cpae Search Results


94
ATCC cpae endothelial cells
Cpae Endothelial Cells, supplied by ATCC, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Vec Technologies cpae cells
Cpae Cells, supplied by Vec Technologies, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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90
Viollier AG pilus biogenesis machinery
ParAs involved in macromolecular complex transport Partial sequence alignment shows the conserved deviant Walker-box (grey) that defines the family.
Pilus Biogenesis Machinery, supplied by Viollier AG, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Viollier AG podj
ParAs involved in macromolecular complex transport Partial sequence alignment shows the conserved deviant Walker-box (grey) that defines the family.
Podj, supplied by Viollier AG, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Korean Cell Line Bank bovine pulmonary epithelium cpae kclb no. 10209
Erlotinib inhibits proliferation of human NSCLC A549 and H1299 cells. ( A ) A549, ( B ) H1299, ( C ) <t>CPAE,</t> and ( D ) IMR90 cells were treated with erlotinib for 24–72 h. After incubation, cell viability was measured by the EZ-Cytox Enhanced Cell Viability Assay Kit. Experiments were performed in triplicate. Data represent mean ± SD.
Bovine Pulmonary Epithelium Cpae Kclb No. 10209, supplied by Korean Cell Line Bank, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Korean Cell Line Bank cpae cells
The effects of rocuronium bromide treatment on the viability of calf pulmonary artery endothelial <t>(CPAE)</t> <t>cells.</t> A, control group; B, 1-μg/mL rocuronium bromide-treated group; C, 5-μg/mL rocuronium bromide-treated group; D, 10-μg/mL rocuronium bromide-treated group; E, 50-μg/mL rocuronium bromide-treated group; F, 100-μg/mL rocuronium bromidetreated group; G, 500-μg/mL rocuronium bromide-treated group; H, 1,000-μg/mL rocuronium bromide-treated group. The results are presented as mean±standard error of mean. * P<0.05 compared to the control group.
Cpae Cells, supplied by Korean Cell Line Bank, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Verlag GmbH collected papers of albert einstein (cpae)
The effects of rocuronium bromide treatment on the viability of calf pulmonary artery endothelial <t>(CPAE)</t> <t>cells.</t> A, control group; B, 1-μg/mL rocuronium bromide-treated group; C, 5-μg/mL rocuronium bromide-treated group; D, 10-μg/mL rocuronium bromide-treated group; E, 50-μg/mL rocuronium bromide-treated group; F, 100-μg/mL rocuronium bromidetreated group; G, 500-μg/mL rocuronium bromide-treated group; H, 1,000-μg/mL rocuronium bromide-treated group. The results are presented as mean±standard error of mean. * P<0.05 compared to the control group.
Collected Papers Of Albert Einstein (Cpae), supplied by Verlag GmbH, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Viollier AG antibodies against podj, ctra, flif, plec, cpae and mcpa
The effects of rocuronium bromide treatment on the viability of calf pulmonary artery endothelial <t>(CPAE)</t> <t>cells.</t> A, control group; B, 1-μg/mL rocuronium bromide-treated group; C, 5-μg/mL rocuronium bromide-treated group; D, 10-μg/mL rocuronium bromide-treated group; E, 50-μg/mL rocuronium bromide-treated group; F, 100-μg/mL rocuronium bromidetreated group; G, 500-μg/mL rocuronium bromide-treated group; H, 1,000-μg/mL rocuronium bromide-treated group. The results are presented as mean±standard error of mean. * P<0.05 compared to the control group.
Antibodies Against Podj, Ctra, Flif, Plec, Cpae And Mcpa, supplied by Viollier AG, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Vec Technologies calf pulmonary arterial endothelial cells
The effects of rocuronium bromide treatment on the viability of calf pulmonary artery endothelial <t>(CPAE)</t> <t>cells.</t> A, control group; B, 1-μg/mL rocuronium bromide-treated group; C, 5-μg/mL rocuronium bromide-treated group; D, 10-μg/mL rocuronium bromide-treated group; E, 50-μg/mL rocuronium bromide-treated group; F, 100-μg/mL rocuronium bromidetreated group; G, 500-μg/mL rocuronium bromide-treated group; H, 1,000-μg/mL rocuronium bromide-treated group. The results are presented as mean±standard error of mean. * P<0.05 compared to the control group.
Calf Pulmonary Arterial Endothelial Cells, supplied by Vec Technologies, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Korean Cell Line Bank bovine pulmonary epithelium cpae
Gefitinib inhibits proliferation of human NSCLC A549 cells. (A) A549, (B) <t>CPAE</t> and (C) IMR90 cells were treated with gefitinib for 24–72 h. After incubation, cell viability was measured by the CCK-8 assay. (D) A549 cells were incubated with gefitinib for 72 h, and then the populations of apoptotic and dead cells were analyzed using the MUSE Cell Analyzer. Experiments were performed in triplicate. Data represent mean ± SD. * p < 0.05, ** p < 0.01 versus untreated control.
Bovine Pulmonary Epithelium Cpae, supplied by Korean Cell Line Bank, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Verlag GmbH cpae 8 b, doc
Gefitinib inhibits proliferation of human NSCLC A549 cells. (A) A549, (B) <t>CPAE</t> and (C) IMR90 cells were treated with gefitinib for 24–72 h. After incubation, cell viability was measured by the CCK-8 assay. (D) A549 cells were incubated with gefitinib for 72 h, and then the populations of apoptotic and dead cells were analyzed using the MUSE Cell Analyzer. Experiments were performed in triplicate. Data represent mean ± SD. * p < 0.05, ** p < 0.01 versus untreated control.
Cpae 8 B, Doc, supplied by Verlag GmbH, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Jiangyin Tianjiang Pharmaceutical Co cpae (cat. no. 20071561)
Gefitinib inhibits proliferation of human NSCLC A549 cells. (A) A549, (B) <t>CPAE</t> and (C) IMR90 cells were treated with gefitinib for 24–72 h. After incubation, cell viability was measured by the CCK-8 assay. (D) A549 cells were incubated with gefitinib for 72 h, and then the populations of apoptotic and dead cells were analyzed using the MUSE Cell Analyzer. Experiments were performed in triplicate. Data represent mean ± SD. * p < 0.05, ** p < 0.01 versus untreated control.
Cpae (Cat. No. 20071561), supplied by Jiangyin Tianjiang Pharmaceutical Co, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Image Search Results


