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p2ry2 antibody  (Alomone Labs)


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    Alomone Labs p2ry2 antibody
    P2ry2 Antibody, supplied by Alomone Labs, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/apr-102/pm41887391-341-37-40?v=Alomone+Labs
    Average 94 stars, based on 1 article reviews
    p2ry2 antibody - by Bioz Stars, 2026-07
    94/100 stars

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    p2ry2 antibody  (Alomone Labs)
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    Alomone Labs p2ry2 antibody
    P2ry2 Antibody, supplied by Alomone Labs, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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    anti kcnn4 receptor extracellular  (Alomone Labs)
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    A , Effect on migration. ATP increased Sca‐1 + cell migration, which was significantly inhibited by the IK Ca channel blocker TRAM34 ( a ). MCP‐1 also enhanced migration, and TRAM34 similarly suppressed this increase ( b ). (Statistical significance was confirmed by HC3‐robust interaction model with 6 biological replicates.) B , The ATP‐enhanced migration of Sca‐1 + cells was significantly abolished by IK Ca channel inhibition with shKCNN4 ( P =0.0360, HC3‐robust interaction model, n=5). C , The proliferation of Sca‐1 + cells induced by ATP ( a , b ) and PDGF‐BB ( c ) was significantly suppressed by inhibition of IK Ca channels with TRAM34 or shKCNN4 ( P values are indicated on the graph; HC3‐robust interaction model, n=5). ATP indicates adenosine triphosphate; CON, control; HC3, heteroskedasticity‐consistent robust SE; IK Ca , intermediate conductance calcium‐activated potassium channel; MCP‐1, monocyte chemoattractant protein‐1; PDGF‐BB, platelet‐derived growth factor‐BB; Sca‐1 + , stem cell antigen‐1 positive; shKCNN4, a short hairpin RNA construct specifically designed to target and silence the expression of the <t>KCNN4</t> gene; and TRAM34, 1‐ [(2‐chlorophenyl) diphenylmethyl]‐1h‐pyrazole.
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    anti p2y2 receptor extracellular antibody  (Alomone Labs)
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    A , Effect on migration. ATP increased Sca‐1 + cell migration, which was significantly inhibited by the IK Ca channel blocker TRAM34 ( a ). MCP‐1 also enhanced migration, and TRAM34 similarly suppressed this increase ( b ). (Statistical significance was confirmed by HC3‐robust interaction model with 6 biological replicates.) B , The ATP‐enhanced migration of Sca‐1 + cells was significantly abolished by IK Ca channel inhibition with shKCNN4 ( P =0.0360, HC3‐robust interaction model, n=5). C , The proliferation of Sca‐1 + cells induced by ATP ( a , b ) and PDGF‐BB ( c ) was significantly suppressed by inhibition of IK Ca channels with TRAM34 or shKCNN4 ( P values are indicated on the graph; HC3‐robust interaction model, n=5). ATP indicates adenosine triphosphate; CON, control; HC3, heteroskedasticity‐consistent robust SE; IK Ca , intermediate conductance calcium‐activated potassium channel; MCP‐1, monocyte chemoattractant protein‐1; PDGF‐BB, platelet‐derived growth factor‐BB; Sca‐1 + , stem cell antigen‐1 positive; shKCNN4, a short hairpin RNA construct specifically designed to target and silence the expression of the <t>KCNN4</t> gene; and TRAM34, 1‐ [(2‐chlorophenyl) diphenylmethyl]‐1h‐pyrazole.
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    rabbit polyclonal antibody  (Alomone Labs)
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    A , Effect on migration. ATP increased Sca‐1 + cell migration, which was significantly inhibited by the IK Ca channel blocker TRAM34 ( a ). MCP‐1 also enhanced migration, and TRAM34 similarly suppressed this increase ( b ). (Statistical significance was confirmed by HC3‐robust interaction model with 6 biological replicates.) B , The ATP‐enhanced migration of Sca‐1 + cells was significantly abolished by IK Ca channel inhibition with shKCNN4 ( P =0.0360, HC3‐robust interaction model, n=5). C , The proliferation of Sca‐1 + cells induced by ATP ( a , b ) and PDGF‐BB ( c ) was significantly suppressed by inhibition of IK Ca channels with TRAM34 or shKCNN4 ( P values are indicated on the graph; HC3‐robust interaction model, n=5). ATP indicates adenosine triphosphate; CON, control; HC3, heteroskedasticity‐consistent robust SE; IK Ca , intermediate conductance calcium‐activated potassium channel; MCP‐1, monocyte chemoattractant protein‐1; PDGF‐BB, platelet‐derived growth factor‐BB; Sca‐1 + , stem cell antigen‐1 positive; shKCNN4, a short hairpin RNA construct specifically designed to target and silence the expression of the <t>KCNN4</t> gene; and TRAM34, 1‐ [(2‐chlorophenyl) diphenylmethyl]‐1h‐pyrazole.
