apc3 Search Results


antibody against myosin viia  (Boster Bio)
94
Boster Bio antibody against myosin viia
Antibody Against Myosin Viia, supplied by Boster Bio, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/apc3/Anti-MYO7A+Monoclonal+Antibody/pmc12894672-349-6-12
Average 94 stars, based on 1 article reviews
antibody against myosin viia - by Bioz Stars, 2026-10
94/100 stars
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anti cdc27  (Proteintech)
93
Proteintech anti cdc27
Anti Cdc27, supplied by Proteintech, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/apc3/CDC27%3B+APC3+Antibody/10__1016_slash_j__jff__2025__107065-57-4-14
Average 93 stars, based on 1 article reviews
anti cdc27 - by Bioz Stars, 2026-10
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slc25a25  (Proteintech)
91
Proteintech slc25a25
Zn 2+ -mediated mitochondrial dysfunction and increased ROS promote RSL3-induced ferroptosis. A, PANC1 cells were exposed to RSL3 (1 μM) plus CFX (50 μg/ml) for 12 h. Mitochondria and Zn 2+ were stained with Mito-Tracker Green and TSQ, respectively. Scale bar represents 10 μm. B, JC-1 staining was indicated for mitochondrial membrane potential in PANC1 and HeLa cells treated with RSL3 (1 μM) in the presence or absence of CFX (50 μg/ml) for 12 h. JC-1 monomers indicate decreased mitochondrial membrane potential; JC-1 aggregates indicate increased mitochondrial membrane potential. Scale bar represents 200 μm. C, ROS of PANC1 and HeLa cells treated with RSL3 (1 μM), CFX (50 μg/ml), and N -acetylcysteine (NAC, 2 mM) for 12 h. D, mitochondrial ROS of PANC1 and HeLa cells treated with RSL3 (1 μM), CFX (50 μg/ml), and Mito-TEMPO (MT, 20 μM) for 12 h. E, cell viability of the indicated PANC1 and HeLa cells after treatment with RSL3 (1 μM), CFX (50 μg/ml), NAC (2 mM), and MT (20 μM) for 24 h. F, Mitochondrial ROS of PANC1 and HeLa cells treated with RSL3 (1 μM), CFX (50 μg/ml), and TPEN (10 μM) for 12 h. G, Western blot analysis of protein expression in control and <t>SLC25A25</t> -knockdown tumor cells. H, indicated PANC1 cells were exposed to RSL3 (1 μM) plus CFX (50 μg/ml) for 12 h. Mitochondria and Zn 2+ were stained with Mito-Tracker Green and TSQ, respectively. Scale bar represents 10 μm. I, cell viability of the indicated PANC1 and HeLa cells after treatment with RSL3 in the absence or presence of CFX (50 μg/ml) for 24 h. J, mitochondrial ROS of indicated PANC1 and HeLa cells treated with RSL3 (1 μM) and CFX (50 μg/ml) for 12 h. Data are presented as the mean ± SD from at least three independent experiments. p Values were calculated using one-way ANOVA ( C – F ) or two-way ANOVA ( I and J ). CFX, ciprofloxacin; ROS, reactive oxygen species.
