mct1 Search Results


anti mct1  (Novus Biologicals)
93
Novus Biologicals anti mct1
Anti Mct1, supplied by Novus Biologicals, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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human mct1 slc16a1 monoclonal antibody  (R&D Systems)
91
R&D Systems human mct1 slc16a1 monoclonal antibody
Human Mct1 Slc16a1 Monoclonal Antibody, supplied by R&D Systems, used in various techniques. Bioz Stars score: 91/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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anti human mouse mct1 pe  (R&D Systems Hematology)
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R&D Systems Hematology anti human mouse mct1 pe
Anti Human Mouse Mct1 Pe, supplied by R&D Systems Hematology, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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fab8275v 100ug  (R&D Systems)
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R&D Systems fab8275v 100ug
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antibodies against mct 1  (R&D Systems)
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R&D Systems antibodies against mct 1
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anti mouse mct1 ab  (Proteintech)
96
Proteintech anti mouse mct1 ab
Figure 3. Bsg deficiency impairs hepatic availability of lactate and pyruvate in vivo. (A) Schematic illustrating impaired gluconeogenesis resulting from decreased substrate import due to Bsg deficiency. OAA, oxaloacetate. (B) Blood glucose levels in Bsg+/+ and Bsg–/– mice under fasting conditions. White columns and circles, Bsg+/+ mice; gray columns and black circles, Bsg–/– mice (n = 10–12/genotype). Scatter plots display the data for individual mice. (C) Dif- ferences in blood lactate values between feeding and fasting states in Bsg+/+ or Bsg–/– female mice (n = 5–6/genotype). (D) Differences in serum pyruvate values between feeding and fasting states in Bsg+/+ or Bsg–/– female mice (n = 5–6/genotype). (E) G6P and F6P with incorporation of 13C3-labeled carbon in isolated Bsg+/+ or Bsg–/– hepatocytes. White columns, Bsg+/+ hepatocytes; gray columns, Bsg–/– hepatocytes. n = 5 for independent experiments. N.D., no detection. (F) Blood glucose excursions and the AUC scores in fasting Bsg+/+ and Bsg–/– female mice during lactate tolerance tests (n = 7–11/genotype). (G) Blood glucose excursions and AUC scores during pyruvate tolerance tests in female mice (n = 8–10/genotype). (H) Endogenous glucose production in isolat- ed hepatocytes of Bsg+/+ and Bsg–/– female mice cultured in medium supplemented with 20 mM sodium lactate and 2 mM sodium pyruvate in the absence or presence of 100 nM AZD3965 (an inhibitor of <t>MCT1</t> activity). n = 6 for independent experiments. For all relevant panels, data are presented as means ± SEM. *P < 0.05, **P < 0.01, ***P < 0.001, for the comparison of Bsg+/+ and Bsg–/– at the indicated time point (2-tailed unpaired Student’s t test).
Anti Mouse Mct1 Ab, supplied by Proteintech, used in various techniques. Bioz Stars score: 96/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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anti mouse mcpt  (Proteintech)
93
Proteintech anti mouse mcpt
Figure 3. Bsg deficiency impairs hepatic availability of lactate and pyruvate in vivo. (A) Schematic illustrating impaired gluconeogenesis resulting from decreased substrate import due to Bsg deficiency. OAA, oxaloacetate. (B) Blood glucose levels in Bsg+/+ and Bsg–/– mice under fasting conditions. White columns and circles, Bsg+/+ mice; gray columns and black circles, Bsg–/– mice (n = 10–12/genotype). Scatter plots display the data for individual mice. (C) Dif- ferences in blood lactate values between feeding and fasting states in Bsg+/+ or Bsg–/– female mice (n = 5–6/genotype). (D) Differences in serum pyruvate values between feeding and fasting states in Bsg+/+ or Bsg–/– female mice (n = 5–6/genotype). (E) G6P and F6P with incorporation of 13C3-labeled carbon in isolated Bsg+/+ or Bsg–/– hepatocytes. White columns, Bsg+/+ hepatocytes; gray columns, Bsg–/– hepatocytes. n = 5 for independent experiments. N.D., no detection. (F) Blood glucose excursions and the AUC scores in fasting Bsg+/+ and Bsg–/– female mice during lactate tolerance tests (n = 7–11/genotype). (G) Blood glucose excursions and AUC scores during pyruvate tolerance tests in female mice (n = 8–10/genotype). (H) Endogenous glucose production in isolat- ed hepatocytes of Bsg+/+ and Bsg–/– female mice cultured in medium supplemented with 20 mM sodium lactate and 2 mM sodium pyruvate in the absence or presence of 100 nM AZD3965 (an inhibitor of <t>MCT1</t> activity). n = 6 for independent experiments. For all relevant panels, data are presented as means ± SEM. *P < 0.05, **P < 0.01, ***P < 0.001, for the comparison of Bsg+/+ and Bsg–/– at the indicated time point (2-tailed unpaired Student’s t test).
