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rabbit polyclonal antibody against clostridium perfringens type d  (Bioss)
90
Bioss rabbit polyclonal antibody against clostridium perfringens type d
Immunohistochemistry of HSV-1 proteins in brain sections from ten AD patients and PCR analysis of HSV-1 DNA. Entorhinal cortex/hipoccampus (ERH) sections from ten AD patients. Panel A: samples were immunostained with mouse monoclonal antibody (1:50) against HSV ICP0 (green), and rabbit <t>polyclonal</t> antibody (1:100) against C. albicans (red). The different patients are numbered from AD1 to AD10 and one field is shown for each patient. In addition, four selected sections are shown at higher magnification below the ten AD patients. Panel B: samples were immunostained with mouse monoclonal antibody (1:50) against HSV-1 ICP5 (green) and rabbit polyclonal antibody (1:100) against C. albicans (red). DAPI staining of nuclei appears in blue. Scale bar as shown in the figure. Panel C: PCR analysis of HSV-1 and β-globin DNA in frozen brain tissue from ten AD patients. Left panel: Nested PCR analysis of ten ERH samples using primers that amplify HSV-1 glycoprotein D gene. The primers employed were HSV-1 FE (forward external) and HSV-1 RE (reverse external) for the first PCR and primers HSV-1 FI (forward internal) and HSV-1 RI (reverse internal) for the second PCR. As positive control, DNA from HSV-1-infected HeLa cells was used. Right panel: PCR analysis using β-globin oligonucleotide primers. As positive control, DNA extracted from HeLa cells was used. Control PCR: PCR without DNA. DNA markers are indicated on the left.
Rabbit Polyclonal Antibody Against Clostridium Perfringens Type D, supplied by Bioss, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Immunohistochemistry of HSV-1 proteins in brain sections from ten AD patients and PCR analysis of HSV-1 DNA. Entorhinal cortex/hipoccampus (ERH) sections from ten AD patients. Panel A: samples were immunostained with mouse monoclonal antibody (1:50) against HSV ICP0 (green), and rabbit polyclonal antibody (1:100) against C. albicans (red). The different patients are numbered from AD1 to AD10 and one field is shown for each patient. In addition, four selected sections are shown at higher magnification below the ten AD patients. Panel B: samples were immunostained with mouse monoclonal antibody (1:50) against HSV-1 ICP5 (green) and rabbit polyclonal antibody (1:100) against C. albicans (red). DAPI staining of nuclei appears in blue. Scale bar as shown in the figure. Panel C: PCR analysis of HSV-1 and β-globin DNA in frozen brain tissue from ten AD patients. Left panel: Nested PCR analysis of ten ERH samples using primers that amplify HSV-1 glycoprotein D gene. The primers employed were HSV-1 FE (forward external) and HSV-1 RE (reverse external) for the first PCR and primers HSV-1 FI (forward internal) and HSV-1 RI (reverse internal) for the second PCR. As positive control, DNA from HSV-1-infected HeLa cells was used. Right panel: PCR analysis using β-globin oligonucleotide primers. As positive control, DNA extracted from HeLa cells was used. Control PCR: PCR without DNA. DNA markers are indicated on the left.

Journal: Scientific Reports

Article Title: Polymicrobial Infections In Brain Tissue From Alzheimer’s Disease Patients

doi: 10.1038/s41598-017-05903-y

Figure Lengend Snippet: Immunohistochemistry of HSV-1 proteins in brain sections from ten AD patients and PCR analysis of HSV-1 DNA. Entorhinal cortex/hipoccampus (ERH) sections from ten AD patients. Panel A: samples were immunostained with mouse monoclonal antibody (1:50) against HSV ICP0 (green), and rabbit polyclonal antibody (1:100) against C. albicans (red). The different patients are numbered from AD1 to AD10 and one field is shown for each patient. In addition, four selected sections are shown at higher magnification below the ten AD patients. Panel B: samples were immunostained with mouse monoclonal antibody (1:50) against HSV-1 ICP5 (green) and rabbit polyclonal antibody (1:100) against C. albicans (red). DAPI staining of nuclei appears in blue. Scale bar as shown in the figure. Panel C: PCR analysis of HSV-1 and β-globin DNA in frozen brain tissue from ten AD patients. Left panel: Nested PCR analysis of ten ERH samples using primers that amplify HSV-1 glycoprotein D gene. The primers employed were HSV-1 FE (forward external) and HSV-1 RE (reverse external) for the first PCR and primers HSV-1 FI (forward internal) and HSV-1 RI (reverse internal) for the second PCR. As positive control, DNA from HSV-1-infected HeLa cells was used. Right panel: PCR analysis using β-globin oligonucleotide primers. As positive control, DNA extracted from HeLa cells was used. Control PCR: PCR without DNA. DNA markers are indicated on the left.

