cd13 Search Results


anti mouse aminopeptidase n cd13  (R&D Systems)
93
R&D Systems anti mouse aminopeptidase n cd13
VEGF‐E potently improves vascular density while preserving functionality. (a) Representative fluorescence images of CD31 and <t>CD13</t> immunolabeling at the injury site in vehicle (VEH)‐, VEGF‐E‐, and VEGF‐A‐treated mice 4 days after stroke. (b) Analysis of the density of CD31 + microvessels at the injury site. (c) Analysis of the density of perivascular CD13 + cells at the injury site. (d) Analysis of CD31 and CD13 colocalization at the injury site. (e) Representative laser Speckle contrast imaging (LSCI) in VEH‐, VEGF‐E‐, and VEGF‐A‐treated mice at Day 4 after stroke presented as ipsilateral/contralateral ratio of blood flow at Day 4 over 5 min after stroke. Data are boxplot with min/max or stacked histogram ( n = 5–7 animals/group). **** p < 0.0001 compared to control (b, c, one‐way ANOVA). Abbreviations: ANOVA, analysis of variance; IF, immunofluorescence; VEGF, vascular endothelial growth factor; VEH, vehicle.
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anti ampn af3815 antibodies  (R&D Systems)
93
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VEGF‐E potently improves vascular density while preserving functionality. (a) Representative fluorescence images of CD31 and <t>CD13</t> immunolabeling at the injury site in vehicle (VEH)‐, VEGF‐E‐, and VEGF‐A‐treated mice 4 days after stroke. (b) Analysis of the density of CD31 + microvessels at the injury site. (c) Analysis of the density of perivascular CD13 + cells at the injury site. (d) Analysis of CD31 and CD13 colocalization at the injury site. (e) Representative laser Speckle contrast imaging (LSCI) in VEH‐, VEGF‐E‐, and VEGF‐A‐treated mice at Day 4 after stroke presented as ipsilateral/contralateral ratio of blood flow at Day 4 over 5 min after stroke. Data are boxplot with min/max or stacked histogram ( n = 5–7 animals/group). **** p < 0.0001 compared to control (b, c, one‐way ANOVA). Abbreviations: ANOVA, analysis of variance; IF, immunofluorescence; VEGF, vascular endothelial growth factor; VEH, vehicle.
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goat anti mouse aminopeptidase n anpep  (R&D Systems)
94
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VEGF‐E potently improves vascular density while preserving functionality. (a) Representative fluorescence images of CD31 and <t>CD13</t> immunolabeling at the injury site in vehicle (VEH)‐, VEGF‐E‐, and VEGF‐A‐treated mice 4 days after stroke. (b) Analysis of the density of CD31 + microvessels at the injury site. (c) Analysis of the density of perivascular CD13 + cells at the injury site. (d) Analysis of CD31 and CD13 colocalization at the injury site. (e) Representative laser Speckle contrast imaging (LSCI) in VEH‐, VEGF‐E‐, and VEGF‐A‐treated mice at Day 4 after stroke presented as ipsilateral/contralateral ratio of blood flow at Day 4 over 5 min after stroke. Data are boxplot with min/max or stacked histogram ( n = 5–7 animals/group). **** p < 0.0001 compared to control (b, c, one‐way ANOVA). Abbreviations: ANOVA, analysis of variance; IF, immunofluorescence; VEGF, vascular endothelial growth factor; VEH, vehicle.
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af2335 r d systems macrophage  (R&D Systems)
96
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VEGF‐E potently improves vascular density while preserving functionality. (a) Representative fluorescence images of CD31 and <t>CD13</t> immunolabeling at the injury site in vehicle (VEH)‐, VEGF‐E‐, and VEGF‐A‐treated mice 4 days after stroke. (b) Analysis of the density of CD31 + microvessels at the injury site. (c) Analysis of the density of perivascular CD13 + cells at the injury site. (d) Analysis of CD31 and CD13 colocalization at the injury site. (e) Representative laser Speckle contrast imaging (LSCI) in VEH‐, VEGF‐E‐, and VEGF‐A‐treated mice at Day 4 after stroke presented as ipsilateral/contralateral ratio of blood flow at Day 4 over 5 min after stroke. Data are boxplot with min/max or stacked histogram ( n = 5–7 animals/group). **** p < 0.0001 compared to control (b, c, one‐way ANOVA). Abbreviations: ANOVA, analysis of variance; IF, immunofluorescence; VEGF, vascular endothelial growth factor; VEH, vehicle.
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human aminopeptidase n cd13 duoset elisa  (R&D Systems)
94
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VEGF‐E potently improves vascular density while preserving functionality. (a) Representative fluorescence images of CD31 and <t>CD13</t> immunolabeling at the injury site in vehicle (VEH)‐, VEGF‐E‐, and VEGF‐A‐treated mice 4 days after stroke. (b) Analysis of the density of CD31 + microvessels at the injury site. (c) Analysis of the density of perivascular CD13 + cells at the injury site. (d) Analysis of CD31 and CD13 colocalization at the injury site. (e) Representative laser Speckle contrast imaging (LSCI) in VEH‐, VEGF‐E‐, and VEGF‐A‐treated mice at Day 4 after stroke presented as ipsilateral/contralateral ratio of blood flow at Day 4 over 5 min after stroke. Data are boxplot with min/max or stacked histogram ( n = 5–7 animals/group). **** p < 0.0001 compared to control (b, c, one‐way ANOVA). Abbreviations: ANOVA, analysis of variance; IF, immunofluorescence; VEGF, vascular endothelial growth factor; VEH, vehicle.
Human Aminopeptidase N Cd13 Duoset Elisa, supplied by R&D Systems, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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bio rad mca2183el coliv  (Bio-Rad)
93
Bio-Rad bio rad mca2183el coliv
VEGF‐E potently improves vascular density while preserving functionality. (a) Representative fluorescence images of CD31 and <t>CD13</t> immunolabeling at the injury site in vehicle (VEH)‐, VEGF‐E‐, and VEGF‐A‐treated mice 4 days after stroke. (b) Analysis of the density of CD31 + microvessels at the injury site. (c) Analysis of the density of perivascular CD13 + cells at the injury site. (d) Analysis of CD31 and CD13 colocalization at the injury site. (e) Representative laser Speckle contrast imaging (LSCI) in VEH‐, VEGF‐E‐, and VEGF‐A‐treated mice at Day 4 after stroke presented as ipsilateral/contralateral ratio of blood flow at Day 4 over 5 min after stroke. Data are boxplot with min/max or stacked histogram ( n = 5–7 animals/group). **** p < 0.0001 compared to control (b, c, one‐way ANOVA). Abbreviations: ANOVA, analysis of variance; IF, immunofluorescence; VEGF, vascular endothelial growth factor; VEH, vehicle.
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anti cd13  (Novus Biologicals)
92
Novus Biologicals anti cd13