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    fitcconjugated antibody  (Alomone Labs)
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    A , Effect on migration. ATP increased Sca‐1 + cell migration, which was significantly inhibited by the IK Ca channel blocker TRAM34 ( a ). MCP‐1 also enhanced migration, and TRAM34 similarly suppressed this increase ( b ). (Statistical significance was confirmed by HC3‐robust interaction model with 6 biological replicates.) B , The ATP‐enhanced migration of Sca‐1 + cells was significantly abolished by IK Ca channel inhibition with shKCNN4 ( P =0.0360, HC3‐robust interaction model, n=5). C , The proliferation of Sca‐1 + cells induced by ATP ( a , b ) and PDGF‐BB ( c ) was significantly suppressed by inhibition of IK Ca channels with TRAM34 or shKCNN4 ( P values are indicated on the graph; HC3‐robust interaction model, n=5). ATP indicates adenosine triphosphate; CON, control; HC3, heteroskedasticity‐consistent robust SE; IK Ca , intermediate conductance calcium‐activated potassium channel; MCP‐1, monocyte chemoattractant protein‐1; PDGF‐BB, platelet‐derived growth factor‐BB; Sca‐1 + , stem cell antigen‐1 positive; shKCNN4, a short hairpin RNA construct specifically designed to target and silence the expression of the <t>KCNN4</t> gene; and TRAM34, 1‐ [(2‐chlorophenyl) diphenylmethyl]‐1h‐pyrazole.
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    A , Effect on migration. ATP increased Sca‐1 + cell migration, which was significantly inhibited by the IK Ca channel blocker TRAM34 ( a ). MCP‐1 also enhanced migration, and TRAM34 similarly suppressed this increase ( b ). (Statistical significance was confirmed by HC3‐robust interaction model with 6 biological replicates.) B , The ATP‐enhanced migration of Sca‐1 + cells was significantly abolished by IK Ca channel inhibition with shKCNN4 ( P =0.0360, HC3‐robust interaction model, n=5). C , The proliferation of Sca‐1 + cells induced by ATP ( a , b ) and PDGF‐BB ( c ) was significantly suppressed by inhibition of IK Ca channels with TRAM34 or shKCNN4 ( P values are indicated on the graph; HC3‐robust interaction model, n=5). ATP indicates adenosine triphosphate; CON, control; HC3, heteroskedasticity‐consistent robust SE; IK Ca , intermediate conductance calcium‐activated potassium channel; MCP‐1, monocyte chemoattractant protein‐1; PDGF‐BB, platelet‐derived growth factor‐BB; Sca‐1 + , stem cell antigen‐1 positive; shKCNN4, a short hairpin RNA construct specifically designed to target and silence the expression of the <t>KCNN4</t> gene; and TRAM34, 1‐ [(2‐chlorophenyl) diphenylmethyl]‐1h‐pyrazole.
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    Anti P2y2 Receptor Extracellular, supplied by Alomone Labs, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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    p2y2r  (Alomone Labs)
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    A , Effect on migration. ATP increased Sca‐1 + cell migration, which was significantly inhibited by the IK Ca channel blocker TRAM34 ( a ). MCP‐1 also enhanced migration, and TRAM34 similarly suppressed this increase ( b ). (Statistical significance was confirmed by HC3‐robust interaction model with 6 biological replicates.) B , The ATP‐enhanced migration of Sca‐1 + cells was significantly abolished by IK Ca channel inhibition with shKCNN4 ( P =0.0360, HC3‐robust interaction model, n=5). C , The proliferation of Sca‐1 + cells induced by ATP ( a , b ) and PDGF‐BB ( c ) was significantly suppressed by inhibition of IK Ca channels with TRAM34 or shKCNN4 ( P values are indicated on the graph; HC3‐robust interaction model, n=5). ATP indicates adenosine triphosphate; CON, control; HC3, heteroskedasticity‐consistent robust SE; IK Ca , intermediate conductance calcium‐activated potassium channel; MCP‐1, monocyte chemoattractant protein‐1; PDGF‐BB, platelet‐derived growth factor‐BB; Sca‐1 + , stem cell antigen‐1 positive; shKCNN4, a short hairpin RNA construct specifically designed to target and silence the expression of the <t>KCNN4</t> gene; and TRAM34, 1‐ [(2‐chlorophenyl) diphenylmethyl]‐1h‐pyrazole.