Article Snippet: The following antibodies were purchased: mouse monoclonal antibody against HSV-1 ICP0, used at 1:50 dilution; mouse monoclonal antibody against HSV-1/2 ICP5 major capsid protein, used at 1:50 dilution (both from Santa Cruz Biotechnology, Santa Cruz, CA); rabbit polyclonal antibody against Borrelia burgdorferi (Genetex, Irvine, CA), used at 1:50 dilution; mouse monoclonal antibody against Borrelia burgdorferi (Abcam, Cambridge, UK), used at 1:10 dilution; rabbit polyclonal antibody against C. pneumoniae , which immunoreacts with the major outer porin (Biorbyt, Cambridge, UK), used at 1:20 dilution; mouse monoclonal antibody against Chlamydia (Abcam), used at 1:10 dilution; rabbit polyclonal antibody against Clostridium perfringens type D (Bioss Antibodies, Woburn, MA), used at 1:20 dilution; mouse monoclonal antibody against peptidoglycan (Thermo Fisher Scientific, Waltham, MA), used at 1:20 dilution.

Techniques: Immunohistochemistry, Staining, Nested PCR, Positive Control, Infection

Detection of Borrelia proteins and DNA in ERH samples from ten AD patients. PCR assay of B. burgdorferi DNA. Panel A: ERH sections were immunostained with rabbit polyclonal antibody (1:50) against B. burgdorferi (green) and rat polyclonal antibody (1:20) against T. viride (red). Panel B: ERH sections were immunostained with mouse monoclonal antibody (1:10) against B. burgdorferi (green) and rabbit polyclonal antibody (1:100 dilution) against C. albicans (red). DAPI staining of nuclei appears in blue. Scale bar as shown in the figure. Panel C: PCR analysis of Borrelia DNA in frozen brain tissue from ten AD patients. Nested PCR analysis of ten ERH samples using Borr primers to amplify flagellin gene. The primers employed were Borr FE–Borr RE for the first PCR and primers Borr FI–Borr RI for the second PCR. As positive control, DNA extracted from B. burgdorferi was used. Control PCR: PCR without DNA. DNA markers are indicated on the left.

Journal: Scientific Reports

Article Title: Polymicrobial Infections In Brain Tissue From Alzheimer’s Disease Patients

doi: 10.1038/s41598-017-05903-y

Figure Lengend Snippet: Detection of Borrelia proteins and DNA in ERH samples from ten AD patients. PCR assay of B. burgdorferi DNA. Panel A: ERH sections were immunostained with rabbit polyclonal antibody (1:50) against B. burgdorferi (green) and rat polyclonal antibody (1:20) against T. viride (red). Panel B: ERH sections were immunostained with mouse monoclonal antibody (1:10) against B. burgdorferi (green) and rabbit polyclonal antibody (1:100 dilution) against C. albicans (red). DAPI staining of nuclei appears in blue. Scale bar as shown in the figure. Panel C: PCR analysis of Borrelia DNA in frozen brain tissue from ten AD patients. Nested PCR analysis of ten ERH samples using Borr primers to amplify flagellin gene. The primers employed were Borr FE–Borr RE for the first PCR and primers Borr FI–Borr RI for the second PCR. As positive control, DNA extracted from B. burgdorferi was used. Control PCR: PCR without DNA. DNA markers are indicated on the left.

