Review




Structured Review

Polysciences inc diffquick kit
Diffquick Kit, supplied by Polysciences inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/diff-quick+ii+staining+kit/diffquick+kit/pmc04352037-57-9-11
Average 90 stars, based on 1 article reviews
diffquick kit - by Bioz Stars, 2026-09
90/100 stars

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Related Articles

Staining:

Article Title: Pulmonary Toxicity of Instilled Silver Nanoparticles: Influence of Size, Coating and Rat Strain
Article Snippet: .. Staining for differential cell count was performed using the DiffQuick kit (Polysciences Inc). ..

Article Title: Diesel exhaust particulate associated chemicals attenuate expression of CXCL10 in human primary bronchial epithelial cells.
Article Snippet: Air pollution affects a large proportion of the population particularly in urban areas, with diesel particulates recognised as particular causes for concern in respiratory conditions such as asthma.. In this study we examined the response of human primary airway epithelial cells to diesel particulate chemical extracts (DE) and characterised gene expression alterations using RNA-SEQ.. Using the antagonist CH223191, DE induced CYP1A1 and attenuation of CXCL10 among other genes were observed to be aryl hydrocarbon receptor dependent.

Cell Characterization:

Article Title: Pulmonary Toxicity of Instilled Silver Nanoparticles: Influence of Size, Coating and Rat Strain
Article Snippet: .. Staining for differential cell count was performed using the DiffQuick kit (Polysciences Inc). ..

Cell Counting:

Article Title: Diesel exhaust particulate associated chemicals attenuate expression of CXCL10 in human primary bronchial epithelial cells.
Article Snippet: Air pollution affects a large proportion of the population particularly in urban areas, with diesel particulates recognised as particular causes for concern in respiratory conditions such as asthma.. In this study we examined the response of human primary airway epithelial cells to diesel particulate chemical extracts (DE) and characterised gene expression alterations using RNA-SEQ.. Using the antagonist CH223191, DE induced CYP1A1 and attenuation of CXCL10 among other genes were observed to be aryl hydrocarbon receptor dependent.



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