    P2y2r, supplied by Alomone Labs, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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    anti p2y12  (Alomone Labs)
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    A , Effect on migration. ATP increased Sca‐1 + cell migration, which was significantly inhibited by the IK Ca channel blocker TRAM34 ( a ). MCP‐1 also enhanced migration, and TRAM34 similarly suppressed this increase ( b ). (Statistical significance was confirmed by HC3‐robust interaction model with 6 biological replicates.) B , The ATP‐enhanced migration of Sca‐1 + cells was significantly abolished by IK Ca channel inhibition with shKCNN4 ( P =0.0360, HC3‐robust interaction model, n=5). C , The proliferation of Sca‐1 + cells induced by ATP ( a , b ) and PDGF‐BB ( c ) was significantly suppressed by inhibition of IK Ca channels with TRAM34 or shKCNN4 ( P values are indicated on the graph; HC3‐robust interaction model, n=5). ATP indicates adenosine triphosphate; CON, control; HC3, heteroskedasticity‐consistent robust SE; IK Ca , intermediate conductance calcium‐activated potassium channel; MCP‐1, monocyte chemoattractant protein‐1; PDGF‐BB, platelet‐derived growth factor‐BB; Sca‐1 + , stem cell antigen‐1 positive; shKCNN4, a short hairpin RNA construct specifically designed to target and silence the expression of the <t>KCNN4</t> gene; and TRAM34, 1‐ [(2‐chlorophenyl) diphenylmethyl]‐1h‐pyrazole.
    Anti P2y12, supplied by Alomone Labs, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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    p2y12  (Alomone Labs)
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    Alomone Labs p2y12
    A , Effect on migration. ATP increased Sca‐1 + cell migration, which was significantly inhibited by the IK Ca channel blocker TRAM34 ( a ). MCP‐1 also enhanced migration, and TRAM34 similarly suppressed this increase ( b ). (Statistical significance was confirmed by HC3‐robust interaction model with 6 biological replicates.) B , The ATP‐enhanced migration of Sca‐1 + cells was significantly abolished by IK Ca channel inhibition with shKCNN4 ( P =0.0360, HC3‐robust interaction model, n=5). C , The proliferation of Sca‐1 + cells induced by ATP ( a , b ) and PDGF‐BB ( c ) was significantly suppressed by inhibition of IK Ca channels with TRAM34 or shKCNN4 ( P values are indicated on the graph; HC3‐robust interaction model, n=5). ATP indicates adenosine triphosphate; CON, control; HC3, heteroskedasticity‐consistent robust SE; IK Ca , intermediate conductance calcium‐activated potassium channel; MCP‐1, monocyte chemoattractant protein‐1; PDGF‐BB, platelet‐derived growth factor‐BB; Sca‐1 + , stem cell antigen‐1 positive; shKCNN4, a short hairpin RNA construct specifically designed to target and silence the expression of the <t>KCNN4</t> gene; and TRAM34, 1‐ [(2‐chlorophenyl) diphenylmethyl]‐1h‐pyrazole.
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    A , Effect on migration. ATP increased Sca‐1 + cell migration, which was significantly inhibited by the IK Ca channel blocker TRAM34 ( a ). MCP‐1 also enhanced migration, and TRAM34 similarly suppressed this increase ( b ). (Statistical significance was confirmed by HC3‐robust interaction model with 6 biological replicates.) B , The ATP‐enhanced migration of Sca‐1 + cells was significantly abolished by IK Ca channel inhibition with shKCNN4 ( P =0.0360, HC3‐robust interaction model, n=5). C , The proliferation of Sca‐1 + cells induced by ATP ( a , b ) and PDGF‐BB ( c ) was significantly suppressed by inhibition of IK Ca channels with TRAM34 or shKCNN4 ( P values are indicated on the graph; HC3‐robust interaction model, n=5). ATP indicates adenosine triphosphate; CON, control; HC3, heteroskedasticity‐consistent robust SE; IK Ca , intermediate conductance calcium‐activated potassium channel; MCP‐1, monocyte chemoattractant protein‐1; PDGF‐BB, platelet‐derived growth factor‐BB; Sca‐1 + , stem cell antigen‐1 positive; shKCNN4, a short hairpin RNA construct specifically designed to target and silence the expression of the KCNN4 gene; and TRAM34, 1‐ [(2‐chlorophenyl) diphenylmethyl]‐1h‐pyrazole.

