senktide (MedChemExpress)
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MedChemExpress
senktide
Senktide, supplied by MedChemExpress, used in various techniques. Bioz Stars score: 94/100, based on 5 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/hy-p0187/Senktide/pm38011644-311-0-4
Average 94 stars, based on 5 article reviews
Senktide, supplied by MedChemExpress, used in various techniques. Bioz Stars score: 94/100, based on 5 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/hy-p0187/Senktide/pm38011644-311-0-4
Average 94 stars, based on 5 article reviews
senktide - by Bioz Stars,
2026-10
94/100 stars
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Transferring:Article Title: Senktide blocks aberrant RTN3 interactome to retard memory decline and tau pathology in social isolated Alzheimer's disease mice. Article Snippet: .. Article Title: Senktide blocks aberrant RTN3 interactome to retard memory decline and tau pathology in social isolated Alzheimer’s disease mice Article Snippet: .. Isolation:Article Title: Senktide blocks aberrant RTN3 interactome to retard memory decline and tau pathology in social isolated Alzheimer's disease mice. Article Snippet: .. Article Title: Senktide blocks aberrant RTN3 interactome to retard memory decline and tau pathology in social isolated Alzheimer’s disease mice Article Snippet: .. other:Article Title: Deletion of<i>Stim1</i>in Hypothalamic Arcuate Nucleus Kiss1 Neurons Potentiates Synchronous GCaMP Activity and Protects against Diet-Induced Obesity Article Snippet: Thapsigargin (Tg; 2 mM), Picrotoxin (100 mM), TacR3 agonist senktide (1 mM), and TRPC4/5 antagonist, Generated:Article Title: Tachykinin signaling defines distinct populations of glia in the enteric nervous system. Article Snippet: Article Tachykinin signaling defines distinct populations of glia in the enteric nervous system Recombinant:Article Title: Tachykinin signaling defines distinct populations of glia in the enteric nervous system. Article Snippet: Article Tachykinin signaling defines distinct populations of glia in the enteric nervous system Electron Microscopy:Article Title: Tachykinin signaling defines distinct populations of glia in the enteric nervous system. Article Snippet: Article Tachykinin signaling defines distinct populations of glia in the enteric nervous system Saline:Article Title: Tachykinin signaling defines distinct populations of glia in the enteric nervous system. Article Snippet: Article Tachykinin signaling defines distinct populations of glia in the enteric nervous system Article Title: Neurokinin 3 receptor agonist senktide stimulates GnRH release in isolated hypogonadotropic hypogonadism mice Article Snippet: Isolated hypogonadotropic hypogonadism (IHH) is caused by defective gonadotropinreleasing hormone (GnRH) secretion or action, resulting in absent or incomplete pubertal development and infertility.1 Timely release of GnRH is critical for the onset of puberty and subsequent sexual maturation.. Misregulation in the GnRH axis can result in delayed or absent puberty and infertility.2 IHH is divided into anosmic hypogonadotropic hypogonadism (Kallmann syndrome) and normosmic isolated hypogonadotropic hypogonadism (nIHH).3 The incidence of IHH is approximately 1–10 in 100 000 live births.4 IHH is genetically heterogeneous, with both X chromosomelinked recessive, autosomal recessive, and dominant forms.. More than 25 different genes have been implicated in IHH, which accounts for about 50% of cases, including KAL1, FGFR1, FGF8, CHD7, HS6ST1, SOX10, SEMA3A, WDR11, IL17RD, PROKR2, and PROK2.5 Unlike other genes, the inheritance pattern of prokineticin receptor 2 (PROKR2) and prokineticin 2 (PROK2) in IHH is autosomal recessive, with most patients carrying PROKR2 or PROK2 mutations in a heterozygous state.6,7 Genetic data from IHH Received: 24 December 2024 | Revised: 20 February 2025 | Accepted: 19 March 2025 DOI: 10.1096/fj.202403365R RNAscope:Article Title: Tachykinin signaling defines distinct populations of glia in the enteric nervous system. Article Snippet: Article Tachykinin signaling defines distinct populations of glia in the enteric nervous system In Vitro:Article Title: Neurokinin 3 receptor agonist senktide stimulates GnRH release in isolated hypogonadotropic hypogonadism mice Article Snippet: Isolated hypogonadotropic hypogonadism (IHH) is caused by defective gonadotropinreleasing hormone (GnRH) secretion or action, resulting in absent or incomplete pubertal development and infertility.1 Timely release of GnRH is critical for the onset of puberty and subsequent sexual maturation.. Misregulation in the GnRH axis can result in delayed or absent puberty and infertility.2 IHH is divided into anosmic hypogonadotropic hypogonadism (Kallmann syndrome) and normosmic isolated hypogonadotropic hypogonadism (nIHH).3 The incidence of IHH is approximately 1–10 in 100 000 live births.4 IHH is genetically heterogeneous, with both X chromosomelinked recessive, autosomal recessive, and dominant forms.. More than 25 different genes have been implicated in IHH, which accounts for about 50% of cases, including KAL1, FGFR1, FGF8, CHD7, HS6ST1, SOX10, SEMA3A, WDR11, IL17RD, PROKR2, and PROK2.5 Unlike other genes, the inheritance pattern of prokineticin receptor 2 (PROKR2) and prokineticin 2 (PROK2) in IHH is autosomal recessive, with most patients carrying PROKR2 or PROK2 mutations in a heterozygous state.6,7 Genetic data from IHH Received: 24 December 2024 | Revised: 20 February 2025 | Accepted: 19 March 2025 DOI: 10.1096/fj.202403365R In Vivo:Article Title: Neurokinin 3 receptor agonist senktide stimulates GnRH release in isolated hypogonadotropic hypogonadism mice Article Snippet: Isolated hypogonadotropic hypogonadism (IHH) is caused by defective gonadotropinreleasing hormone (GnRH) secretion or action, resulting in absent or incomplete pubertal development and infertility.1 Timely release of GnRH is critical for the onset of puberty and subsequent sexual maturation.. Misregulation in the GnRH axis can result in delayed or absent puberty and infertility.2 IHH is divided into anosmic hypogonadotropic hypogonadism (Kallmann syndrome) and normosmic isolated hypogonadotropic hypogonadism (nIHH).3 The incidence of IHH is approximately 1–10 in 100 000 live births.4 IHH is genetically heterogeneous, with both X chromosomelinked recessive, autosomal recessive, and dominant forms.. More than 25 different genes have been implicated in IHH, which accounts for about 50% of cases, including KAL1, FGFR1, FGF8, CHD7, HS6ST1, SOX10, SEMA3A, WDR11, IL17RD, PROKR2, and PROK2.5 Unlike other genes, the inheritance pattern of prokineticin receptor 2 (PROKR2) and prokineticin 2 (PROK2) in IHH is autosomal recessive, with most patients carrying PROKR2 or PROK2 mutations in a heterozygous state.6,7 Genetic data from IHH Received: 24 December 2024 | Revised: 20 February 2025 | Accepted: 19 March 2025 DOI: 10.1096/fj.202403365R |