C) NOD2-deficient mice were treated with SaLTA (0 or 3 μg/ml) and/or MDP (0 or 10 μg/ml) for 24 h. Then, nitrite levels in the supernatants were measured. The data represent mean ± S.D. of three replicates for each group. * indicates a significant difference compared to the control group at P < 0.05.

Journal: Frontiers in Immunology

Article Title: Muramyl dipeptide potentiates Staphylococcus aureus lipoteichoic acid-induced nitric oxide production via TLR2/NOD2/PAFR signaling pathways

doi: 10.3389/fimmu.2024.1451315

Figure Lengend Snippet: Enhanced SaLTA-induced NO production through MDP is mediated by TLR2 and CD14/MyD88-, and NOD2-dependent pathway. (A) RAW 264.7 cells (500 μl of 5 × 10 5 cells/ml) were treated with SaLTA (0 or 3 μg/ml) and/or MDP (0 or 10 μg/ml) for 24 h. The cells were then stained with anti-mouse TLR2 conjugated with PE or CD14 conjugated with FITC antibodies. For intracellular NOD2, the cells were permeabilized and stained with anti-mouse NOD2 conjugated with PE. The stained cells were then subjected to flow cytometry. Upper , a representative TLR2, NOD2, and CD14 expression determined by flow cytometric analysis. Values given in each histogram indicate mean fluorescence intensity (MFI) of 10,000 events of live cells in the treatment group. Lower , the expression of TLR2, NOD2, and CD14 is presented as mean of MFI ± S.D. of three replicates for each group. Asterisk (*) indicates a significant difference between the indicated treatment groups at P < 0.05. NT, non-treatment group; IC, isotype control. (B, C) BMMs (200 μl of 5 × 10 5 cells/ml) from wild-type, (B) TLR2-, MyD88- CD14- or (C) NOD2-deficient mice were treated with SaLTA (0 or 3 μg/ml) and/or MDP (0 or 10 μg/ml) for 24 h. Then, nitrite levels in the supernatants were measured. The data represent mean ± S.D. of three replicates for each group. * indicates a significant difference compared to the control group at P < 0.05.

Article Snippet: For flow cytometric analysis, antibodies specific to TLR2 conjugated with phycoerythrin (PE, Catalog No. 12-9021-82), and to CD14 conjugated with fluorescein isothiocyanate (FITC, Catalog No. 123308) were obtained from eBioscience (San Diego, CA, USA) and Biolegend (San Diego, CA, USA), respectively, while antibody specific to NOD2 conjugated with PE (Catalog No. NB100-524PE) was purchased from Novus (Los Angeles, CA, USA).

Techniques: Staining, Flow Cytometry, Expressing, Fluorescence, Control