    Journal: Journal of the American Heart Association: Cardiovascular and Cerebrovascular Disease

    Article Title: Intermediate Conductance Calcium‐Activated Potassium Channel Activation in Stem Cell Antigen‐1 Positive Cells Contributed to Vascular Restenosis

    doi: 10.1161/JAHA.125.045943

    Figure Lengend Snippet: A , Effect on migration. ATP increased Sca‐1 + cell migration, which was significantly inhibited by the IK Ca channel blocker TRAM34 ( a ). MCP‐1 also enhanced migration, and TRAM34 similarly suppressed this increase ( b ). (Statistical significance was confirmed by HC3‐robust interaction model with 6 biological replicates.) B , The ATP‐enhanced migration of Sca‐1 + cells was significantly abolished by IK Ca channel inhibition with shKCNN4 ( P =0.0360, HC3‐robust interaction model, n=5). C , The proliferation of Sca‐1 + cells induced by ATP ( a , b ) and PDGF‐BB ( c ) was significantly suppressed by inhibition of IK Ca channels with TRAM34 or shKCNN4 ( P values are indicated on the graph; HC3‐robust interaction model, n=5). ATP indicates adenosine triphosphate; CON, control; HC3, heteroskedasticity‐consistent robust SE; IK Ca , intermediate conductance calcium‐activated potassium channel; MCP‐1, monocyte chemoattractant protein‐1; PDGF‐BB, platelet‐derived growth factor‐BB; Sca‐1 + , stem cell antigen‐1 positive; shKCNN4, a short hairpin RNA construct specifically designed to target and silence the expression of the KCNN4 gene; and TRAM34, 1‐ [(2‐chlorophenyl) diphenylmethyl]‐1h‐pyrazole.

    Article Snippet: They were then blocked with BSA and incubated overnight with an anti‐ KCNN4 receptor (extracellular) antibody (1:200, Alomone, Jerusalem, Israel, #APR‐102).

    Techniques: Migration, Inhibition, Control, Derivative Assay, shRNA, Construct, Expressing