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phospho p38 mapk antibody thr180 tyr182 d3f9  (Cell Signaling Technology Inc)


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    Cell Signaling Technology Inc phospho p38 mapk antibody thr180 tyr182 d3f9
    Phospho P38 Mapk Antibody Thr180 Tyr182 D3f9, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/phospho p38 mapk antibody thr180 tyr182 d3f9/product/Cell Signaling Technology Inc
    Average 86 stars, based on 1 article reviews
    Price from $9.99 to $1999.99
    phospho p38 mapk antibody thr180 tyr182 d3f9 - by Bioz Stars, 2024-10
    86/100 stars

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    Impact of levosimendan, OR-1855, and OR-1896 on endothelial inflammatory MAPK signalling. Endothelial cells were pre-incubated with levosimendan, OR-1855, OR-1896 (10 µM; 30 min.), or sham solution (DMSO v/v ) followed by stimulation with IL-1β (10 ng/mL) for 30 min before lysis and subsequent western blotting. ( a ) Levosimendan, OR-1855, and OR-1896 decreased IL-1β-dependent phosphorylation of <t>p38</t> <t>MAPK</t> (n = 4). ( b ) Levosimendan, OR-1855, and OR-1896 decreased IL-1β-dependent phosphorylation of ERK1/2 MAPK (n = 4–5). ( c ) OR-1855 and OR-1896 diminished IL-1β-dependent phosphorylation of JNK (n = 15), while levosimendan had no effect (n = 15). ( d ) Levosimendan, OR-1855, and OR-1896 decreased IL-1β-dependent phosphorylation of cjun (AP-1) (n = 7). The graphs show protein band densities of phosphorylated forms of the MAP Kinases and cjun normalized to GAPDH (* p < 0.05).
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    Impact of levosimendan, OR-1855, and OR-1896 on endothelial inflammatory MAPK signalling. Endothelial cells were pre-incubated with levosimendan, OR-1855, OR-1896 (10 µM; 30 min.), or sham solution (DMSO v/v ) followed by stimulation with IL-1β (10 ng/mL) for 30 min before lysis and subsequent western blotting. ( a ) Levosimendan, OR-1855, and OR-1896 decreased IL-1β-dependent phosphorylation of <t>p38</t> <t>MAPK</t> (n = 4). ( b ) Levosimendan, OR-1855, and OR-1896 decreased IL-1β-dependent phosphorylation of ERK1/2 MAPK (n = 4–5). ( c ) OR-1855 and OR-1896 diminished IL-1β-dependent phosphorylation of JNK (n = 15), while levosimendan had no effect (n = 15). ( d ) Levosimendan, OR-1855, and OR-1896 decreased IL-1β-dependent phosphorylation of cjun (AP-1) (n = 7). The graphs show protein band densities of phosphorylated forms of the MAP Kinases and cjun normalized to GAPDH (* p < 0.05).
    Anti Phosphorylated Phospho P38 Mapk Thr180 Tyr182 D3f9, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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    Average 86 stars, based on 1 article reviews
    Price from $9.99 to $1999.99
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    rabbit anti phospho thr180 tyr182 p38 mapk d3f9 antibody  (Cell Signaling Technology Inc)
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    Impact of levosimendan, OR-1855, and OR-1896 on endothelial inflammatory MAPK signalling. Endothelial cells were pre-incubated with levosimendan, OR-1855, OR-1896 (10 µM; 30 min.), or sham solution (DMSO v/v ) followed by stimulation with IL-1β (10 ng/mL) for 30 min before lysis and subsequent western blotting. ( a ) Levosimendan, OR-1855, and OR-1896 decreased IL-1β-dependent phosphorylation of <t>p38</t> <t>MAPK</t> (n = 4). ( b ) Levosimendan, OR-1855, and OR-1896 decreased IL-1β-dependent phosphorylation of ERK1/2 MAPK (n = 4–5). ( c ) OR-1855 and OR-1896 diminished IL-1β-dependent phosphorylation of JNK (n = 15), while levosimendan had no effect (n = 15). ( d ) Levosimendan, OR-1855, and OR-1896 decreased IL-1β-dependent phosphorylation of cjun (AP-1) (n = 7). The graphs show protein band densities of phosphorylated forms of the MAP Kinases and cjun normalized to GAPDH (* p < 0.05).
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    rabbit anti phospho thr180 tyr182 p38 mapk d3f9 antibody - by Bioz Stars, 2024-10
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    rabbit monoclonal anti phospho p38 mapk thr180 tyr182 d3f9 xp antibody  (Cell Signaling Technology Inc)
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    Impact of levosimendan, OR-1855, and OR-1896 on endothelial inflammatory MAPK signalling. Endothelial cells were pre-incubated with levosimendan, OR-1855, OR-1896 (10 µM; 30 min.), or sham solution (DMSO v/v ) followed by stimulation with IL-1β (10 ng/mL) for 30 min before lysis and subsequent western blotting. ( a ) Levosimendan, OR-1855, and OR-1896 decreased IL-1β-dependent phosphorylation of <t>p38</t> <t>MAPK</t> (n = 4). ( b ) Levosimendan, OR-1855, and OR-1896 decreased IL-1β-dependent phosphorylation of ERK1/2 MAPK (n = 4–5). ( c ) OR-1855 and OR-1896 diminished IL-1β-dependent phosphorylation of JNK (n = 15), while levosimendan had no effect (n = 15). ( d ) Levosimendan, OR-1855, and OR-1896 decreased IL-1β-dependent phosphorylation of cjun (AP-1) (n = 7). The graphs show protein band densities of phosphorylated forms of the MAP Kinases and cjun normalized to GAPDH (* p < 0.05).
    Rabbit Monoclonal Anti Phospho P38 Mapk Thr180 Tyr182 D3f9 Xp Antibody, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 98/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/rabbit monoclonal anti phospho p38 mapk thr180 tyr182 d3f9 xp antibody/product/Cell Signaling Technology Inc
    Average 98 stars, based on 1 article reviews
    Price from $9.99 to $1999.99
    rabbit monoclonal anti phospho p38 mapk thr180 tyr182 d3f9 xp antibody - by Bioz Stars, 2024-10
    98/100 stars
    		  Buy from Supplier