Anti Mouse Mcpt, supplied by Proteintech, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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mct1  (Santa Cruz Biotechnology)
95
Santa Cruz Biotechnology mct1
Figure 3. Bsg deficiency impairs hepatic availability of lactate and pyruvate in vivo. (A) Schematic illustrating impaired gluconeogenesis resulting from decreased substrate import due to Bsg deficiency. OAA, oxaloacetate. (B) Blood glucose levels in Bsg+/+ and Bsg–/– mice under fasting conditions. White columns and circles, Bsg+/+ mice; gray columns and black circles, Bsg–/– mice (n = 10–12/genotype). Scatter plots display the data for individual mice. (C) Dif- ferences in blood lactate values between feeding and fasting states in Bsg+/+ or Bsg–/– female mice (n = 5–6/genotype). (D) Differences in serum pyruvate values between feeding and fasting states in Bsg+/+ or Bsg–/– female mice (n = 5–6/genotype). (E) G6P and F6P with incorporation of 13C3-labeled carbon in isolated Bsg+/+ or Bsg–/– hepatocytes. White columns, Bsg+/+ hepatocytes; gray columns, Bsg–/– hepatocytes. n = 5 for independent experiments. N.D., no detection. (F) Blood glucose excursions and the AUC scores in fasting Bsg+/+ and Bsg–/– female mice during lactate tolerance tests (n = 7–11/genotype). (G) Blood glucose excursions and AUC scores during pyruvate tolerance tests in female mice (n = 8–10/genotype). (H) Endogenous glucose production in isolat- ed hepatocytes of Bsg+/+ and Bsg–/– female mice cultured in medium supplemented with 20 mM sodium lactate and 2 mM sodium pyruvate in the absence or presence of 100 nM AZD3965 (an inhibitor of <t>MCT1</t> activity). n = 6 for independent experiments. For all relevant panels, data are presented as means ± SEM. *P < 0.05, **P < 0.01, ***P < 0.001, for the comparison of Bsg+/+ and Bsg–/– at the indicated time point (2-tailed unpaired Student’s t test).
