a h7n9 ha  (Sino Biological)


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    Name:
    Influenza A H7N9 Gene ORF cDNA clone expression plasmid C HA tag
    Description:
    Full length Clone DNA of Influenza A H7N9 A Anhui 1 2013 Neuraminidase with C terminal HA tag
    Catalog Number:
    VG40108-CY
    Price:
    295.0
    Category:
    cDNA Clone
    Applications:
    Stable or Transient mammalian expression
    Size:
    1Unit
    Product Aliases:
    NA cDNA ORF Clone H7N9
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    Structured Review

    Sino Biological a h7n9 ha
    Influenza A H7N9 Gene ORF cDNA clone expression plasmid C HA tag
    Full length Clone DNA of Influenza A H7N9 A Anhui 1 2013 Neuraminidase with C terminal HA tag
    https://www.bioz.com/result/a h7n9 ha/product/Sino Biological
    Average 90 stars, based on 1 article reviews
    Price from $9.99 to $1999.99
    a h7n9 ha - by Bioz Stars, 2021-04
    90/100 stars

    Images

    1) Product Images from "Differences in the Pathogenicity and Inflammatory Responses Induced by Avian Influenza A/H7N9 Virus Infection in BALB/c and C57BL/6 Mouse Models"

    Article Title: Differences in the Pathogenicity and Inflammatory Responses Induced by Avian Influenza A/H7N9 Virus Infection in BALB/c and C57BL/6 Mouse Models

    Journal: PLoS ONE

    doi: 10.1371/journal.pone.0092987

    Innate immune responses induced by H7N9 virus infection in C57BL/6 and BALB/c mice. The experimental setup was the same as that described in Figure 2 . (A–F) Levels of IL-1 β, TNF- α , IL-6, IFN-γ, MCP-1, and KC in mouse sera on days 0, 1, 3, 5, 7, and 14 postviral infection, which were measured by ELISA ( n = 6). The data are expressed as the mean ± SD (bar). *, **, and *** indicate P
    Figure Legend Snippet: Innate immune responses induced by H7N9 virus infection in C57BL/6 and BALB/c mice. The experimental setup was the same as that described in Figure 2 . (A–F) Levels of IL-1 β, TNF- α , IL-6, IFN-γ, MCP-1, and KC in mouse sera on days 0, 1, 3, 5, 7, and 14 postviral infection, which were measured by ELISA ( n = 6). The data are expressed as the mean ± SD (bar). *, **, and *** indicate P

    Techniques Used: Infection, Mouse Assay, Enzyme-linked Immunosorbent Assay

    Lung injury in C57BL/6 and BALB/c mice following H7N9 virus infection. C57BL/6 and BALB/c mice were challenged intranasally with 10 6 TCID 50 of AH1/H7N9 virus and euthanized on days 0, 1, 3, 5, 7, and 14 after viral infection. (A–J) Histological changes in mouse lung tissues on days 0, 1, 3, 5, 7, and 14 postviral challenge. White and yellow arrows indicate the regeneration of pneumocytes and dissociation cells, respectively. (K) Semiquantitative assessments of lung lesions in mice on days 1, 3, 5, and 7 postviral infection ( n = 6). The data are expressed as the mean ± SD (bar). *, **, and *** indicate P
    Figure Legend Snippet: Lung injury in C57BL/6 and BALB/c mice following H7N9 virus infection. C57BL/6 and BALB/c mice were challenged intranasally with 10 6 TCID 50 of AH1/H7N9 virus and euthanized on days 0, 1, 3, 5, 7, and 14 after viral infection. (A–J) Histological changes in mouse lung tissues on days 0, 1, 3, 5, 7, and 14 postviral challenge. White and yellow arrows indicate the regeneration of pneumocytes and dissociation cells, respectively. (K) Semiquantitative assessments of lung lesions in mice on days 1, 3, 5, and 7 postviral infection ( n = 6). The data are expressed as the mean ± SD (bar). *, **, and *** indicate P

    Techniques Used: Mouse Assay, Infection

    H7N9 virus antigen distribution in lung tissues after H7N9 virus infection. (A–H) Immunohistochemical staining of H7N9 virus antigen in the lungs of C57BL/6 and BALB/c mice on days 1, 5, 7, and 14 postviral infection. (I) Dynamic changes in the viral titers in the lungs of C57BL/6 and BALB/c mice on days 1, 3, 5, and 7 postviral infection ( n = 6). The data are expressed as the mean ± SD (bar). *, **, and *** indicate P
    Figure Legend Snippet: H7N9 virus antigen distribution in lung tissues after H7N9 virus infection. (A–H) Immunohistochemical staining of H7N9 virus antigen in the lungs of C57BL/6 and BALB/c mice on days 1, 5, 7, and 14 postviral infection. (I) Dynamic changes in the viral titers in the lungs of C57BL/6 and BALB/c mice on days 1, 3, 5, and 7 postviral infection ( n = 6). The data are expressed as the mean ± SD (bar). *, **, and *** indicate P

