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Image Search Results
Journal: Cellular physiology and biochemistry : international journal of experimental cellular physiology, biochemistry, and pharmacology
Article Title: Morphine Postconditioning Protects against Reperfusion Injury via Inhibiting JNK/p38 MAPK and Mitochondrial Permeability Transition Pores Signaling Pathways.
doi: 10.1159/000445605
Figure Lengend Snippet: Fig. 5. Effects of various treatments on Cytochrome c (cyt-c) in cytosol. The up per panel shows representative Cyt-c levels of 5 groups. The densities of Cyt-c were normalized against that of β-actin, and the results were expressed as percen tage to TC as shown in the low panel. TC, time control; I/R, ischemia reperfusion; MpostC, morphine postcondtioning (3.0 μmol/L); aniso, anisomycin (1.0 μmol/L); Cyt-c, cytochrome c. Values were presen ted as mean ± SD (n = 5/group). *P < 0.05 versus TC, †P < 0.05 versus I/R, §P < 0.05 versus MpostC.
Article Snippet: The proteins were loaded on SDS-polyacrylamide gel and transferred to a nitrocellulose membrane and incubated with antibodies against phosphorylated JNK (p-JNK), JNK, phosphorylated p38 MAPK (p-p38 MAPK), p38 MAPK and
Techniques: Control