Mct1, supplied by Santa Cruz Biotechnology, used in various techniques. Bioz Stars score: 95/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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mct1 slc16a3 bs 10249r antibodies  (Bioss)
94
Bioss mct1 slc16a3 bs 10249r antibodies
Figure 3. Bsg deficiency impairs hepatic availability of lactate and pyruvate in vivo. (A) Schematic illustrating impaired gluconeogenesis resulting from decreased substrate import due to Bsg deficiency. OAA, oxaloacetate. (B) Blood glucose levels in Bsg+/+ and Bsg–/– mice under fasting conditions. White columns and circles, Bsg+/+ mice; gray columns and black circles, Bsg–/– mice (n = 10–12/genotype). Scatter plots display the data for individual mice. (C) Dif- ferences in blood lactate values between feeding and fasting states in Bsg+/+ or Bsg–/– female mice (n = 5–6/genotype). (D) Differences in serum pyruvate values between feeding and fasting states in Bsg+/+ or Bsg–/– female mice (n = 5–6/genotype). (E) G6P and F6P with incorporation of 13C3-labeled carbon in isolated Bsg+/+ or Bsg–/– hepatocytes. White columns, Bsg+/+ hepatocytes; gray columns, Bsg–/– hepatocytes. n = 5 for independent experiments. N.D., no detection. (F) Blood glucose excursions and the AUC scores in fasting Bsg+/+ and Bsg–/– female mice during lactate tolerance tests (n = 7–11/genotype). (G) Blood glucose excursions and AUC scores during pyruvate tolerance tests in female mice (n = 8–10/genotype). (H) Endogenous glucose production in isolat- ed hepatocytes of Bsg+/+ and Bsg–/– female mice cultured in medium supplemented with 20 mM sodium lactate and 2 mM sodium pyruvate in the absence or presence of 100 nM AZD3965 (an inhibitor of <t>MCT1</t> activity). n = 6 for independent experiments. For all relevant panels, data are presented as means ± SEM. *P < 0.05, **P < 0.01, ***P < 0.001, for the comparison of Bsg+/+ and Bsg–/– at the indicated time point (2-tailed unpaired Student’s t test).
Mct1 Slc16a3 Bs 10249r Antibodies, supplied by Bioss, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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antibodies mct1 slc16a1  (Alomone Labs)
92
Alomone Labs antibodies mct1 slc16a1
Figure 3. Bsg deficiency impairs hepatic availability of lactate and pyruvate in vivo. (A) Schematic illustrating impaired gluconeogenesis resulting from decreased substrate import due to Bsg deficiency. OAA, oxaloacetate. (B) Blood glucose levels in Bsg+/+ and Bsg–/– mice under fasting conditions. White columns and circles, Bsg+/+ mice; gray columns and black circles, Bsg–/– mice (n = 10–12/genotype). Scatter plots display the data for individual mice. (C) Dif- ferences in blood lactate values between feeding and fasting states in Bsg+/+ or Bsg–/– female mice (n = 5–6/genotype). (D) Differences in serum pyruvate values between feeding and fasting states in Bsg+/+ or Bsg–/– female mice (n = 5–6/genotype). (E) G6P and F6P with incorporation of 13C3-labeled carbon in isolated Bsg+/+ or Bsg–/– hepatocytes. White columns, Bsg+/+ hepatocytes; gray columns, Bsg–/– hepatocytes. n = 5 for independent experiments. N.D., no detection. (F) Blood glucose excursions and the AUC scores in fasting Bsg+/+ and Bsg–/– female mice during lactate tolerance tests (n = 7–11/genotype). (G) Blood glucose excursions and AUC scores during pyruvate tolerance tests in female mice (n = 8–10/genotype). (H) Endogenous glucose production in isolat- ed hepatocytes of Bsg+/+ and Bsg–/– female mice cultured in medium supplemented with 20 mM sodium lactate and 2 mM sodium pyruvate in the absence or presence of 100 nM AZD3965 (an inhibitor of <t>MCT1</t> activity). n = 6 for independent experiments. For all relevant panels, data are presented as means ± SEM. *P < 0.05, **P < 0.01, ***P < 0.001, for the comparison of Bsg+/+ and Bsg–/– at the indicated time point (2-tailed unpaired Student’s t test).