Slc25a25, supplied by Proteintech, used in various techniques. Bioz Stars score: 91/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/apc3/SLC25A25+Antibody/pmc12489925-206-30-41
Average 91 stars, based on 1 article reviews
slc25a25 - by Bioz Stars, 2026-10
91/100 stars
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apc3  (Cell Signaling Technology Inc)
93
Cell Signaling Technology Inc apc3
Zn 2+ -mediated mitochondrial dysfunction and increased ROS promote RSL3-induced ferroptosis. A, PANC1 cells were exposed to RSL3 (1 μM) plus CFX (50 μg/ml) for 12 h. Mitochondria and Zn 2+ were stained with Mito-Tracker Green and TSQ, respectively. Scale bar represents 10 μm. B, JC-1 staining was indicated for mitochondrial membrane potential in PANC1 and HeLa cells treated with RSL3 (1 μM) in the presence or absence of CFX (50 μg/ml) for 12 h. JC-1 monomers indicate decreased mitochondrial membrane potential; JC-1 aggregates indicate increased mitochondrial membrane potential. Scale bar represents 200 μm. C, ROS of PANC1 and HeLa cells treated with RSL3 (1 μM), CFX (50 μg/ml), and N -acetylcysteine (NAC, 2 mM) for 12 h. D, mitochondrial ROS of PANC1 and HeLa cells treated with RSL3 (1 μM), CFX (50 μg/ml), and Mito-TEMPO (MT, 20 μM) for 12 h. E, cell viability of the indicated PANC1 and HeLa cells after treatment with RSL3 (1 μM), CFX (50 μg/ml), NAC (2 mM), and MT (20 μM) for 24 h. F, Mitochondrial ROS of PANC1 and HeLa cells treated with RSL3 (1 μM), CFX (50 μg/ml), and TPEN (10 μM) for 12 h. G, Western blot analysis of protein expression in control and <t>SLC25A25</t> -knockdown tumor cells. H, indicated PANC1 cells were exposed to RSL3 (1 μM) plus CFX (50 μg/ml) for 12 h. Mitochondria and Zn 2+ were stained with Mito-Tracker Green and TSQ, respectively. Scale bar represents 10 μm. I, cell viability of the indicated PANC1 and HeLa cells after treatment with RSL3 in the absence or presence of CFX (50 μg/ml) for 24 h. J, mitochondrial ROS of indicated PANC1 and HeLa cells treated with RSL3 (1 μM) and CFX (50 μg/ml) for 12 h. Data are presented as the mean ± SD from at least three independent experiments. p Values were calculated using one-way ANOVA ( C – F ) or two-way ANOVA ( I and J ). CFX, ciprofloxacin; ROS, reactive oxygen species.
Apc3, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/apc3/APC3+Rabbit+mAb/pmc12080931-29-0-6
Average 93 stars, based on 1 article reviews
apc3 - by Bioz Stars, 2026-10
93/100 stars
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cdc27  (ABclonal Biotechnology)
92
ABclonal Biotechnology cdc27
Zn 2+ -mediated mitochondrial dysfunction and increased ROS promote RSL3-induced ferroptosis. A, PANC1 cells were exposed to RSL3 (1 μM) plus CFX (50 μg/ml) for 12 h. Mitochondria and Zn 2+ were stained with Mito-Tracker Green and TSQ, respectively. Scale bar represents 10 μm. B, JC-1 staining was indicated for mitochondrial membrane potential in PANC1 and HeLa cells treated with RSL3 (1 μM) in the presence or absence of CFX (50 μg/ml) for 12 h. JC-1 monomers indicate decreased mitochondrial membrane potential; JC-1 aggregates indicate increased mitochondrial membrane potential. Scale bar represents 200 μm. C, ROS of PANC1 and HeLa cells treated with RSL3 (1 μM), CFX (50 μg/ml), and N -acetylcysteine (NAC, 2 mM) for 12 h. D, mitochondrial ROS of PANC1 and HeLa cells treated with RSL3 (1 μM), CFX (50 μg/ml), and Mito-TEMPO (MT, 20 μM) for 12 h. E, cell viability of the indicated PANC1 and HeLa cells after treatment with RSL3 (1 μM), CFX (50 μg/ml), NAC (2 mM), and MT (20 μM) for 24 h. F, Mitochondrial ROS of PANC1 and HeLa cells treated with RSL3 (1 μM), CFX (50 μg/ml), and TPEN (10 μM) for 12 h. G, Western blot analysis of protein expression in control and <t>SLC25A25</t> -knockdown tumor cells. H, indicated PANC1 cells were exposed to RSL3 (1 μM) plus CFX (50 μg/ml) for 12 h. Mitochondria and Zn 2+ were stained with Mito-Tracker Green and TSQ, respectively. Scale bar represents 10 μm. I, cell viability of the indicated PANC1 and HeLa cells after treatment with RSL3 in the absence or presence of CFX (50 μg/ml) for 24 h. J, mitochondrial ROS of indicated PANC1 and HeLa cells treated with RSL3 (1 μM) and CFX (50 μg/ml) for 12 h. Data are presented as the mean ± SD from at least three independent experiments. p Values were calculated using one-way ANOVA ( C – F ) or two-way ANOVA ( I and J ). CFX, ciprofloxacin; ROS, reactive oxygen species.