ParAs involved in macromolecular complex transport Partial sequence alignment shows the conserved deviant Walker-box (grey) that defines the family.

Journal: Molecular microbiology

Article Title: SURFING BIOLOGICAL SURFACES: EXPLOITING THE NUCLEOID FOR PARTITION AND TRANSPORT IN BACTERIA

doi: 10.1111/mmi.12017

Figure Lengend Snippet: ParAs involved in macromolecular complex transport Partial sequence alignment shows the conserved deviant Walker-box (grey) that defines the family.

Article Snippet: CpaE d C. crescentus , Open in a separate window , Pilus Biogenesis Machinery , Piliated Pole , ( Viollier et al ., 2002 ).

Techniques: Sequencing, Plasmid Preparation, Chemotaxis Assay

Erlotinib inhibits proliferation of human NSCLC A549 and H1299 cells. ( A ) A549, ( B ) H1299, ( C ) CPAE, and ( D ) IMR90 cells were treated with erlotinib for 24–72 h. After incubation, cell viability was measured by the EZ-Cytox Enhanced Cell Viability Assay Kit. Experiments were performed in triplicate. Data represent mean ± SD.

Journal: International Journal of Molecular Sciences

Article Title: Combination Effect of Cilengitide with Erlotinib on TGF-β1-Induced Epithelial-to-Mesenchymal Transition in Human Non-Small Cell Lung Cancer Cells

doi: 10.3390/ijms23073423

Figure Lengend Snippet: Erlotinib inhibits proliferation of human NSCLC A549 and H1299 cells. ( A ) A549, ( B ) H1299, ( C ) CPAE, and ( D ) IMR90 cells were treated with erlotinib for 24–72 h. After incubation, cell viability was measured by the EZ-Cytox Enhanced Cell Viability Assay Kit. Experiments were performed in triplicate. Data represent mean ± SD.

Article Snippet: Bovine pulmonary epithelium CPAE (KCLB No. 10209), human lung fibroblast IMR90 (KCLB No. 10186), human NSCLC A549 (KCLB No. 10185), and H1299 (KCLB No. 91299) cell lines were obtained from Korean Cell Line Bank.