    Techniques Used: Infection, Immunohistochemistry, Staining, Mouse Assay

    Weight change and gross pathology of the lungs in C57BL/6 and BALB/c mice infected with H7N9 virus. (A) Weight changes in C57BL/6 and BALB/c mice after challenge with AH/1/H7N9 virus. Healthy young C57BL/6 and BALB/c mice (equal numbers of males and females) were infected intranasally with 10 6 TCID 50 of AH1/H7N9 virus and monitored for 14 days. The body weights were measured daily ( n = 12) and the data are expressed as the mean ± standard deviation (SD). (B) Gross pathology of the lungs from C57BL/6 and BALB/c mice at day 3 postviral infection. The gross lesion area percentage is indicated on the right ( n = 6) and the data are expressed as the mean ± SD (bar). *, **, and *** indicate P
    Figure Legend Snippet: Weight change and gross pathology of the lungs in C57BL/6 and BALB/c mice infected with H7N9 virus. (A) Weight changes in C57BL/6 and BALB/c mice after challenge with AH/1/H7N9 virus. Healthy young C57BL/6 and BALB/c mice (equal numbers of males and females) were infected intranasally with 10 6 TCID 50 of AH1/H7N9 virus and monitored for 14 days. The body weights were measured daily ( n = 12) and the data are expressed as the mean ± standard deviation (SD). (B) Gross pathology of the lungs from C57BL/6 and BALB/c mice at day 3 postviral infection. The gross lesion area percentage is indicated on the right ( n = 6) and the data are expressed as the mean ± SD (bar). *, **, and *** indicate P

    Techniques Used: Mouse Assay, Infection, Standard Deviation

    Dynamic changes in neutrophil infiltration in the lung tissues of mice after H7N9 virus challenge. The experimental setup was the same as that described in Figure 2 . (A–H) Immunohistochemical staining of neutrophils in the lungs of BALB/c and C57BL/6 mice on days 1, 3, 5, and 7 postviral infection. The red arrows indicate infiltrating neutrophils. (I) Semiquantitative assessments of neutrophil infiltration in the lungs on days 1 and 3 postviral infection ( n = 6). The data are expressed as the mean ± SD (bar). * and *** indicate P
    Figure Legend Snippet: Dynamic changes in neutrophil infiltration in the lung tissues of mice after H7N9 virus challenge. The experimental setup was the same as that described in Figure 2 . (A–H) Immunohistochemical staining of neutrophils in the lungs of BALB/c and C57BL/6 mice on days 1, 3, 5, and 7 postviral infection. The red arrows indicate infiltrating neutrophils. (I) Semiquantitative assessments of neutrophil infiltration in the lungs on days 1 and 3 postviral infection ( n = 6). The data are expressed as the mean ± SD (bar). * and *** indicate P

    Techniques Used: Mouse Assay, Immunohistochemistry, Staining, Infection

    Regeneration of lung tissues in mice following H7N9 virus infection. (A–F) Immunohistochemical staining of proliferating cell nuclear antigen (PCNA)-positive cells in the lung tissues of C57BL/6 and BALB/c mice on days 0, 7, and 14 postviral infection. Positive cells were more abundant in the bronchial epithelial cells and interstitial tissues in the lungs. Red arrows indicate PCNA-positive cells.
    Figure Legend Snippet: Regeneration of lung tissues in mice following H7N9 virus infection. (A–F) Immunohistochemical staining of proliferating cell nuclear antigen (PCNA)-positive cells in the lung tissues of C57BL/6 and BALB/c mice on days 0, 7, and 14 postviral infection. Positive cells were more abundant in the bronchial epithelial cells and interstitial tissues in the lungs. Red arrows indicate PCNA-positive cells.