Cdc27, supplied by ABclonal Biotechnology, used in various techniques. Bioz Stars score: 92/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/apc3/CDC27+Rabbit+pAb/pm39112518-275-29-32
Average 92 stars, based on 1 article reviews
cdc27 - by Bioz Stars, 2026-10
92/100 stars
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anti thr p 244 apc3  (Rockland Immunochemicals)
85
Rockland Immunochemicals anti thr p 244 apc3
Zn 2+ -mediated mitochondrial dysfunction and increased ROS promote RSL3-induced ferroptosis. A, PANC1 cells were exposed to RSL3 (1 μM) plus CFX (50 μg/ml) for 12 h. Mitochondria and Zn 2+ were stained with Mito-Tracker Green and TSQ, respectively. Scale bar represents 10 μm. B, JC-1 staining was indicated for mitochondrial membrane potential in PANC1 and HeLa cells treated with RSL3 (1 μM) in the presence or absence of CFX (50 μg/ml) for 12 h. JC-1 monomers indicate decreased mitochondrial membrane potential; JC-1 aggregates indicate increased mitochondrial membrane potential. Scale bar represents 200 μm. C, ROS of PANC1 and HeLa cells treated with RSL3 (1 μM), CFX (50 μg/ml), and N -acetylcysteine (NAC, 2 mM) for 12 h. D, mitochondrial ROS of PANC1 and HeLa cells treated with RSL3 (1 μM), CFX (50 μg/ml), and Mito-TEMPO (MT, 20 μM) for 12 h. E, cell viability of the indicated PANC1 and HeLa cells after treatment with RSL3 (1 μM), CFX (50 μg/ml), NAC (2 mM), and MT (20 μM) for 24 h. F, Mitochondrial ROS of PANC1 and HeLa cells treated with RSL3 (1 μM), CFX (50 μg/ml), and TPEN (10 μM) for 12 h. G, Western blot analysis of protein expression in control and <t>SLC25A25</t> -knockdown tumor cells. H, indicated PANC1 cells were exposed to RSL3 (1 μM) plus CFX (50 μg/ml) for 12 h. Mitochondria and Zn 2+ were stained with Mito-Tracker Green and TSQ, respectively. Scale bar represents 10 μm. I, cell viability of the indicated PANC1 and HeLa cells after treatment with RSL3 in the absence or presence of CFX (50 μg/ml) for 24 h. J, mitochondrial ROS of indicated PANC1 and HeLa cells treated with RSL3 (1 μM) and CFX (50 μg/ml) for 12 h. Data are presented as the mean ± SD from at least three independent experiments. p Values were calculated using one-way ANOVA ( C – F ) or two-way ANOVA ( I and J ). CFX, ciprofloxacin; ROS, reactive oxygen species.
Anti Thr P 244 Apc3, supplied by Rockland Immunochemicals, used in various techniques. Bioz Stars score: 85/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/apc3/APC3+Antibody/pmc02878026-190-69-73
Average 85 stars, based on 1 article reviews
anti thr p 244 apc3 - by Bioz Stars, 2026-10
85/100 stars
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apc3 gfp parasite lines  (Proteintech)
88
Proteintech apc3 gfp parasite lines
Zn 2+ -mediated mitochondrial dysfunction and increased ROS promote RSL3-induced ferroptosis. A, PANC1 cells were exposed to RSL3 (1 μM) plus CFX (50 μg/ml) for 12 h. Mitochondria and Zn 2+ were stained with Mito-Tracker Green and TSQ, respectively. Scale bar represents 10 μm. B, JC-1 staining was indicated for mitochondrial membrane potential in PANC1 and HeLa cells treated with RSL3 (1 μM) in the presence or absence of CFX (50 μg/ml) for 12 h. JC-1 monomers indicate decreased mitochondrial membrane potential; JC-1 aggregates indicate increased mitochondrial membrane potential. Scale bar represents 200 μm. C, ROS of PANC1 and HeLa cells treated with RSL3 (1 μM), CFX (50 μg/ml), and N -acetylcysteine (NAC, 2 mM) for 12 h. D, mitochondrial ROS of PANC1 and HeLa cells treated with RSL3 (1 μM), CFX (50 μg/ml), and Mito-TEMPO (MT, 20 μM) for 12 h. E, cell viability of the indicated PANC1 and HeLa cells after treatment with RSL3 (1 μM), CFX (50 μg/ml), NAC (2 mM), and MT (20 μM) for 24 h. F, Mitochondrial ROS of PANC1 and HeLa cells treated with RSL3 (1 μM), CFX (50 μg/ml), and TPEN (10 μM) for 12 h. G, Western blot analysis of protein expression in control and <t>SLC25A25</t> -knockdown tumor cells. H, indicated PANC1 cells were exposed to RSL3 (1 μM) plus CFX (50 μg/ml) for 12 h. Mitochondria and Zn 2+ were stained with Mito-Tracker Green and TSQ, respectively. Scale bar represents 10 μm. I, cell viability of the indicated PANC1 and HeLa cells after treatment with RSL3 in the absence or presence of CFX (50 μg/ml) for 24 h. J, mitochondrial ROS of indicated PANC1 and HeLa cells treated with RSL3 (1 μM) and CFX (50 μg/ml) for 12 h. Data are presented as the mean ± SD from at least three independent experiments. p Values were calculated using one-way ANOVA ( C – F ) or two-way ANOVA ( I and J ). CFX, ciprofloxacin; ROS, reactive oxygen species.
Apc3 Gfp Parasite Lines, supplied by Proteintech, used in various techniques. Bioz Stars score: 88/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/apc3/CDC27%3B+APC3+Fusion+Protein/pmc05884774-165-9-25
Average 88 stars, based on 1 article reviews
apc3 gfp parasite lines - by Bioz Stars, 2026-10
88/100 stars
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apc3  (Becton Dickinson)
90
Becton Dickinson apc3
Zn 2+ -mediated mitochondrial dysfunction and increased ROS promote RSL3-induced ferroptosis. A, PANC1 cells were exposed to RSL3 (1 μM) plus CFX (50 μg/ml) for 12 h. Mitochondria and Zn 2+ were stained with Mito-Tracker Green and TSQ, respectively. Scale bar represents 10 μm. B, JC-1 staining was indicated for mitochondrial membrane potential in PANC1 and HeLa cells treated with RSL3 (1 μM) in the presence or absence of CFX (50 μg/ml) for 12 h. JC-1 monomers indicate decreased mitochondrial membrane potential; JC-1 aggregates indicate increased mitochondrial membrane potential. Scale bar represents 200 μm. C, ROS of PANC1 and HeLa cells treated with RSL3 (1 μM), CFX (50 μg/ml), and N -acetylcysteine (NAC, 2 mM) for 12 h. D, mitochondrial ROS of PANC1 and HeLa cells treated with RSL3 (1 μM), CFX (50 μg/ml), and Mito-TEMPO (MT, 20 μM) for 12 h. E, cell viability of the indicated PANC1 and HeLa cells after treatment with RSL3 (1 μM), CFX (50 μg/ml), NAC (2 mM), and MT (20 μM) for 24 h. F, Mitochondrial ROS of PANC1 and HeLa cells treated with RSL3 (1 μM), CFX (50 μg/ml), and TPEN (10 μM) for 12 h. G, Western blot analysis of protein expression in control and <t>SLC25A25</t> -knockdown tumor cells. H, indicated PANC1 cells were exposed to RSL3 (1 μM) plus CFX (50 μg/ml) for 12 h. Mitochondria and Zn 2+ were stained with Mito-Tracker Green and TSQ, respectively. Scale bar represents 10 μm. I, cell viability of the indicated PANC1 and HeLa cells after treatment with RSL3 in the absence or presence of CFX (50 μg/ml) for 24 h. J, mitochondrial ROS of indicated PANC1 and HeLa cells treated with RSL3 (1 μM) and CFX (50 μg/ml) for 12 h. Data are presented as the mean ± SD from at least three independent experiments. p Values were calculated using one-way ANOVA ( C – F ) or two-way ANOVA ( I and J ). CFX, ciprofloxacin; ROS, reactive oxygen species.
