exp2 function Search Results


90
MathWorks Inc general exponential functions exp2
General Exponential Functions Exp2, supplied by MathWorks Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/general exponential functions exp2/product/MathWorks Inc
Average 90 stars, based on 1 article reviews
general exponential functions exp2 - by Bioz Stars, 2026-03
90/100 stars
  Buy from Supplier

90
MathWorks Inc exp2 fitting routine
Exp2 Fitting Routine, supplied by MathWorks Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/exp2 fitting routine/product/MathWorks Inc
Average 90 stars, based on 1 article reviews
exp2 fitting routine - by Bioz Stars, 2026-03
90/100 stars
  Buy from Supplier

90
MathWorks Inc matlab function fit(exp2)
Matlab Function Fit(Exp2), supplied by MathWorks Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/matlab function fit(exp2)/product/MathWorks Inc
Average 90 stars, based on 1 article reviews
matlab function fit(exp2) - by Bioz Stars, 2026-03
90/100 stars
  Buy from Supplier

90
MathWorks Inc curve fitting toolbox exp2 function
Curve Fitting Toolbox Exp2 Function, supplied by MathWorks Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/curve fitting toolbox exp2 function/product/MathWorks Inc
Average 90 stars, based on 1 article reviews
curve fitting toolbox exp2 function - by Bioz Stars, 2026-03
90/100 stars
  Buy from Supplier

93
Proteintech cse1l
Firstly, we leveraged ABC model to establish genome-wide regulatory maps genes across 20 cancer types by integrating large-scale multi-omics data including chromatin accessibility (ATAC-seq or DNase-seq), histone modifications (H3K27ac ChIP-seq), and chromatin interaction (Hi-C). We further characterized the functional characteristics of ABC variants based on genomic distribution, functional annotation and GWAS enrichment. Meanwhile, pathway analysis, mutation landscape, immune infiltration and drug response were integrated to the effects of ABC genes in tumorigenesis and clinical application. Mechanistically, we systematically screened ABC variants associated with CRC risk in 17,789 cases and 19,951 controls using GWAS chip data and independently validated in a large-scale population consisting of 6024 cases and 10,022 controls. We identified an ABC regulatory variant rs4810856 that is significantly associated with an increased risk of CRC (OR = 1.11, 95%CI = 1.05–1.16, P = 4.02×10 -5 ). Mechanistically, the ABC variant acted as an allele-specific enhancer mediated by TF ZEB1 to distally facilitate expression of PREX1 , <t>CSE1L</t> and STAU1 , which synergistically activated p-AKT signaling to drive CRC cell proliferation. The graph was created with BioRender.com.
Cse1l, supplied by Proteintech, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/cse1l/product/Proteintech
Average 93 stars, based on 1 article reviews
cse1l - by Bioz Stars, 2026-03
93/100 stars
  Buy from Supplier

96
MathWorks Inc toolbox 2009 exp2 function
Firstly, we leveraged ABC model to establish genome-wide regulatory maps genes across 20 cancer types by integrating large-scale multi-omics data including chromatin accessibility (ATAC-seq or DNase-seq), histone modifications (H3K27ac ChIP-seq), and chromatin interaction (Hi-C). We further characterized the functional characteristics of ABC variants based on genomic distribution, functional annotation and GWAS enrichment. Meanwhile, pathway analysis, mutation landscape, immune infiltration and drug response were integrated to the effects of ABC genes in tumorigenesis and clinical application. Mechanistically, we systematically screened ABC variants associated with CRC risk in 17,789 cases and 19,951 controls using GWAS chip data and independently validated in a large-scale population consisting of 6024 cases and 10,022 controls. We identified an ABC regulatory variant rs4810856 that is significantly associated with an increased risk of CRC (OR = 1.11, 95%CI = 1.05–1.16, P = 4.02×10 -5 ). Mechanistically, the ABC variant acted as an allele-specific enhancer mediated by TF ZEB1 to distally facilitate expression of PREX1 , <t>CSE1L</t> and STAU1 , which synergistically activated p-AKT signaling to drive CRC cell proliferation. The graph was created with BioRender.com.
Toolbox 2009 Exp2 Function, supplied by MathWorks Inc, used in various techniques. Bioz Stars score: 96/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/toolbox 2009 exp2 function/product/MathWorks Inc
Average 96 stars, based on 1 article reviews
toolbox 2009 exp2 function - by Bioz Stars, 2026-03
96/100 stars
  Buy from Supplier