    Techniques Used: Mouse Assay, Infection, Immunohistochemistry, Staining

    Related Articles

    Infection:

    Article Title: Recombinant influenza H7 hemagglutinin containing CFLLC minidomain in the transmembrane domain showed enhanced cross-protection in mice.
    Article Snippet: Since February 2013, H7N9 influenza virus, causing human infections with high mortality in China, has been a potential pandemic threat. .. Since February 2013, H7N9 influenza virus, causing human infections with high mortality in China, has been a potential pandemic threat. .. Since February 2013, H7N9 influenza virus, causing human infections with high mortality in China, has been a potential pandemic threat.

    Incubation:

    Article Title: Recombinant influenza H7 hemagglutinin containing CFLLC minidomain in the transmembrane domain showed enhanced cross-protection in mice.
    Article Snippet: Since February 2013, H7N9 influenza virus, causing human infections with high mortality in China, has been a potential pandemic threat. .. Since February 2013, H7N9 influenza virus, causing human infections with high mortality in China, has been a potential pandemic threat. .. Since February 2013, H7N9 influenza virus, causing human infections with high mortality in China, has been a potential pandemic threat.

    Marker:

    Article Title: Differences in the Pathogenicity and Inflammatory Responses Induced by Avian Influenza A/H7N9 Virus Infection in BALB/c and C57BL/6 Mouse Models
    Article Snippet: Immunohistochemical staining of a neutrophil marker and the hemagglutinin (HA) protein of H7N9 virus in lung tissueFormalin-fixed, paraffin-embedded lung sections were de-paraffinized with xylene and hydrated using a graded alcohol series. .. The changes in the levels of HA protein of A/H7N9 virus and infiltrating neutrophils were assessed using a rabbit polyclonal antibody to A/H7N9 HA (Sino Biological Inc., China) and neutrophil marker NIMP-R14 (Santa Cruz Biotechnology Inc, Santa Cruz, CA), respectively. .. Antibodies were detected using a standard streptavidin-biotin detection system (Beijing Zhongshan Biotechnology Co. Ltd, Beijing, China), according to the manufacturer's instructions.

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    Sino Biological a h7n9 ha
    Innate immune responses induced by <t>H7N9</t> virus infection in C57BL/6 and BALB/c mice. The experimental setup was the same as that described in Figure 2 . (A–F) Levels of IL-1 β, TNF- α , IL-6, IFN-γ, MCP-1, and KC in mouse sera on days 0, 1, 3, 5, 7, and 14 postviral infection, which were measured by ELISA ( n = 6). The data are expressed as the mean ± SD (bar). *, **, and *** indicate P
    A H7n9 Ha, supplied by Sino Biological, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/a h7n9 ha/product/Sino Biological
    Average 90 stars, based on 1 article reviews
    Price from $9.99 to $1999.99
    a h7n9 ha - by Bioz Stars, 2021-04
    90/100 stars
      Buy from Supplier

    Image Search Results


    Innate immune responses induced by H7N9 virus infection in C57BL/6 and BALB/c mice. The experimental setup was the same as that described in Figure 2 . (A–F) Levels of IL-1 β, TNF- α , IL-6, IFN-γ, MCP-1, and KC in mouse sera on days 0, 1, 3, 5, 7, and 14 postviral infection, which were measured by ELISA ( n = 6). The data are expressed as the mean ± SD (bar). *, **, and *** indicate P

    Journal: PLoS ONE

    Article Title: Differences in the Pathogenicity and Inflammatory Responses Induced by Avian Influenza A/H7N9 Virus Infection in BALB/c and C57BL/6 Mouse Models

    doi: 10.1371/journal.pone.0092987

    Figure Lengend Snippet: Innate immune responses induced by H7N9 virus infection in C57BL/6 and BALB/c mice. The experimental setup was the same as that described in Figure 2 . (A–F) Levels of IL-1 β, TNF- α , IL-6, IFN-γ, MCP-1, and KC in mouse sera on days 0, 1, 3, 5, 7, and 14 postviral infection, which were measured by ELISA ( n = 6). The data are expressed as the mean ± SD (bar). *, **, and *** indicate P

    Article Snippet: The changes in the levels of HA protein of A/H7N9 virus and infiltrating neutrophils were assessed using a rabbit polyclonal antibody to A/H7N9 HA (Sino Biological Inc., China) and neutrophil marker NIMP-R14 (Santa Cruz Biotechnology Inc, Santa Cruz, CA), respectively.