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    Impact of levosimendan, OR-1855, and OR-1896 on endothelial inflammatory MAPK signalling. Endothelial cells were pre-incubated with levosimendan, OR-1855, OR-1896 (10 µM; 30 min.), or sham solution (DMSO v/v ) followed by stimulation with IL-1β (10 ng/mL) for 30 min before lysis and subsequent western blotting. ( a ) Levosimendan, OR-1855, and OR-1896 decreased IL-1β-dependent phosphorylation of p38 MAPK (n = 4). ( b ) Levosimendan, OR-1855, and OR-1896 decreased IL-1β-dependent phosphorylation of ERK1/2 MAPK (n = 4–5). ( c ) OR-1855 and OR-1896 diminished IL-1β-dependent phosphorylation of JNK (n = 15), while levosimendan had no effect (n = 15). ( d ) Levosimendan, OR-1855, and OR-1896 decreased IL-1β-dependent phosphorylation of cjun (AP-1) (n = 7). The graphs show protein band densities of phosphorylated forms of the MAP Kinases and cjun normalized to GAPDH (* p < 0.05).

    Journal: Biomedicines

    Article Title: The Effects of the Levosimendan Metabolites OR-1855 and OR-1896 on Endothelial Pro-Inflammatory Responses

    doi: 10.3390/biomedicines11030918

    Figure Lengend Snippet: Impact of levosimendan, OR-1855, and OR-1896 on endothelial inflammatory MAPK signalling. Endothelial cells were pre-incubated with levosimendan, OR-1855, OR-1896 (10 µM; 30 min.), or sham solution (DMSO v/v ) followed by stimulation with IL-1β (10 ng/mL) for 30 min before lysis and subsequent western blotting. ( a ) Levosimendan, OR-1855, and OR-1896 decreased IL-1β-dependent phosphorylation of p38 MAPK (n = 4). ( b ) Levosimendan, OR-1855, and OR-1896 decreased IL-1β-dependent phosphorylation of ERK1/2 MAPK (n = 4–5). ( c ) OR-1855 and OR-1896 diminished IL-1β-dependent phosphorylation of JNK (n = 15), while levosimendan had no effect (n = 15). ( d ) Levosimendan, OR-1855, and OR-1896 decreased IL-1β-dependent phosphorylation of cjun (AP-1) (n = 7). The graphs show protein band densities of phosphorylated forms of the MAP Kinases and cjun normalized to GAPDH (* p < 0.05).

    Article Snippet: Rabbit phospho-p38 MAPK (Thr180/Tyr182) (D3F9) XP™ (#4511), rabbit VCAM-1 (#12367), rabbit phospho-ERK1/2 (#4307), rabbit phospho-JNK (#4668), rabbit phospho-cjun (#320T), rabbit β-Actin (13E5) (#4790) antibodies were from Cell Signaling Technology (Leiden, the Netherlands).

    Techniques: Incubation, Lysis, Western Blot