dimethyl citrate (MedChemExpress)
93
Structured Review
MedChemExpress
dimethyl citrate
Dimethyl Citrate, supplied by MedChemExpress, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/hy-n9542/Dimethyl+Citric+acid/pmc11914110-8-0-3
Average 93 stars, based on 1 article reviews
Dimethyl Citrate, supplied by MedChemExpress, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/hy-n9542/Dimethyl+Citric+acid/pmc11914110-8-0-3
Average 93 stars, based on 1 article reviews
dimethyl citrate - by Bioz Stars,
2026-10
93/100 stars
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other:Article Title: SLC25A1 and ACLY maintain cytosolic acetyl-CoA and regulate ferroptosis susceptibility via FSP1 acetylation Article Snippet: The cells were treated with reagents including the ferroptosis inducers RSL3 (Selleck, S8155) and erastin (Selleck, S7242), ferroptosis inhibitors Ferrostatin-1 (Fer-1) (Selleck, S7243), apoptosis inhibitor Z-VAD-FMK (Z-V) (Selleck, S7023), necrosis inhibitor Necrostatin-1 (Nec-1) (Selleck, S8037), autophagy inhibitor Bafilomycin A1 (Baf-A1) (Selleck, S1413), SLC25A1 inhibitor BTA (Macklin, B801861), ACLY inhibitor SB204990 (Targetmol, T16861), Article Title: SLC25A1 and ACLY maintain cytosolic acetyl-CoA and regulate ferroptosis susceptibility via FSP1 acetylation Article Snippet: Article Title: SLC25A1 and ACLY maintain cytosolic acetyl-CoA and regulate ferroptosis susceptibility via FSP1 acetylation. Article Snippet: Ferroptosis, an iron-dependent form of programmed cell death characterized by excessive lipid hydroperoxides accumulation, emerges as a promising target in cancer therapy.. Among the solute carrier (SLC) superfamily, the cystine/glutamate transporter system antiporter components SLC3A2 and SLC7A11 are known to regulate ferroptosis by facilitating cystine import for ferroptosis inhibition.. However, the contribution of additional SLC superfamily members to ferroptosis remains poorly understood. |