Antibodies Mct1 Slc16a1, supplied by Alomone Labs, used in various techniques. Bioz Stars score: 92/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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mct1  (Bioss)
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Bioss mct1
Figure 3. Bsg deficiency impairs hepatic availability of lactate and pyruvate in vivo. (A) Schematic illustrating impaired gluconeogenesis resulting from decreased substrate import due to Bsg deficiency. OAA, oxaloacetate. (B) Blood glucose levels in Bsg+/+ and Bsg–/– mice under fasting conditions. White columns and circles, Bsg+/+ mice; gray columns and black circles, Bsg–/– mice (n = 10–12/genotype). Scatter plots display the data for individual mice. (C) Dif- ferences in blood lactate values between feeding and fasting states in Bsg+/+ or Bsg–/– female mice (n = 5–6/genotype). (D) Differences in serum pyruvate values between feeding and fasting states in Bsg+/+ or Bsg–/– female mice (n = 5–6/genotype). (E) G6P and F6P with incorporation of 13C3-labeled carbon in isolated Bsg+/+ or Bsg–/– hepatocytes. White columns, Bsg+/+ hepatocytes; gray columns, Bsg–/– hepatocytes. n = 5 for independent experiments. N.D., no detection. (F) Blood glucose excursions and the AUC scores in fasting Bsg+/+ and Bsg–/– female mice during lactate tolerance tests (n = 7–11/genotype). (G) Blood glucose excursions and AUC scores during pyruvate tolerance tests in female mice (n = 8–10/genotype). (H) Endogenous glucose production in isolat- ed hepatocytes of Bsg+/+ and Bsg–/– female mice cultured in medium supplemented with 20 mM sodium lactate and 2 mM sodium pyruvate in the absence or presence of 100 nM AZD3965 (an inhibitor of <t>MCT1</t> activity). n = 6 for independent experiments. For all relevant panels, data are presented as means ± SEM. *P < 0.05, **P < 0.01, ***P < 0.001, for the comparison of Bsg+/+ and Bsg–/– at the indicated time point (2-tailed unpaired Student’s t test).
Mct1, supplied by Bioss, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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anti mct1  (Boster Bio)
90
Boster Bio anti mct1
Figure 3. Bsg deficiency impairs hepatic availability of lactate and pyruvate in vivo. (A) Schematic illustrating impaired gluconeogenesis resulting from decreased substrate import due to Bsg deficiency. OAA, oxaloacetate. (B) Blood glucose levels in Bsg+/+ and Bsg–/– mice under fasting conditions. White columns and circles, Bsg+/+ mice; gray columns and black circles, Bsg–/– mice (n = 10–12/genotype). Scatter plots display the data for individual mice. (C) Dif- ferences in blood lactate values between feeding and fasting states in Bsg+/+ or Bsg–/– female mice (n = 5–6/genotype). (D) Differences in serum pyruvate values between feeding and fasting states in Bsg+/+ or Bsg–/– female mice (n = 5–6/genotype). (E) G6P and F6P with incorporation of 13C3-labeled carbon in isolated Bsg+/+ or Bsg–/– hepatocytes. White columns, Bsg+/+ hepatocytes; gray columns, Bsg–/– hepatocytes. n = 5 for independent experiments. N.D., no detection. (F) Blood glucose excursions and the AUC scores in fasting Bsg+/+ and Bsg–/– female mice during lactate tolerance tests (n = 7–11/genotype). (G) Blood glucose excursions and AUC scores during pyruvate tolerance tests in female mice (n = 8–10/genotype). (H) Endogenous glucose production in isolat- ed hepatocytes of Bsg+/+ and Bsg–/– female mice cultured in medium supplemented with 20 mM sodium lactate and 2 mM sodium pyruvate in the absence or presence of 100 nM AZD3965 (an inhibitor of <t>MCT1</t> activity). n = 6 for independent experiments. For all relevant panels, data are presented as means ± SEM. *P < 0.05, **P < 0.01, ***P < 0.001, for the comparison of Bsg+/+ and Bsg–/– at the indicated time point (2-tailed unpaired Student’s t test).