Apc3, supplied by Becton Dickinson, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/apc3/apc3+antibody/pmc02224378-115-16-19
Average 90 stars, based on 1 article reviews
apc3 - by Bioz Stars, 2026-10
90/100 stars
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codon-optimised cdna of wildtype apc3 loopwt  (GenScript corporation)
90
GenScript corporation codon-optimised cdna of wildtype apc3 loopwt
Zn 2+ -mediated mitochondrial dysfunction and increased ROS promote RSL3-induced ferroptosis. A, PANC1 cells were exposed to RSL3 (1 μM) plus CFX (50 μg/ml) for 12 h. Mitochondria and Zn 2+ were stained with Mito-Tracker Green and TSQ, respectively. Scale bar represents 10 μm. B, JC-1 staining was indicated for mitochondrial membrane potential in PANC1 and HeLa cells treated with RSL3 (1 μM) in the presence or absence of CFX (50 μg/ml) for 12 h. JC-1 monomers indicate decreased mitochondrial membrane potential; JC-1 aggregates indicate increased mitochondrial membrane potential. Scale bar represents 200 μm. C, ROS of PANC1 and HeLa cells treated with RSL3 (1 μM), CFX (50 μg/ml), and N -acetylcysteine (NAC, 2 mM) for 12 h. D, mitochondrial ROS of PANC1 and HeLa cells treated with RSL3 (1 μM), CFX (50 μg/ml), and Mito-TEMPO (MT, 20 μM) for 12 h. E, cell viability of the indicated PANC1 and HeLa cells after treatment with RSL3 (1 μM), CFX (50 μg/ml), NAC (2 mM), and MT (20 μM) for 24 h. F, Mitochondrial ROS of PANC1 and HeLa cells treated with RSL3 (1 μM), CFX (50 μg/ml), and TPEN (10 μM) for 12 h. G, Western blot analysis of protein expression in control and <t>SLC25A25</t> -knockdown tumor cells. H, indicated PANC1 cells were exposed to RSL3 (1 μM) plus CFX (50 μg/ml) for 12 h. Mitochondria and Zn 2+ were stained with Mito-Tracker Green and TSQ, respectively. Scale bar represents 10 μm. I, cell viability of the indicated PANC1 and HeLa cells after treatment with RSL3 in the absence or presence of CFX (50 μg/ml) for 24 h. J, mitochondrial ROS of indicated PANC1 and HeLa cells treated with RSL3 (1 μM) and CFX (50 μg/ml) for 12 h. Data are presented as the mean ± SD from at least three independent experiments. p Values were calculated using one-way ANOVA ( C – F ) or two-way ANOVA ( I and J ). CFX, ciprofloxacin; ROS, reactive oxygen species.
Codon Optimised Cdna Of Wildtype Apc3 Loopwt, supplied by GenScript corporation, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/apc3/codon+optimised+cdna+of+wildtype+apc3+loopwt/pm39143240-283-6-22
Average 90 stars, based on 1 article reviews
codon-optimised cdna of wildtype apc3 loopwt - by Bioz Stars, 2026-10
90/100 stars
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apc 3 plasma surgery module  (Erbe Elektromedizin GmbH)
90
Erbe Elektromedizin GmbH apc 3 plasma surgery module
Zn 2+ -mediated mitochondrial dysfunction and increased ROS promote RSL3-induced ferroptosis. A, PANC1 cells were exposed to RSL3 (1 μM) plus CFX (50 μg/ml) for 12 h. Mitochondria and Zn 2+ were stained with Mito-Tracker Green and TSQ, respectively. Scale bar represents 10 μm. B, JC-1 staining was indicated for mitochondrial membrane potential in PANC1 and HeLa cells treated with RSL3 (1 μM) in the presence or absence of CFX (50 μg/ml) for 12 h. JC-1 monomers indicate decreased mitochondrial membrane potential; JC-1 aggregates indicate increased mitochondrial membrane potential. Scale bar represents 200 μm. C, ROS of PANC1 and HeLa cells treated with RSL3 (1 μM), CFX (50 μg/ml), and N -acetylcysteine (NAC, 2 mM) for 12 h. D, mitochondrial ROS of PANC1 and HeLa cells treated with RSL3 (1 μM), CFX (50 μg/ml), and Mito-TEMPO (MT, 20 μM) for 12 h. E, cell viability of the indicated PANC1 and HeLa cells after treatment with RSL3 (1 μM), CFX (50 μg/ml), NAC (2 mM), and MT (20 μM) for 24 h. F, Mitochondrial ROS of PANC1 and HeLa cells treated with RSL3 (1 μM), CFX (50 μg/ml), and TPEN (10 μM) for 12 h. G, Western blot analysis of protein expression in control and <t>SLC25A25</t> -knockdown tumor cells. H, indicated PANC1 cells were exposed to RSL3 (1 μM) plus CFX (50 μg/ml) for 12 h. Mitochondria and Zn 2+ were stained with Mito-Tracker Green and TSQ, respectively. Scale bar represents 10 μm. I, cell viability of the indicated PANC1 and HeLa cells after treatment with RSL3 in the absence or presence of CFX (50 μg/ml) for 24 h. J, mitochondrial ROS of indicated PANC1 and HeLa cells treated with RSL3 (1 μM) and CFX (50 μg/ml) for 12 h. Data are presented as the mean ± SD from at least three independent experiments. p Values were calculated using one-way ANOVA ( C – F ) or two-way ANOVA ( I and J ). CFX, ciprofloxacin; ROS, reactive oxygen species.