Techniques: Incubation, Viability Assay

The effects of rocuronium bromide treatment on the viability of calf pulmonary artery endothelial (CPAE) cells. A, control group; B, 1-μg/mL rocuronium bromide-treated group; C, 5-μg/mL rocuronium bromide-treated group; D, 10-μg/mL rocuronium bromide-treated group; E, 50-μg/mL rocuronium bromide-treated group; F, 100-μg/mL rocuronium bromidetreated group; G, 500-μg/mL rocuronium bromide-treated group; H, 1,000-μg/mL rocuronium bromide-treated group. The results are presented as mean±standard error of mean. * P<0.05 compared to the control group.

Journal: International Neurourology Journal

Article Title: Rocuronium Bromide Inhibits Inflammation and Pain by Suppressing Nitric Oxide Production and Enhancing Prostaglandin E 2 Synthesis in Endothelial Cells

doi: 10.5213/inj.1632796.398

Figure Lengend Snippet: The effects of rocuronium bromide treatment on the viability of calf pulmonary artery endothelial (CPAE) cells. A, control group; B, 1-μg/mL rocuronium bromide-treated group; C, 5-μg/mL rocuronium bromide-treated group; D, 10-μg/mL rocuronium bromide-treated group; E, 50-μg/mL rocuronium bromide-treated group; F, 100-μg/mL rocuronium bromidetreated group; G, 500-μg/mL rocuronium bromide-treated group; H, 1,000-μg/mL rocuronium bromide-treated group. The results are presented as mean±standard error of mean. * P<0.05 compared to the control group.

Article Snippet: CPAE cells were purchased from the Korean Cell Line Bank (KCLB, Seoul, Korea).

Techniques: Control

The effects of rocuronium bromide treatment on cyclooxygenase-1 (COX-1) in calf pulmonary artery endothelial (CPAE) cells. A, Control group; B, 10-μg/mL rocuronium bromide-treated group; C, 100-μg/mL rocuronium bromide-treated group; D, 1,000-μg/mL rocuronium bromide-treated group; E, 2-μg/mL lipopolysaccharide-treated group. The results are presented as mean±standard error of mean.

Journal: International Neurourology Journal

Article Title: Rocuronium Bromide Inhibits Inflammation and Pain by Suppressing Nitric Oxide Production and Enhancing Prostaglandin E 2 Synthesis in Endothelial Cells

doi: 10.5213/inj.1632796.398

Figure Lengend Snippet: The effects of rocuronium bromide treatment on cyclooxygenase-1 (COX-1) in calf pulmonary artery endothelial (CPAE) cells. A, Control group; B, 10-μg/mL rocuronium bromide-treated group; C, 100-μg/mL rocuronium bromide-treated group; D, 1,000-μg/mL rocuronium bromide-treated group; E, 2-μg/mL lipopolysaccharide-treated group. The results are presented as mean±standard error of mean.

Article Snippet: CPAE cells were purchased from the Korean Cell Line Bank (KCLB, Seoul, Korea).

Techniques: Control

The effects of rocuronium bromide treatment on cyclooxygenase-2 (COX-2) in calf pulmonary artery endothelial (CPAE) cells. A, Control group; B, 10-μg/mL rocuronium bromide-treated group; C, 100-μg/mL rocuronium bromide-treated group; D, 1,000-μg/mL rocuronium bromide-treated group; E, 2-μg/mL lipopolysaccharide-treated group. The results are presented as mean±standard error of mean. * P<0.05 compared to the control group.

Journal: International Neurourology Journal

Article Title: Rocuronium Bromide Inhibits Inflammation and Pain by Suppressing Nitric Oxide Production and Enhancing Prostaglandin E 2 Synthesis in Endothelial Cells

doi: 10.5213/inj.1632796.398

Figure Lengend Snippet: The effects of rocuronium bromide treatment on cyclooxygenase-2 (COX-2) in calf pulmonary artery endothelial (CPAE) cells. A, Control group; B, 10-μg/mL rocuronium bromide-treated group; C, 100-μg/mL rocuronium bromide-treated group; D, 1,000-μg/mL rocuronium bromide-treated group; E, 2-μg/mL lipopolysaccharide-treated group. The results are presented as mean±standard error of mean. * P<0.05 compared to the control group.

Article Snippet: CPAE cells were purchased from the Korean Cell Line Bank (KCLB, Seoul, Korea).