Anti Cd13, supplied by Novus Biologicals, used in various techniques. Bioz Stars score: 92/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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cd13 hapn  (R&D Systems)
88
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ihc anti cd13 antibody proteintech 14553 1 ap  (Proteintech)
93
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rmapn  (R&D Systems)
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recombinant human aminopeptidase n  (R&D Systems)
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c cst brookfield  (Cell Signaling Technology Inc)
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Image Search Results


VEGF‐E potently improves vascular density while preserving functionality. (a) Representative fluorescence images of CD31 and CD13 immunolabeling at the injury site in vehicle (VEH)‐, VEGF‐E‐, and VEGF‐A‐treated mice 4 days after stroke. (b) Analysis of the density of CD31 + microvessels at the injury site. (c) Analysis of the density of perivascular CD13 + cells at the injury site. (d) Analysis of CD31 and CD13 colocalization at the injury site. (e) Representative laser Speckle contrast imaging (LSCI) in VEH‐, VEGF‐E‐, and VEGF‐A‐treated mice at Day 4 after stroke presented as ipsilateral/contralateral ratio of blood flow at Day 4 over 5 min after stroke. Data are boxplot with min/max or stacked histogram ( n = 5–7 animals/group). **** p < 0.0001 compared to control (b, c, one‐way ANOVA). Abbreviations: ANOVA, analysis of variance; IF, immunofluorescence; VEGF, vascular endothelial growth factor; VEH, vehicle.

Journal: The European Journal of Neuroscience

Article Title: VEGF‐E Attenuates Injury After Ischemic Stroke by Promoting Reparative Revascularization

doi: 10.1111/ejn.70114

Figure Lengend Snippet: VEGF‐E potently improves vascular density while preserving functionality. (a) Representative fluorescence images of CD31 and CD13 immunolabeling at the injury site in vehicle (VEH)‐, VEGF‐E‐, and VEGF‐A‐treated mice 4 days after stroke. (b) Analysis of the density of CD31 + microvessels at the injury site. (c) Analysis of the density of perivascular CD13 + cells at the injury site. (d) Analysis of CD31 and CD13 colocalization at the injury site. (e) Representative laser Speckle contrast imaging (LSCI) in VEH‐, VEGF‐E‐, and VEGF‐A‐treated mice at Day 4 after stroke presented as ipsilateral/contralateral ratio of blood flow at Day 4 over 5 min after stroke. Data are boxplot with min/max or stacked histogram ( n = 5–7 animals/group). **** p < 0.0001 compared to control (b, c, one‐way ANOVA). Abbreviations: ANOVA, analysis of variance; IF, immunofluorescence; VEGF, vascular endothelial growth factor; VEH, vehicle.

