α myosin heavy chain peptide (MedChemExpress)
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MedChemExpress
α myosin heavy chain peptide
α Myosin Heavy Chain Peptide, supplied by MedChemExpress, used in various techniques. Bioz Stars score: 93/100, based on 2 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/hy-p2464/Myosin+H+Chain+Fragment%2C+mouse/10__1002_slash_adfm__202501018-458-16-21
Average 93 stars, based on 2 article reviews
α Myosin Heavy Chain Peptide, supplied by MedChemExpress, used in various techniques. Bioz Stars score: 93/100, based on 2 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/hy-p2464/Myosin+H+Chain+Fragment%2C+mouse/10__1002_slash_adfm__202501018-458-16-21
Average 93 stars, based on 2 article reviews
α myosin heavy chain peptide - by Bioz Stars,
2026-10
93/100 stars
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Related Articles
CCK-8 Assay:Article Title: Bifunctional Peptide‐Engineered Mitochondria Alleviate Myocarditis and Cardiac Fibrosis by Site‐Specifically Inhibiting NETosis Article Snippet: Myocarditis is a leading cause of sudden cardiac death and dilated cardiomyopathy in young adults, and effective therapies are urgently needed.. Here, it is found that intravenous transplantation of exogenous mitochondria (Mito) significantly attenuates inflammation and oxidative stress in the heart, mitigates myocardial injury, improves cardiac function, and alleviates fibrosis and heart failure in mice with experimental autoimmune myocarditis (EAM).. Mechanistically, injected Mito can be internalized by neutrophils and subsequently transported to the inflamed hearts of EAM mice. Mouse Assay:Article Title: Bifunctional Peptide‐Engineered Mitochondria Alleviate Myocarditis and Cardiac Fibrosis by Site‐Specifically Inhibiting NETosis Article Snippet: Myocarditis is a leading cause of sudden cardiac death and dilated cardiomyopathy in young adults, and effective therapies are urgently needed.. Here, it is found that intravenous transplantation of exogenous mitochondria (Mito) significantly attenuates inflammation and oxidative stress in the heart, mitigates myocardial injury, improves cardiac function, and alleviates fibrosis and heart failure in mice with experimental autoimmune myocarditis (EAM).. Mechanistically, injected Mito can be internalized by neutrophils and subsequently transported to the inflamed hearts of EAM mice. Adjuvant:Article Title: Multifunctional nanoparticles for immune regulation and oxidative stress alleviation in myocarditis. Article Snippet: Cardiac autoimmune injury and oxidative stress play critical roles in the development of myocarditis.. Promising approaches for treating this condition include suppressing excessive immune responses and reducing oxidative stress in the myocardium.. The programmed cell death protein 1/programmed cell death ligand 1 (PD-1/PD-L1) axis is known to regulate immune responses and prevent damage caused by T-cell overactivation, while elevated reactive oxygen species (ROS) contribute to the progression of myocarditis. Injection:Article Title: Multifunctional nanoparticles for immune regulation and oxidative stress alleviation in myocarditis. Article Snippet: Cardiac autoimmune injury and oxidative stress play critical roles in the development of myocarditis.. Promising approaches for treating this condition include suppressing excessive immune responses and reducing oxidative stress in the myocardium.. The programmed cell death protein 1/programmed cell death ligand 1 (PD-1/PD-L1) axis is known to regulate immune responses and prevent damage caused by T-cell overactivation, while elevated reactive oxygen species (ROS) contribute to the progression of myocarditis. |