Techniques: Control

The effects of rocuronium bromide treatment on inducible nitric oxide synthase (iNOS) in calf pulmonary artery endothelial (CPAE) cells. A, Control group; B, 10-μg/mL rocuronium bromide-treated group; C, 100-μg/mL rocuronium bromide-treated group; D, 1,000-μg/mL rocuronium bromidetreated group; E, 2-μg/mL lipopolysaccharide-treated group. The results are presented as mean±standard error of mean. * P<0.05 compared to the control group

Journal: International Neurourology Journal

Article Title: Rocuronium Bromide Inhibits Inflammation and Pain by Suppressing Nitric Oxide Production and Enhancing Prostaglandin E 2 Synthesis in Endothelial Cells

doi: 10.5213/inj.1632796.398

Figure Lengend Snippet: The effects of rocuronium bromide treatment on inducible nitric oxide synthase (iNOS) in calf pulmonary artery endothelial (CPAE) cells. A, Control group; B, 10-μg/mL rocuronium bromide-treated group; C, 100-μg/mL rocuronium bromide-treated group; D, 1,000-μg/mL rocuronium bromidetreated group; E, 2-μg/mL lipopolysaccharide-treated group. The results are presented as mean±standard error of mean. * P<0.05 compared to the control group

Article Snippet: CPAE cells were purchased from the Korean Cell Line Bank (KCLB, Seoul, Korea).

Techniques: Control

The effects of rocuronium bromide treatment on endothelial nitric oxide synthase (eNOS) in calf pulmonary artery endothelial (CPAE) cells. A, Control group; B, 10-μg/mL rocuronium bromide-treated group; C, 100-μg/mL rocuronium bromide-treated group; D, 1,000-μg/mL rocuronium bromidetreated group; E, 2-μg/mL lipopolysaccharide-treated group. The results are presented as mean±standard error of mean. * P<0.05 compared to the control group.

Journal: International Neurourology Journal

Article Title: Rocuronium Bromide Inhibits Inflammation and Pain by Suppressing Nitric Oxide Production and Enhancing Prostaglandin E 2 Synthesis in Endothelial Cells

doi: 10.5213/inj.1632796.398

Figure Lengend Snippet: The effects of rocuronium bromide treatment on endothelial nitric oxide synthase (eNOS) in calf pulmonary artery endothelial (CPAE) cells. A, Control group; B, 10-μg/mL rocuronium bromide-treated group; C, 100-μg/mL rocuronium bromide-treated group; D, 1,000-μg/mL rocuronium bromidetreated group; E, 2-μg/mL lipopolysaccharide-treated group. The results are presented as mean±standard error of mean. * P<0.05 compared to the control group.

Article Snippet: CPAE cells were purchased from the Korean Cell Line Bank (KCLB, Seoul, Korea).

Techniques: Control

Measurement of nitric oxide (NO) production in calf pulmonary artery endothelial (CPAE) cells. A, Control group; B, 10-μg/mL rocuronium bromide-treated group; C, 100-μg/mL rocuronium bromide-treated group; D, 1,000-μg/mL rocuronium bromide-treated group; E, 2-μg/mL lipopolysaccharidetreated group. The results are presented as mean±standard error of mean. * P<0.05 compared to the control group.

Journal: International Neurourology Journal

Article Title: Rocuronium Bromide Inhibits Inflammation and Pain by Suppressing Nitric Oxide Production and Enhancing Prostaglandin E 2 Synthesis in Endothelial Cells

doi: 10.5213/inj.1632796.398

Figure Lengend Snippet: Measurement of nitric oxide (NO) production in calf pulmonary artery endothelial (CPAE) cells. A, Control group; B, 10-μg/mL rocuronium bromide-treated group; C, 100-μg/mL rocuronium bromide-treated group; D, 1,000-μg/mL rocuronium bromide-treated group; E, 2-μg/mL lipopolysaccharidetreated group. The results are presented as mean±standard error of mean. * P<0.05 compared to the control group.

Article Snippet: CPAE cells were purchased from the Korean Cell Line Bank (KCLB, Seoul, Korea).

Techniques: Control

Measurement of prostaglandin E 2 (PGE 2 ) synthesis in calf pulmonary artery endothelial (CPAE) cells. A, Control group; B, 10-μg/mL rocuronium bromide-treated group; C, 100-μg/mL rocuronium bromide-treated group; D, 1,000-μg/mL rocuronium bromide-treated group; E, 2-μg/mL lipopolysaccharidetreated group. The results are presented as mean±standard error of mean. * P<0.05 compared to the control group.