    Techniques: Infection, Mouse Assay, Enzyme-linked Immunosorbent Assay

    Lung injury in C57BL/6 and BALB/c mice following H7N9 virus infection. C57BL/6 and BALB/c mice were challenged intranasally with 10 6 TCID 50 of AH1/H7N9 virus and euthanized on days 0, 1, 3, 5, 7, and 14 after viral infection. (A–J) Histological changes in mouse lung tissues on days 0, 1, 3, 5, 7, and 14 postviral challenge. White and yellow arrows indicate the regeneration of pneumocytes and dissociation cells, respectively. (K) Semiquantitative assessments of lung lesions in mice on days 1, 3, 5, and 7 postviral infection ( n = 6). The data are expressed as the mean ± SD (bar). *, **, and *** indicate P

    Journal: PLoS ONE

    Article Title: Differences in the Pathogenicity and Inflammatory Responses Induced by Avian Influenza A/H7N9 Virus Infection in BALB/c and C57BL/6 Mouse Models

    doi: 10.1371/journal.pone.0092987

    Figure Lengend Snippet: Lung injury in C57BL/6 and BALB/c mice following H7N9 virus infection. C57BL/6 and BALB/c mice were challenged intranasally with 10 6 TCID 50 of AH1/H7N9 virus and euthanized on days 0, 1, 3, 5, 7, and 14 after viral infection. (A–J) Histological changes in mouse lung tissues on days 0, 1, 3, 5, 7, and 14 postviral challenge. White and yellow arrows indicate the regeneration of pneumocytes and dissociation cells, respectively. (K) Semiquantitative assessments of lung lesions in mice on days 1, 3, 5, and 7 postviral infection ( n = 6). The data are expressed as the mean ± SD (bar). *, **, and *** indicate P

    Article Snippet: The changes in the levels of HA protein of A/H7N9 virus and infiltrating neutrophils were assessed using a rabbit polyclonal antibody to A/H7N9 HA (Sino Biological Inc., China) and neutrophil marker NIMP-R14 (Santa Cruz Biotechnology Inc, Santa Cruz, CA), respectively.

    Techniques: Mouse Assay, Infection

    H7N9 virus antigen distribution in lung tissues after H7N9 virus infection. (A–H) Immunohistochemical staining of H7N9 virus antigen in the lungs of C57BL/6 and BALB/c mice on days 1, 5, 7, and 14 postviral infection. (I) Dynamic changes in the viral titers in the lungs of C57BL/6 and BALB/c mice on days 1, 3, 5, and 7 postviral infection ( n = 6). The data are expressed as the mean ± SD (bar). *, **, and *** indicate P

    Journal: PLoS ONE

    Article Title: Differences in the Pathogenicity and Inflammatory Responses Induced by Avian Influenza A/H7N9 Virus Infection in BALB/c and C57BL/6 Mouse Models

    doi: 10.1371/journal.pone.0092987

    Figure Lengend Snippet: H7N9 virus antigen distribution in lung tissues after H7N9 virus infection. (A–H) Immunohistochemical staining of H7N9 virus antigen in the lungs of C57BL/6 and BALB/c mice on days 1, 5, 7, and 14 postviral infection. (I) Dynamic changes in the viral titers in the lungs of C57BL/6 and BALB/c mice on days 1, 3, 5, and 7 postviral infection ( n = 6). The data are expressed as the mean ± SD (bar). *, **, and *** indicate P

    Article Snippet: The changes in the levels of HA protein of A/H7N9 virus and infiltrating neutrophils were assessed using a rabbit polyclonal antibody to A/H7N9 HA (Sino Biological Inc., China) and neutrophil marker NIMP-R14 (Santa Cruz Biotechnology Inc, Santa Cruz, CA), respectively.

    Techniques: Infection, Immunohistochemistry, Staining, Mouse Assay

    Weight change and gross pathology of the lungs in C57BL/6 and BALB/c mice infected with H7N9 virus. (A) Weight changes in C57BL/6 and BALB/c mice after challenge with AH/1/H7N9 virus. Healthy young C57BL/6 and BALB/c mice (equal numbers of males and females) were infected intranasally with 10 6 TCID 50 of AH1/H7N9 virus and monitored for 14 days. The body weights were measured daily ( n = 12) and the data are expressed as the mean ± standard deviation (SD). (B) Gross pathology of the lungs from C57BL/6 and BALB/c mice at day 3 postviral infection. The gross lesion area percentage is indicated on the right ( n = 6) and the data are expressed as the mean ± SD (bar). *, **, and *** indicate P

