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    • anti perlecan a7l6 antibody  (Novus Biologicals)
    91
    Novus Biologicals anti perlecan a7l6 antibody
    Severe trabecular osteoporosis in <t>perlecan-deficient</t> (Hypo) male skeletons. Distal femur (long bone) was examined ( A ) in various age groups by ex vivo μCT imaging and ( B ) in a longitudinal study using in vivo μCT imaging. ( C ) Histomorphometric and histological analysis of osteoblastic bone formation indices and osteoclastic TRAP staining. The axial skeleton (spinal column and/or the vertebral body L4) was examined ( D ) in various age groups by ex vivo μCT imaging ( E ) and in a longitudinal study by in vivo μCT imaging. Significance for within-genotype age comparisons is indicated by different letters (uppercase for WT and lowercase for Hypo) and between-genotype comparisons are indicated by * p < 0.05). Scale bar in D = 1 mm.
    Anti Perlecan A7l6 Antibody, supplied by Novus Biologicals, used in various techniques. Bioz Stars score: 91/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/anti perlecan a7l6 antibody/product/Novus Biologicals
    Average 91 stars, based on 1 article reviews
    Price from $9.99 to $1999.99
    anti perlecan a7l6 antibody - by Bioz Stars, 2023-05
    91/100 stars
    		  Buy from Supplier
    pet29b clbppep chis plasmid 48243  (Addgene inc)
    N/A
    Standard format Plasmid sent in bacteria as agar stab
    		   Buy from Supplier

    Image Search Results


    Severe trabecular osteoporosis in perlecan-deficient (Hypo) male skeletons. Distal femur (long bone) was examined ( A ) in various age groups by ex vivo μCT imaging and ( B ) in a longitudinal study using in vivo μCT imaging. ( C ) Histomorphometric and histological analysis of osteoblastic bone formation indices and osteoclastic TRAP staining. The axial skeleton (spinal column and/or the vertebral body L4) was examined ( D ) in various age groups by ex vivo μCT imaging ( E ) and in a longitudinal study by in vivo μCT imaging. Significance for within-genotype age comparisons is indicated by different letters (uppercase for WT and lowercase for Hypo) and between-genotype comparisons are indicated by * p < 0.05). Scale bar in D = 1 mm.

    Journal: Biomolecules

    Article Title: Trabecular Bone Deficit and Enhanced Anabolic Response to Re-Ambulation after Disuse in Perlecan-Deficient Skeleton

    doi: 10.3390/biom10020198

    Figure Lengend Snippet: Severe trabecular osteoporosis in perlecan-deficient (Hypo) male skeletons. Distal femur (long bone) was examined ( A ) in various age groups by ex vivo μCT imaging and ( B ) in a longitudinal study using in vivo μCT imaging. ( C ) Histomorphometric and histological analysis of osteoblastic bone formation indices and osteoclastic TRAP staining. The axial skeleton (spinal column and/or the vertebral body L4) was examined ( D ) in various age groups by ex vivo μCT imaging ( E ) and in a longitudinal study by in vivo μCT imaging. Significance for within-genotype age comparisons is indicated by different letters (uppercase for WT and lowercase for Hypo) and between-genotype comparisons are indicated by * p < 0.05). Scale bar in D = 1 mm.

    Article Snippet: The dot-blot immunoassay was performed using an anti-perlecan A7L6 antibody (Novus Biologicals, LLC, Centennial, CO, USA) [ ] and a goat-anti-rabbit HRP conjugated secondary antibody (1:100,000 dilution, Abcam, Cambridge, MA, USA).

    Techniques: Ex Vivo, Imaging, In Vivo, Staining

    Perlecan deficiency altered MSC differentiation, but not proliferation ( A ), and increased osteoclastogenesis and resorptive activity ( B ). Perlecan content was reduced in Hypo marrow ( C ). Treating the primary MSC and osteoclast progenitor cells with exogenous heparin rescued the effects from the perlecan deficiency ( D ). Significance for within-genotype dose comparisons are indicated by different letters (uppercase for WT and lowercase for Hypo) and between-genotype comparisons indicated by * ( p < 0.05). Open bars: WT; filled bars: Hypo.

    Journal: Biomolecules

    Article Title: Trabecular Bone Deficit and Enhanced Anabolic Response to Re-Ambulation after Disuse in Perlecan-Deficient Skeleton

    doi: 10.3390/biom10020198

    Figure Lengend Snippet: Perlecan deficiency altered MSC differentiation, but not proliferation ( A ), and increased osteoclastogenesis and resorptive activity ( B ). Perlecan content was reduced in Hypo marrow ( C ). Treating the primary MSC and osteoclast progenitor cells with exogenous heparin rescued the effects from the perlecan deficiency ( D ). Significance for within-genotype dose comparisons are indicated by different letters (uppercase for WT and lowercase for Hypo) and between-genotype comparisons indicated by * ( p < 0.05). Open bars: WT; filled bars: Hypo.

    Article Snippet: The dot-blot immunoassay was performed using an anti-perlecan A7L6 antibody (Novus Biologicals, LLC, Centennial, CO, USA) [ ] and a goat-anti-rabbit HRP conjugated secondary antibody (1:100,000 dilution, Abcam, Cambridge, MA, USA).

    Techniques: Activity Assay