Apc 3 Plasma Surgery Module, supplied by Erbe Elektromedizin GmbH, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/apc3/argon+plasma+sources+erbe+apc3/pmc10026650-86-10-5
Average 90 stars, based on 1 article reviews
apc 3 plasma surgery module - by Bioz Stars, 2026-10
90/100 stars
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apc3  (MBL Life science)
90
MBL Life science apc3
Zn 2+ -mediated mitochondrial dysfunction and increased ROS promote RSL3-induced ferroptosis. A, PANC1 cells were exposed to RSL3 (1 μM) plus CFX (50 μg/ml) for 12 h. Mitochondria and Zn 2+ were stained with Mito-Tracker Green and TSQ, respectively. Scale bar represents 10 μm. B, JC-1 staining was indicated for mitochondrial membrane potential in PANC1 and HeLa cells treated with RSL3 (1 μM) in the presence or absence of CFX (50 μg/ml) for 12 h. JC-1 monomers indicate decreased mitochondrial membrane potential; JC-1 aggregates indicate increased mitochondrial membrane potential. Scale bar represents 200 μm. C, ROS of PANC1 and HeLa cells treated with RSL3 (1 μM), CFX (50 μg/ml), and N -acetylcysteine (NAC, 2 mM) for 12 h. D, mitochondrial ROS of PANC1 and HeLa cells treated with RSL3 (1 μM), CFX (50 μg/ml), and Mito-TEMPO (MT, 20 μM) for 12 h. E, cell viability of the indicated PANC1 and HeLa cells after treatment with RSL3 (1 μM), CFX (50 μg/ml), NAC (2 mM), and MT (20 μM) for 24 h. F, Mitochondrial ROS of PANC1 and HeLa cells treated with RSL3 (1 μM), CFX (50 μg/ml), and TPEN (10 μM) for 12 h. G, Western blot analysis of protein expression in control and <t>SLC25A25</t> -knockdown tumor cells. H, indicated PANC1 cells were exposed to RSL3 (1 μM) plus CFX (50 μg/ml) for 12 h. Mitochondria and Zn 2+ were stained with Mito-Tracker Green and TSQ, respectively. Scale bar represents 10 μm. I, cell viability of the indicated PANC1 and HeLa cells after treatment with RSL3 in the absence or presence of CFX (50 μg/ml) for 24 h. J, mitochondrial ROS of indicated PANC1 and HeLa cells treated with RSL3 (1 μM) and CFX (50 μg/ml) for 12 h. Data are presented as the mean ± SD from at least three independent experiments. p Values were calculated using one-way ANOVA ( C – F ) or two-way ANOVA ( I and J ). CFX, ciprofloxacin; ROS, reactive oxygen species.
