p65 (Santa Cruz Biotechnology)
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Santa Cruz Biotechnology
p65
P65, supplied by Santa Cruz Biotechnology, used in various techniques. Bioz Stars score: 96/100, based on 14412 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/SC-8008/NF%CE%BAB+p65+Antibody/pm41904146-250-65-67
Average 96 stars, based on 14412 article reviews
P65, supplied by Santa Cruz Biotechnology, used in various techniques. Bioz Stars score: 96/100, based on 14412 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/SC-8008/NF%CE%BAB+p65+Antibody/pm41904146-250-65-67
Average 96 stars, based on 14412 article reviews
p65 - by Bioz Stars,
2026-09
96/100 stars
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Incubation:Article Title: O-GlcNAcylation regulates microglial neuroinflammation in Parkinson's disease. Article Snippet: Western Blotting AR TI CL E IN P RE SS Equal amounts of protein (30 μg) were separated on 10% or 15% SDS-PAGE gels and transferred to nitrocellulose membranes (Amersham). .. Membranes were blocked with 5% nonfat milk in PBST for 1 h at room temperature, then incubated overnight at 4°C with primary antibodies (1:1,000) against various targets, including O-GlcNAc (sc-59623, RL2, Santa Cruz, Dallas, TX, USA), OGT (sc-74546, Santa Cruz), OGA (14771-1-AP, Proteintech, Rosemont, IL, USA), GFAT2 (A15374, Abclonal, Woburn, MA, USA) tyrosine hydroxylase (T2928,TH, Sigma-Aldrich), β-actin (sc28365, Santa Cruz), Article Title: Targeting the DSTYK-ULK1 axis rewires TNFR1 signaling to overcome treatment resistance in lung cancer. Article Snippet: Electrophoresis was performed in a running 10× Tris-Glycine SDS buffer, and nitrocellulose membranes were used for proteins transfer (#1620112; Bio-Rad). .. Membranes were incubated overnight with the indicated antibodies: Rabbit anti-DSTYK (Novus Biologicals, NBP1-92336, 1:1000); Rabbit anti-Total ULK1 (Cell Signaling Technology, 8054, 1:1000); Rabbit anti-p-ULK1 (Cell Signaling Technology, 37762, 1:1000); Rabbit anti-p-ULK1 (ThermoFisher Scientific, PA5-105421, 1:1000); Rabbit anti-p-ULK1 (Abcam, ab131210, 1:1000); Rabbit anti-pRIPK1 (Cell Signaling Technology, 44590, 1:1000); Rabbit anti-Total RIPK1 (Cell Signaling Technology, 3493, 1:1000); Rabbit antiTotal RIPK3 (Human) (Cell Signaling Technology, 13526, 1:1000); Rabbit anti-Total RIPK3 (Mouse) (Cell Signaling Technology, 95702, 1:1000); Rabbit anti-p-RIPK3 (Human) (Cell Signaling Technology, 93654, 1:1000); Rabbit anti-p-RIPK3 (Mouse) (Cell Signaling Technology, 91702, 1:1000); Article Title: Two arabinans APRP70-1-1 and APRP70-1-2 from Angelica pubescens: structural characterization and immunomodulatory mechanism Article Snippet: This is a PDF of an article that has undergone enhancements after acceptance, such as the addition of a cover page and metadata, and formatting for readability.. This version will undergo additional copyediting, typesetting and review before it is published in its final form.. As such, this version is no longer the Accepted Manuscript, but it is not yet the definitive Version of Record; we are providing this early version to give early visibility of the article. Molecular Weight:Article Title: Orphan G protein-coupled receptor 3 as a potential modulator of microglial inflammatory responses: implications for tau-associated neuroinflammation in Alzheimer's disease. Article Snippet: Introduction: Neuroinflammation, driven largely by microglial activation, is a core pathological feature of Alzheimer's disease (AD).. Pathological tau species represent one of the key drivers of inflammatory activation in AD; however, the upstream regulators governing microglial inflammatory responses remain incompletely understood.. Orphan G protein–coupled receptors (GPCRs) are emerging neuroimmune modulators, yet their specific roles in tau-mediated neuroinflammation are not fully characterized. Expressing:Article Title: Orphan G protein-coupled receptor 3 as a potential modulator of microglial inflammatory responses: implications for tau-associated neuroinflammation in Alzheimer's disease. Article Snippet: Introduction: Neuroinflammation, driven largely by microglial activation, is a core pathological feature of Alzheimer's disease (AD).. Pathological tau species represent one of the key drivers of inflammatory activation in AD; however, the upstream regulators governing microglial inflammatory responses remain incompletely understood.. Orphan G protein–coupled receptors (GPCRs) are emerging neuroimmune modulators, yet their specific roles in tau-mediated neuroinflammation are not fully characterized. Staining:Article Title: Two arabinans APRP70-1-1 and APRP70-1-2 from Angelica pubescens: structural characterization and immunomodulatory mechanism Article Snippet: This is a PDF of an article that has undergone enhancements after acceptance, such as the addition of a cover page and metadata, and formatting for readability.. This version will undergo additional copyediting, typesetting and review before it is published in its final form.. As such, this version is no longer the Accepted Manuscript, but it is not yet the definitive Version of Record; we are providing this early version to give early visibility of the article. |