Article Snippet: The following antibodies were purchased: mouse monoclonal antibody against HSV-1 ICP0, used at 1:50 dilution; mouse monoclonal antibody against HSV-1/2 ICP5 major capsid protein, used at 1:50 dilution (both from Santa Cruz Biotechnology, Santa Cruz, CA); rabbit polyclonal antibody against Borrelia burgdorferi (Genetex, Irvine, CA), used at 1:50 dilution; mouse monoclonal antibody against Borrelia burgdorferi (Abcam, Cambridge, UK), used at 1:10 dilution; rabbit polyclonal antibody against C. pneumoniae , which immunoreacts with the major outer porin (Biorbyt, Cambridge, UK), used at 1:20 dilution; mouse monoclonal antibody against Chlamydia (Abcam), used at 1:10 dilution; rabbit polyclonal antibody against Clostridium perfringens type D (Bioss Antibodies, Woburn, MA), used at 1:20 dilution; mouse monoclonal antibody against peptidoglycan (Thermo Fisher Scientific, Waltham, MA), used at 1:20 dilution.

Techniques: Staining, Nested PCR, Positive Control

Immunohistochemistry and PCR analysis to detect C. pneumoniae proteins and DNA in ERH samples from AD patients. Panel A: ERH sections were immunostained with rabbit polyclonal antibody (1:20) against C. pneumoniae (green) and rat polyclonal antibody (1:20) against T. viride (red). Panel B: samples were immunostained with mouse monoclonal antibody (1:10) against C. pneumoniae (green) and rabbit polyclonal antibody (1:100) against C. albicans (red). DAPI staining of nuclei appears in blue. Scale bar as shown in the figure. Panel C: PCR analysis of C. pneumoniae DNA in frozen brain tissue from ten AD patients. PCR analysis of ten ERH samples using primers Clam to amplify MOMP gene. The primers employed were Clam FE–Clam RE for the first PCR and primers Clam FI–Clam RI for the second PCR assay. As positive control, DNA extracted from C. pneumoniae was used. Control PCR: PCR without DNA. DNA markers are indicated on the left.

Journal: Scientific Reports

Article Title: Polymicrobial Infections In Brain Tissue From Alzheimer’s Disease Patients

doi: 10.1038/s41598-017-05903-y

Figure Lengend Snippet: Immunohistochemistry and PCR analysis to detect C. pneumoniae proteins and DNA in ERH samples from AD patients. Panel A: ERH sections were immunostained with rabbit polyclonal antibody (1:20) against C. pneumoniae (green) and rat polyclonal antibody (1:20) against T. viride (red). Panel B: samples were immunostained with mouse monoclonal antibody (1:10) against C. pneumoniae (green) and rabbit polyclonal antibody (1:100) against C. albicans (red). DAPI staining of nuclei appears in blue. Scale bar as shown in the figure. Panel C: PCR analysis of C. pneumoniae DNA in frozen brain tissue from ten AD patients. PCR analysis of ten ERH samples using primers Clam to amplify MOMP gene. The primers employed were Clam FE–Clam RE for the first PCR and primers Clam FI–Clam RI for the second PCR assay. As positive control, DNA extracted from C. pneumoniae was used. Control PCR: PCR without DNA. DNA markers are indicated on the left.

Article Snippet: The following antibodies were purchased: mouse monoclonal antibody against HSV-1 ICP0, used at 1:50 dilution; mouse monoclonal antibody against HSV-1/2 ICP5 major capsid protein, used at 1:50 dilution (both from Santa Cruz Biotechnology, Santa Cruz, CA); rabbit polyclonal antibody against Borrelia burgdorferi (Genetex, Irvine, CA), used at 1:50 dilution; mouse monoclonal antibody against Borrelia burgdorferi (Abcam, Cambridge, UK), used at 1:10 dilution; rabbit polyclonal antibody against C. pneumoniae , which immunoreacts with the major outer porin (Biorbyt, Cambridge, UK), used at 1:20 dilution; mouse monoclonal antibody against Chlamydia (Abcam), used at 1:10 dilution; rabbit polyclonal antibody against Clostridium perfringens type D (Bioss Antibodies, Woburn, MA), used at 1:20 dilution; mouse monoclonal antibody against peptidoglycan (Thermo Fisher Scientific, Waltham, MA), used at 1:20 dilution.