90
MathWorks Inc exp2 function
Firstly, we leveraged ABC model to establish genome-wide regulatory maps genes across 20 cancer types by integrating large-scale multi-omics data including chromatin accessibility (ATAC-seq or DNase-seq), histone modifications (H3K27ac ChIP-seq), and chromatin interaction (Hi-C). We further characterized the functional characteristics of ABC variants based on genomic distribution, functional annotation and GWAS enrichment. Meanwhile, pathway analysis, mutation landscape, immune infiltration and drug response were integrated to the effects of ABC genes in tumorigenesis and clinical application. Mechanistically, we systematically screened ABC variants associated with CRC risk in 17,789 cases and 19,951 controls using GWAS chip data and independently validated in a large-scale population consisting of 6024 cases and 10,022 controls. We identified an ABC regulatory variant rs4810856 that is significantly associated with an increased risk of CRC (OR = 1.11, 95%CI = 1.05–1.16, P = 4.02×10 -5 ). Mechanistically, the ABC variant acted as an allele-specific enhancer mediated by TF ZEB1 to distally facilitate expression of PREX1 , <t>CSE1L</t> and STAU1 , which synergistically activated p-AKT signaling to drive CRC cell proliferation. The graph was created with BioRender.com.
Exp2 Function, supplied by MathWorks Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/exp2 function/product/MathWorks Inc
Average 90 stars, based on 1 article reviews
exp2 function - by Bioz Stars, 2026-03
90/100 stars
  Buy from Supplier

Image Search Results


Firstly, we leveraged ABC model to establish genome-wide regulatory maps genes across 20 cancer types by integrating large-scale multi-omics data including chromatin accessibility (ATAC-seq or DNase-seq), histone modifications (H3K27ac ChIP-seq), and chromatin interaction (Hi-C). We further characterized the functional characteristics of ABC variants based on genomic distribution, functional annotation and GWAS enrichment. Meanwhile, pathway analysis, mutation landscape, immune infiltration and drug response were integrated to the effects of ABC genes in tumorigenesis and clinical application. Mechanistically, we systematically screened ABC variants associated with CRC risk in 17,789 cases and 19,951 controls using GWAS chip data and independently validated in a large-scale population consisting of 6024 cases and 10,022 controls. We identified an ABC regulatory variant rs4810856 that is significantly associated with an increased risk of CRC (OR = 1.11, 95%CI = 1.05–1.16, P = 4.02×10 -5 ). Mechanistically, the ABC variant acted as an allele-specific enhancer mediated by TF ZEB1 to distally facilitate expression of PREX1 , CSE1L and STAU1 , which synergistically activated p-AKT signaling to drive CRC cell proliferation. The graph was created with BioRender.com.

Journal: Nature Communications

Article Title: Genome-wide enhancer-gene regulatory maps link causal variants to target genes underlying human cancer risk

doi: 10.1038/s41467-023-41690-z

Figure Lengend Snippet: Firstly, we leveraged ABC model to establish genome-wide regulatory maps genes across 20 cancer types by integrating large-scale multi-omics data including chromatin accessibility (ATAC-seq or DNase-seq), histone modifications (H3K27ac ChIP-seq), and chromatin interaction (Hi-C). We further characterized the functional characteristics of ABC variants based on genomic distribution, functional annotation and GWAS enrichment. Meanwhile, pathway analysis, mutation landscape, immune infiltration and drug response were integrated to the effects of ABC genes in tumorigenesis and clinical application. Mechanistically, we systematically screened ABC variants associated with CRC risk in 17,789 cases and 19,951 controls using GWAS chip data and independently validated in a large-scale population consisting of 6024 cases and 10,022 controls. We identified an ABC regulatory variant rs4810856 that is significantly associated with an increased risk of CRC (OR = 1.11, 95%CI = 1.05–1.16, P = 4.02×10 -5 ). Mechanistically, the ABC variant acted as an allele-specific enhancer mediated by TF ZEB1 to distally facilitate expression of PREX1 , CSE1L and STAU1 , which synergistically activated p-AKT signaling to drive CRC cell proliferation. The graph was created with BioRender.com.

Article Snippet: Protein was incubated with antibodies against PREX1(1:1000, CST, Cat# 13168), CSE1L (1:1000, Proteintech, Cat# 22219-1-AP), STAU1 (1:1000, Proteintech, Cat# 14225-1-AP), p-AKT (Ser473, 1:1000, Proteintech, Cat# 66444-1-lg), AKT (1:1000, Proteintech, Cat# 10176-1-AP) and GAPDH (1:1,000, Proteintech, Cat# 60004-1-lg) at 4 °C overnight.

Techniques: Genome Wide, Biomarker Discovery, ChIP-sequencing, Hi-C, Functional Assay, Mutagenesis, Variant Assay, Expressing