Anti Mct1, supplied by Boster Bio, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Image Search Results


Figure 3. Bsg deficiency impairs hepatic availability of lactate and pyruvate in vivo. (A) Schematic illustrating impaired gluconeogenesis resulting from decreased substrate import due to Bsg deficiency. OAA, oxaloacetate. (B) Blood glucose levels in Bsg+/+ and Bsg–/– mice under fasting conditions. White columns and circles, Bsg+/+ mice; gray columns and black circles, Bsg–/– mice (n = 10–12/genotype). Scatter plots display the data for individual mice. (C) Dif- ferences in blood lactate values between feeding and fasting states in Bsg+/+ or Bsg–/– female mice (n = 5–6/genotype). (D) Differences in serum pyruvate values between feeding and fasting states in Bsg+/+ or Bsg–/– female mice (n = 5–6/genotype). (E) G6P and F6P with incorporation of 13C3-labeled carbon in isolated Bsg+/+ or Bsg–/– hepatocytes. White columns, Bsg+/+ hepatocytes; gray columns, Bsg–/– hepatocytes. n = 5 for independent experiments. N.D., no detection. (F) Blood glucose excursions and the AUC scores in fasting Bsg+/+ and Bsg–/– female mice during lactate tolerance tests (n = 7–11/genotype). (G) Blood glucose excursions and AUC scores during pyruvate tolerance tests in female mice (n = 8–10/genotype). (H) Endogenous glucose production in isolat- ed hepatocytes of Bsg+/+ and Bsg–/– female mice cultured in medium supplemented with 20 mM sodium lactate and 2 mM sodium pyruvate in the absence or presence of 100 nM AZD3965 (an inhibitor of MCT1 activity). n = 6 for independent experiments. For all relevant panels, data are presented as means ± SEM. *P < 0.05, **P < 0.01, ***P < 0.001, for the comparison of Bsg+/+ and Bsg–/– at the indicated time point (2-tailed unpaired Student’s t test).

Journal: JCI insight

Article Title: Basigin deficiency prevents anaplerosis and ameliorates insulin resistance and hepatosteatosis.

doi: 10.1172/jci.insight.142464

Figure Lengend Snippet: Figure 3. Bsg deficiency impairs hepatic availability of lactate and pyruvate in vivo. (A) Schematic illustrating impaired gluconeogenesis resulting from decreased substrate import due to Bsg deficiency. OAA, oxaloacetate. (B) Blood glucose levels in Bsg+/+ and Bsg–/– mice under fasting conditions. White columns and circles, Bsg+/+ mice; gray columns and black circles, Bsg–/– mice (n = 10–12/genotype). Scatter plots display the data for individual mice. (C) Dif- ferences in blood lactate values between feeding and fasting states in Bsg+/+ or Bsg–/– female mice (n = 5–6/genotype). (D) Differences in serum pyruvate values between feeding and fasting states in Bsg+/+ or Bsg–/– female mice (n = 5–6/genotype). (E) G6P and F6P with incorporation of 13C3-labeled carbon in isolated Bsg+/+ or Bsg–/– hepatocytes. White columns, Bsg+/+ hepatocytes; gray columns, Bsg–/– hepatocytes. n = 5 for independent experiments. N.D., no detection. (F) Blood glucose excursions and the AUC scores in fasting Bsg+/+ and Bsg–/– female mice during lactate tolerance tests (n = 7–11/genotype). (G) Blood glucose excursions and AUC scores during pyruvate tolerance tests in female mice (n = 8–10/genotype). (H) Endogenous glucose production in isolat- ed hepatocytes of Bsg+/+ and Bsg–/– female mice cultured in medium supplemented with 20 mM sodium lactate and 2 mM sodium pyruvate in the absence or presence of 100 nM AZD3965 (an inhibitor of MCT1 activity). n = 6 for independent experiments. For all relevant panels, data are presented as means ± SEM. *P < 0.05, **P < 0.01, ***P < 0.001, for the comparison of Bsg+/+ and Bsg–/– at the indicated time point (2-tailed unpaired Student’s t test).

Article Snippet: The resulting sections were stained with rabbit monoclonal anti–mouse BSG antibody (Ab) (catalog ab212057; Abcam), rabbit anti–mouse MCT1 Ab (catalog 20139-1-AP; Proteintech), rabbit anti–mouse MCT4 Ab (catalog 22787-1-AP; Proteintech), or FITC-conjugated goat anti–mouse IgG Ab (catalog 115-095- 062; Jackson ImmunoResearch).

Techniques: In Vivo, Labeling, Isolation, Cell Culture, Activity Assay, Comparison