Techniques: Immunohistochemistry, Staining, Positive Control

Immunohistochemistry of bacterial and fungal proteins in ERH sections from AD patients. Panel A: samples were immunostained with rabbit polyclonal antibody (1:20) against C. pneumoniae (green) and rat polyclonal antibody (1:20) against T. viride (red). Panel B: samples were immunostained with rabbit polyclonal antibody (1:20) against C. perfringens (green) and rat polyclonal antibody (1:20) against T. viride (red). DAPI staining of nuclei appears in blue. Scale bar as shown in the figure.

Journal: Scientific Reports

Article Title: Polymicrobial Infections In Brain Tissue From Alzheimer’s Disease Patients

doi: 10.1038/s41598-017-05903-y

Figure Lengend Snippet: Immunohistochemistry of bacterial and fungal proteins in ERH sections from AD patients. Panel A: samples were immunostained with rabbit polyclonal antibody (1:20) against C. pneumoniae (green) and rat polyclonal antibody (1:20) against T. viride (red). Panel B: samples were immunostained with rabbit polyclonal antibody (1:20) against C. perfringens (green) and rat polyclonal antibody (1:20) against T. viride (red). DAPI staining of nuclei appears in blue. Scale bar as shown in the figure.

Article Snippet: The following antibodies were purchased: mouse monoclonal antibody against HSV-1 ICP0, used at 1:50 dilution; mouse monoclonal antibody against HSV-1/2 ICP5 major capsid protein, used at 1:50 dilution (both from Santa Cruz Biotechnology, Santa Cruz, CA); rabbit polyclonal antibody against Borrelia burgdorferi (Genetex, Irvine, CA), used at 1:50 dilution; mouse monoclonal antibody against Borrelia burgdorferi (Abcam, Cambridge, UK), used at 1:10 dilution; rabbit polyclonal antibody against C. pneumoniae , which immunoreacts with the major outer porin (Biorbyt, Cambridge, UK), used at 1:20 dilution; mouse monoclonal antibody against Chlamydia (Abcam), used at 1:10 dilution; rabbit polyclonal antibody against Clostridium perfringens type D (Bioss Antibodies, Woburn, MA), used at 1:20 dilution; mouse monoclonal antibody against peptidoglycan (Thermo Fisher Scientific, Waltham, MA), used at 1:20 dilution.

Techniques: Immunohistochemistry, Staining

Peptidoglycan in ERH sections from AD patients. ERH samples were processed as described in Materials and Methods. Samples were first immunostained with mouse monoclonal antibody (1:20 dilution) against peptidoglycan (green) and afterwards with rabbit polyclonal antibody (1:50 dilution) against C. albicans (red). DAPI staining of nuclei appears in blue. Scale bar as shown in the figure.

Journal: Scientific Reports

Article Title: Polymicrobial Infections In Brain Tissue From Alzheimer’s Disease Patients

doi: 10.1038/s41598-017-05903-y

Figure Lengend Snippet: Peptidoglycan in ERH sections from AD patients. ERH samples were processed as described in Materials and Methods. Samples were first immunostained with mouse monoclonal antibody (1:20 dilution) against peptidoglycan (green) and afterwards with rabbit polyclonal antibody (1:50 dilution) against C. albicans (red). DAPI staining of nuclei appears in blue. Scale bar as shown in the figure.

Article Snippet: The following antibodies were purchased: mouse monoclonal antibody against HSV-1 ICP0, used at 1:50 dilution; mouse monoclonal antibody against HSV-1/2 ICP5 major capsid protein, used at 1:50 dilution (both from Santa Cruz Biotechnology, Santa Cruz, CA); rabbit polyclonal antibody against Borrelia burgdorferi (Genetex, Irvine, CA), used at 1:50 dilution; mouse monoclonal antibody against Borrelia burgdorferi (Abcam, Cambridge, UK), used at 1:10 dilution; rabbit polyclonal antibody against C. pneumoniae , which immunoreacts with the major outer porin (Biorbyt, Cambridge, UK), used at 1:20 dilution; mouse monoclonal antibody against Chlamydia (Abcam), used at 1:10 dilution; rabbit polyclonal antibody against Clostridium perfringens type D (Bioss Antibodies, Woburn, MA), used at 1:20 dilution; mouse monoclonal antibody against peptidoglycan (Thermo Fisher Scientific, Waltham, MA), used at 1:20 dilution.

Techniques: Staining