Article Snippet: The following primary antibodies were used: rat anti‐mouse cluster of differentiation (CD31) (1/500; BD Biosciences, ON, Canada; 550274), goat anti‐mouse aminopeptidase N (CD13) (1/250; R&D systems), rat anti‐mouse glial fibrillary acidic protein (GFAP) (1/500; Invitrogen, MA, USA; 13‐0300), rabbit anti‐mouse ionized calcium binding adaptor molecule (IBA1) (1/500; WAKO, ON, Canada; 019‐19741), goat anti‐rat CD45 (1/500; BD Biosciences; 553076), rabbit anti‐mouse PDGFRβ (1/250; Abcam, ON, Canada; ab32570), rat anti‐mouse endoglin (CD105) (1/250; Novus Biologicals, ON, Canada; NB100‐77666), and rabbit anti‐mouse aquaporine‐4 (AQP4) (1/500; Cell Signaling Technology, MA, USA; 59678S).

Techniques: Preserving, Fluorescence, Immunolabeling, Imaging, Control, Immunofluorescence

Revascularization is stabilized by VEGF‐E associated with attenuation of neuronal degeneration. (a) Representative fluorescence images of CD31 and PDGFRβ immunolabeling at the injury site in vehicle (VEH)‐ and VEGF‐E‐treated mice 4 days after stroke. (b) Analysis of the density of perivascular PDGFRβ + cells at the injury site 4 days after stroke. (c) Analysis of CD31 and PDGFRβ colocalization at the injury site. (d) Analysis of the number of CD31 + microvascular stalls at the injury site. (e) Representative fluorescence images of CD13 and CD105 immunolabeling at the injury site in VEH‐ and VEGF‐E‐treated mice. (f) Analysis of the density of CD105 + microvessels at the injury site. (g) Analysis of CD105 and CD13 colocalization at the injury site. (h) Representative fluorescence images of FJB + degenerating neurons in the ipsilateral cortex and ipsilateral striatum. Stereological analysis of the density of FJB + degenerating neurons in the (i) ipsilateral cortex and (j) ipsilateral striatum. Data are boxplot with min/max or stacked histogram ( n = 6–7 animals/group). * p < 0.05, ** p < 0.01, *** p < 0.001 compared to control (b, d, f, i, unpaired two‐tailed t ‐test). Abbreviations: IF, immunofluorescence; VEGF, vascular endothelial growth factor; VEH, vehicle.

Journal: The European Journal of Neuroscience

Article Title: VEGF‐E Attenuates Injury After Ischemic Stroke by Promoting Reparative Revascularization

doi: 10.1111/ejn.70114

Figure Lengend Snippet: Revascularization is stabilized by VEGF‐E associated with attenuation of neuronal degeneration. (a) Representative fluorescence images of CD31 and PDGFRβ immunolabeling at the injury site in vehicle (VEH)‐ and VEGF‐E‐treated mice 4 days after stroke. (b) Analysis of the density of perivascular PDGFRβ + cells at the injury site 4 days after stroke. (c) Analysis of CD31 and PDGFRβ colocalization at the injury site. (d) Analysis of the number of CD31 + microvascular stalls at the injury site. (e) Representative fluorescence images of CD13 and CD105 immunolabeling at the injury site in VEH‐ and VEGF‐E‐treated mice. (f) Analysis of the density of CD105 + microvessels at the injury site. (g) Analysis of CD105 and CD13 colocalization at the injury site. (h) Representative fluorescence images of FJB + degenerating neurons in the ipsilateral cortex and ipsilateral striatum. Stereological analysis of the density of FJB + degenerating neurons in the (i) ipsilateral cortex and (j) ipsilateral striatum. Data are boxplot with min/max or stacked histogram ( n = 6–7 animals/group). * p < 0.05, ** p < 0.01, *** p < 0.001 compared to control (b, d, f, i, unpaired two‐tailed t ‐test). Abbreviations: IF, immunofluorescence; VEGF, vascular endothelial growth factor; VEH, vehicle.

Article Snippet: The following primary antibodies were used: rat anti‐mouse cluster of differentiation (CD31) (1/500; BD Biosciences, ON, Canada; 550274), goat anti‐mouse aminopeptidase N (CD13) (1/250; R&D systems), rat anti‐mouse glial fibrillary acidic protein (GFAP) (1/500; Invitrogen, MA, USA; 13‐0300), rabbit anti‐mouse ionized calcium binding adaptor molecule (IBA1) (1/500; WAKO, ON, Canada; 019‐19741), goat anti‐rat CD45 (1/500; BD Biosciences; 553076), rabbit anti‐mouse PDGFRβ (1/250; Abcam, ON, Canada; ab32570), rat anti‐mouse endoglin (CD105) (1/250; Novus Biologicals, ON, Canada; NB100‐77666), and rabbit anti‐mouse aquaporine‐4 (AQP4) (1/500; Cell Signaling Technology, MA, USA; 59678S).

Techniques: Fluorescence, Immunolabeling, Control, Two Tailed Test, Immunofluorescence

Journal: eLife

Article Title: Liquid-crystal organization of liver tissue

doi: 10.7554/eLife.44860

Figure Lengend Snippet:

Article Snippet: Antibody , anti-CD13 (rat monoclonal) , Novus , NB100−64843/RRID: AB_959651 , (1:500).

Techniques: Sequencing, Luciferase, Knockdown, Software