Journal: International Neurourology Journal

Article Title: Rocuronium Bromide Inhibits Inflammation and Pain by Suppressing Nitric Oxide Production and Enhancing Prostaglandin E 2 Synthesis in Endothelial Cells

doi: 10.5213/inj.1632796.398

Figure Lengend Snippet: Measurement of prostaglandin E 2 (PGE 2 ) synthesis in calf pulmonary artery endothelial (CPAE) cells. A, Control group; B, 10-μg/mL rocuronium bromide-treated group; C, 100-μg/mL rocuronium bromide-treated group; D, 1,000-μg/mL rocuronium bromide-treated group; E, 2-μg/mL lipopolysaccharidetreated group. The results are presented as mean±standard error of mean. * P<0.05 compared to the control group.

Article Snippet: CPAE cells were purchased from the Korean Cell Line Bank (KCLB, Seoul, Korea).

Techniques: Control

Gefitinib inhibits proliferation of human NSCLC A549 cells. (A) A549, (B) CPAE and (C) IMR90 cells were treated with gefitinib for 24–72 h. After incubation, cell viability was measured by the CCK-8 assay. (D) A549 cells were incubated with gefitinib for 72 h, and then the populations of apoptotic and dead cells were analyzed using the MUSE Cell Analyzer. Experiments were performed in triplicate. Data represent mean ± SD. * p < 0.05, ** p < 0.01 versus untreated control.

Journal: Frontiers in Pharmacology

Article Title: Cyclic RGD Pentapeptide Cilengitide Enhances Efficacy of Gefitinib on TGF-β1-Induced Epithelial-to-Mesenchymal Transition and Invasion in Human Non-Small Cell Lung Cancer Cells

doi: 10.3389/fphar.2021.639095

Figure Lengend Snippet: Gefitinib inhibits proliferation of human NSCLC A549 cells. (A) A549, (B) CPAE and (C) IMR90 cells were treated with gefitinib for 24–72 h. After incubation, cell viability was measured by the CCK-8 assay. (D) A549 cells were incubated with gefitinib for 72 h, and then the populations of apoptotic and dead cells were analyzed using the MUSE Cell Analyzer. Experiments were performed in triplicate. Data represent mean ± SD. * p < 0.05, ** p < 0.01 versus untreated control.

Article Snippet: Human lung fibroblast IMR90 (KCLB No. 10186), bovine pulmonary epithelium CPAE (KCLB No. 10209), NSCLC A549 (KCLB No. 10185), H1650 (KCLB No. 91650), H1299 (KCLB No. 91299), and H358 (KCLB No. 25807) cell lines were obtained from Korean Cell Line Bank.

Techniques: Incubation, CCK-8 Assay, Control

Cilengitide inhibits proliferation of human NSCLC A549 cells. (A) Chemical structure of cilengitide. (B) IMR90, (C) CPAE and (D) A549 cells were treated with cilengitide for 24–72 h. After incubation, cell viability was measured by the CCK-8 assay. Experiments were performed in triplicate. Data represent mean ± SD. * p < 0.05, ** p < 0.01 vs. untreated control.

Journal: Frontiers in Pharmacology

Article Title: Cyclic RGD Pentapeptide Cilengitide Enhances Efficacy of Gefitinib on TGF-β1-Induced Epithelial-to-Mesenchymal Transition and Invasion in Human Non-Small Cell Lung Cancer Cells

doi: 10.3389/fphar.2021.639095

Figure Lengend Snippet: Cilengitide inhibits proliferation of human NSCLC A549 cells. (A) Chemical structure of cilengitide. (B) IMR90, (C) CPAE and (D) A549 cells were treated with cilengitide for 24–72 h. After incubation, cell viability was measured by the CCK-8 assay. Experiments were performed in triplicate. Data represent mean ± SD. * p < 0.05, ** p < 0.01 vs. untreated control.

Article Snippet: Human lung fibroblast IMR90 (KCLB No. 10186), bovine pulmonary epithelium CPAE (KCLB No. 10209), NSCLC A549 (KCLB No. 10185), H1650 (KCLB No. 91650), H1299 (KCLB No. 91299), and H358 (KCLB No. 25807) cell lines were obtained from Korean Cell Line Bank.

Techniques: Incubation, CCK-8 Assay, Control