    Journal: PLoS ONE

    Article Title: Differences in the Pathogenicity and Inflammatory Responses Induced by Avian Influenza A/H7N9 Virus Infection in BALB/c and C57BL/6 Mouse Models

    doi: 10.1371/journal.pone.0092987

    Figure Lengend Snippet: Weight change and gross pathology of the lungs in C57BL/6 and BALB/c mice infected with H7N9 virus. (A) Weight changes in C57BL/6 and BALB/c mice after challenge with AH/1/H7N9 virus. Healthy young C57BL/6 and BALB/c mice (equal numbers of males and females) were infected intranasally with 10 6 TCID 50 of AH1/H7N9 virus and monitored for 14 days. The body weights were measured daily ( n = 12) and the data are expressed as the mean ± standard deviation (SD). (B) Gross pathology of the lungs from C57BL/6 and BALB/c mice at day 3 postviral infection. The gross lesion area percentage is indicated on the right ( n = 6) and the data are expressed as the mean ± SD (bar). *, **, and *** indicate P

    Article Snippet: The changes in the levels of HA protein of A/H7N9 virus and infiltrating neutrophils were assessed using a rabbit polyclonal antibody to A/H7N9 HA (Sino Biological Inc., China) and neutrophil marker NIMP-R14 (Santa Cruz Biotechnology Inc, Santa Cruz, CA), respectively.

    Techniques: Mouse Assay, Infection, Standard Deviation

    Dynamic changes in neutrophil infiltration in the lung tissues of mice after H7N9 virus challenge. The experimental setup was the same as that described in Figure 2 . (A–H) Immunohistochemical staining of neutrophils in the lungs of BALB/c and C57BL/6 mice on days 1, 3, 5, and 7 postviral infection. The red arrows indicate infiltrating neutrophils. (I) Semiquantitative assessments of neutrophil infiltration in the lungs on days 1 and 3 postviral infection ( n = 6). The data are expressed as the mean ± SD (bar). * and *** indicate P

    Journal: PLoS ONE

    Article Title: Differences in the Pathogenicity and Inflammatory Responses Induced by Avian Influenza A/H7N9 Virus Infection in BALB/c and C57BL/6 Mouse Models

    doi: 10.1371/journal.pone.0092987

    Figure Lengend Snippet: Dynamic changes in neutrophil infiltration in the lung tissues of mice after H7N9 virus challenge. The experimental setup was the same as that described in Figure 2 . (A–H) Immunohistochemical staining of neutrophils in the lungs of BALB/c and C57BL/6 mice on days 1, 3, 5, and 7 postviral infection. The red arrows indicate infiltrating neutrophils. (I) Semiquantitative assessments of neutrophil infiltration in the lungs on days 1 and 3 postviral infection ( n = 6). The data are expressed as the mean ± SD (bar). * and *** indicate P

    Article Snippet: The changes in the levels of HA protein of A/H7N9 virus and infiltrating neutrophils were assessed using a rabbit polyclonal antibody to A/H7N9 HA (Sino Biological Inc., China) and neutrophil marker NIMP-R14 (Santa Cruz Biotechnology Inc, Santa Cruz, CA), respectively.

    Techniques: Mouse Assay, Immunohistochemistry, Staining, Infection

    Regeneration of lung tissues in mice following H7N9 virus infection. (A–F) Immunohistochemical staining of proliferating cell nuclear antigen (PCNA)-positive cells in the lung tissues of C57BL/6 and BALB/c mice on days 0, 7, and 14 postviral infection. Positive cells were more abundant in the bronchial epithelial cells and interstitial tissues in the lungs. Red arrows indicate PCNA-positive cells.

    Journal: PLoS ONE

    Article Title: Differences in the Pathogenicity and Inflammatory Responses Induced by Avian Influenza A/H7N9 Virus Infection in BALB/c and C57BL/6 Mouse Models

    doi: 10.1371/journal.pone.0092987

    Figure Lengend Snippet: Regeneration of lung tissues in mice following H7N9 virus infection. (A–F) Immunohistochemical staining of proliferating cell nuclear antigen (PCNA)-positive cells in the lung tissues of C57BL/6 and BALB/c mice on days 0, 7, and 14 postviral infection. Positive cells were more abundant in the bronchial epithelial cells and interstitial tissues in the lungs. Red arrows indicate PCNA-positive cells.

    Article Snippet: The changes in the levels of HA protein of A/H7N9 virus and infiltrating neutrophils were assessed using a rabbit polyclonal antibody to A/H7N9 HA (Sino Biological Inc., China) and neutrophil marker NIMP-R14 (Santa Cruz Biotechnology Inc, Santa Cruz, CA), respectively.

    Techniques: Mouse Assay, Infection, Immunohistochemistry, Staining