Apc3, supplied by MBL Life science, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/apc3/apc3+antibody/pmc05940398-122-44-45
Average 90 stars, based on 1 article reviews
apc3 - by Bioz Stars, 2026-10
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monoclonal antibody to the amino terminus of apc apc-3  (Oncogene Science Inc)
90
Oncogene Science Inc monoclonal antibody to the amino terminus of apc apc-3
Zn 2+ -mediated mitochondrial dysfunction and increased ROS promote RSL3-induced ferroptosis. A, PANC1 cells were exposed to RSL3 (1 μM) plus CFX (50 μg/ml) for 12 h. Mitochondria and Zn 2+ were stained with Mito-Tracker Green and TSQ, respectively. Scale bar represents 10 μm. B, JC-1 staining was indicated for mitochondrial membrane potential in PANC1 and HeLa cells treated with RSL3 (1 μM) in the presence or absence of CFX (50 μg/ml) for 12 h. JC-1 monomers indicate decreased mitochondrial membrane potential; JC-1 aggregates indicate increased mitochondrial membrane potential. Scale bar represents 200 μm. C, ROS of PANC1 and HeLa cells treated with RSL3 (1 μM), CFX (50 μg/ml), and N -acetylcysteine (NAC, 2 mM) for 12 h. D, mitochondrial ROS of PANC1 and HeLa cells treated with RSL3 (1 μM), CFX (50 μg/ml), and Mito-TEMPO (MT, 20 μM) for 12 h. E, cell viability of the indicated PANC1 and HeLa cells after treatment with RSL3 (1 μM), CFX (50 μg/ml), NAC (2 mM), and MT (20 μM) for 24 h. F, Mitochondrial ROS of PANC1 and HeLa cells treated with RSL3 (1 μM), CFX (50 μg/ml), and TPEN (10 μM) for 12 h. G, Western blot analysis of protein expression in control and <t>SLC25A25</t> -knockdown tumor cells. H, indicated PANC1 cells were exposed to RSL3 (1 μM) plus CFX (50 μg/ml) for 12 h. Mitochondria and Zn 2+ were stained with Mito-Tracker Green and TSQ, respectively. Scale bar represents 10 μm. I, cell viability of the indicated PANC1 and HeLa cells after treatment with RSL3 in the absence or presence of CFX (50 μg/ml) for 24 h. J, mitochondrial ROS of indicated PANC1 and HeLa cells treated with RSL3 (1 μM) and CFX (50 μg/ml) for 12 h. Data are presented as the mean ± SD from at least three independent experiments. p Values were calculated using one-way ANOVA ( C – F ) or two-way ANOVA ( I and J ). CFX, ciprofloxacin; ROS, reactive oxygen species.
Monoclonal Antibody To The Amino Terminus Of Apc Apc 3, supplied by Oncogene Science Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/apc3/monoclonal+antibody+to+the+amino+terminus+of+apc+apc+3/pmc01853030-66-11-13
Average 90 stars, based on 1 article reviews
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Zn 2+ -mediated mitochondrial dysfunction and increased ROS promote RSL3-induced ferroptosis. A, PANC1 cells were exposed to RSL3 (1 μM) plus CFX (50 μg/ml) for 12 h. Mitochondria and Zn 2+ were stained with Mito-Tracker Green and TSQ, respectively. Scale bar represents 10 μm. B, JC-1 staining was indicated for mitochondrial membrane potential in PANC1 and HeLa cells treated with RSL3 (1 μM) in the presence or absence of CFX (50 μg/ml) for 12 h. JC-1 monomers indicate decreased mitochondrial membrane potential; JC-1 aggregates indicate increased mitochondrial membrane potential. Scale bar represents 200 μm. C, ROS of PANC1 and HeLa cells treated with RSL3 (1 μM), CFX (50 μg/ml), and N -acetylcysteine (NAC, 2 mM) for 12 h. D, mitochondrial ROS of PANC1 and HeLa cells treated with RSL3 (1 μM), CFX (50 μg/ml), and Mito-TEMPO (MT, 20 μM) for 12 h. E, cell viability of the indicated PANC1 and HeLa cells after treatment with RSL3 (1 μM), CFX (50 μg/ml), NAC (2 mM), and MT (20 μM) for 24 h. F, Mitochondrial ROS of PANC1 and HeLa cells treated with RSL3 (1 μM), CFX (50 μg/ml), and TPEN (10 μM) for 12 h. G, Western blot analysis of protein expression in control and SLC25A25 -knockdown tumor cells. H, indicated PANC1 cells were exposed to RSL3 (1 μM) plus CFX (50 μg/ml) for 12 h. Mitochondria and Zn 2+ were stained with Mito-Tracker Green and TSQ, respectively. Scale bar represents 10 μm. I, cell viability of the indicated PANC1 and HeLa cells after treatment with RSL3 in the absence or presence of CFX (50 μg/ml) for 24 h. J, mitochondrial ROS of indicated PANC1 and HeLa cells treated with RSL3 (1 μM) and CFX (50 μg/ml) for 12 h. Data are presented as the mean ± SD from at least three independent experiments. p Values were calculated using one-way ANOVA ( C – F ) or two-way ANOVA ( I and J ). CFX, ciprofloxacin; ROS, reactive oxygen species.

Journal: The Journal of Biological Chemistry

Article Title: Ciprofloxacin enhances RSL3-induced ferroptosis by promoting mitochondrial Zn 2+ accumulation via the STING1–CAV2 pathway

doi: 10.1016/j.jbc.2025.110653

Figure Lengend Snippet: Zn 2+ -mediated mitochondrial dysfunction and increased ROS promote RSL3-induced ferroptosis. A, PANC1 cells were exposed to RSL3 (1 μM) plus CFX (50 μg/ml) for 12 h. Mitochondria and Zn 2+ were stained with Mito-Tracker Green and TSQ, respectively. Scale bar represents 10 μm. B, JC-1 staining was indicated for mitochondrial membrane potential in PANC1 and HeLa cells treated with RSL3 (1 μM) in the presence or absence of CFX (50 μg/ml) for 12 h. JC-1 monomers indicate decreased mitochondrial membrane potential; JC-1 aggregates indicate increased mitochondrial membrane potential. Scale bar represents 200 μm. C, ROS of PANC1 and HeLa cells treated with RSL3 (1 μM), CFX (50 μg/ml), and N -acetylcysteine (NAC, 2 mM) for 12 h. D, mitochondrial ROS of PANC1 and HeLa cells treated with RSL3 (1 μM), CFX (50 μg/ml), and Mito-TEMPO (MT, 20 μM) for 12 h. E, cell viability of the indicated PANC1 and HeLa cells after treatment with RSL3 (1 μM), CFX (50 μg/ml), NAC (2 mM), and MT (20 μM) for 24 h. F, Mitochondrial ROS of PANC1 and HeLa cells treated with RSL3 (1 μM), CFX (50 μg/ml), and TPEN (10 μM) for 12 h. G, Western blot analysis of protein expression in control and SLC25A25 -knockdown tumor cells. H, indicated PANC1 cells were exposed to RSL3 (1 μM) plus CFX (50 μg/ml) for 12 h. Mitochondria and Zn 2+ were stained with Mito-Tracker Green and TSQ, respectively. Scale bar represents 10 μm. I, cell viability of the indicated PANC1 and HeLa cells after treatment with RSL3 in the absence or presence of CFX (50 μg/ml) for 24 h. J, mitochondrial ROS of indicated PANC1 and HeLa cells treated with RSL3 (1 μM) and CFX (50 μg/ml) for 12 h. Data are presented as the mean ± SD from at least three independent experiments. p Values were calculated using one-way ANOVA ( C – F ) or two-way ANOVA ( I and J ). CFX, ciprofloxacin; ROS, reactive oxygen species.

Article Snippet: Antibodies targeting TOP2B (20549-1-AP), topoisomerase I (20705-1-AP), topoisomerase II alpha (20233-1-AP), ACTB (66009-1-Ig), STING1 (19851-1-AP), p-H2A (83307-2-RR), LMNB1 (12987-1-AP), TOM20 (66777-1-Ig), BAK (29552-1-AP), BAX (50599-2-Ig), transferrin receptor (10084-2-AP), GAPDH (60004-1-Ig), SLC25A25 (21568-1-AP), dihydroorotate dehydrogenase (14877-1-AP), and HSPA5 (66574-1-Ig) were sourced from Proteintech Biotechnology.

Techniques: Staining, Membrane, Western Blot, Expressing, Control, Knockdown