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decanoylcarnitine chloride c10  (Tocris)


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    Tocris decanoylcarnitine chloride c10
    Decanoylcarnitine Chloride C10, supplied by Tocris, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/0477/(%2B%2F-)-Decanoylcarnitine+chloride/pm25770415-39-32-47
    Average 90 stars, based on 1 article reviews
    decanoylcarnitine chloride c10 - by Bioz Stars, 2026-09
    90/100 stars

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    Related Articles

    Irradiation:

    Article Title: Development of a Metabolomic Radiation Signature in Urine from Patients Undergoing Total Body Irradiation
    Article Snippet: Debrisoquine sulfate, 4-nitrobenzoic acid (4-NBA), creatinine, uric acid, hypoxanthine, xanthine and Nε,Nε,Nε-Trimethyl- l -lysine hydrochloride (Trimethyl- l -lysine or TML) were obtained from Sigma-Aldrich (St. Louis, MO). (±)-Decanoylcarnitine chloride (Decanoylcarnitine) and (±)-Octanoylcarnitine chloride (Octanoylcarnitine) were obtained from Tocris Bioscience (Ellisville, MO).

    Article Title: Development of a Metabolomic Radiation Signature in Urine from Patients Undergoing Total Body Irradiation
    Article Snippet: Debrisoquine sulfate, 4-nitrobenzoic acid (4- NBA), creatinine, uric acid, hypoxanthine, xanthine and Ne,Ne,NeTrimethyl-L-lysine hydrochloride (Trimethyl-L-lysine or TML) were obtained from Sigma-Aldrich (St. Louis, MO). (6)-Decanoylcarnitine chloride (Decanoylcarnitine) and (6)-Octanoylcarnitine chloride (Octanoylcarnitine) were obtained from Tocris Bioscience (Ellisville, MO).

    Comparison:

    Article Title: Development of a Metabolomic Radiation Signature in Urine from Patients Undergoing Total Body Irradiation
    Article Snippet: Debrisoquine sulfate, 4-nitrobenzoic acid (4-NBA), creatinine, uric acid, hypoxanthine, xanthine and Nε,Nε,Nε-Trimethyl- l -lysine hydrochloride (Trimethyl- l -lysine or TML) were obtained from Sigma-Aldrich (St. Louis, MO). (±)-Decanoylcarnitine chloride (Decanoylcarnitine) and (±)-Octanoylcarnitine chloride (Octanoylcarnitine) were obtained from Tocris Bioscience (Ellisville, MO).

    Article Title: Development of a Metabolomic Radiation Signature in Urine from Patients Undergoing Total Body Irradiation
    Article Snippet: Debrisoquine sulfate, 4-nitrobenzoic acid (4- NBA), creatinine, uric acid, hypoxanthine, xanthine and Ne,Ne,NeTrimethyl-L-lysine hydrochloride (Trimethyl-L-lysine or TML) were obtained from Sigma-Aldrich (St. Louis, MO). (6)-Decanoylcarnitine chloride (Decanoylcarnitine) and (6)-Octanoylcarnitine chloride (Octanoylcarnitine) were obtained from Tocris Bioscience (Ellisville, MO).

    Mouse Assay:

    Article Title: Development of a Metabolomic Radiation Signature in Urine from Patients Undergoing Total Body Irradiation
    Article Snippet: Debrisoquine sulfate, 4-nitrobenzoic acid (4-NBA), creatinine, uric acid, hypoxanthine, xanthine and Nε,Nε,Nε-Trimethyl- l -lysine hydrochloride (Trimethyl- l -lysine or TML) were obtained from Sigma-Aldrich (St. Louis, MO). (±)-Decanoylcarnitine chloride (Decanoylcarnitine) and (±)-Octanoylcarnitine chloride (Octanoylcarnitine) were obtained from Tocris Bioscience (Ellisville, MO).

    Article Title: Development of a Metabolomic Radiation Signature in Urine from Patients Undergoing Total Body Irradiation
    Article Snippet: Debrisoquine sulfate, 4-nitrobenzoic acid (4- NBA), creatinine, uric acid, hypoxanthine, xanthine and Ne,Ne,NeTrimethyl-L-lysine hydrochloride (Trimethyl-L-lysine or TML) were obtained from Sigma-Aldrich (St. Louis, MO). (6)-Decanoylcarnitine chloride (Decanoylcarnitine) and (6)-Octanoylcarnitine chloride (Octanoylcarnitine) were obtained from Tocris Bioscience (Ellisville, MO).



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    Image Search Results


    HIF inhibits CRC cells growth. (A, B) Cell viability of HCT15 and HCT116 cells treated with trans‐coumaroyltyramine (trans‐Coum), N‐feruloyloctopamine (Feru), N‐trans‐feruloyltyramine (trans‐Feru), disporopsin (Disp), or HIF for 24 h. (C) IC50 values of HIF in HCT15 and HCT116 cells. (D) Cell migration analysis of HCT15 and HCT116 cells following HIF treatment. (E) Colony formation assessment of HCT15 and HCT116 cells after HIF treatment for 24 h. (F) Apoptosis analysis of HCT15 and HCT116 cells after 24 h of HIF treatment. (G, H) Cell cycle distribution analysis of HCT15 and HCT116 cells following HIF treatment for 24 h. (I) Expression levels of cell cycle proteins (Cyclin D1, Cyclin A2, CDK1, CDK2, CDK7) and apoptosis markers (c‐Caspase3, c‐Caspase8). (J, K) The impact of Z‐VAD‐FMK (Z‐VAD) co‐treatment on HIF‐induced cell death in HCT15 and HCT116 cells. ** p < 0.01, *** p < 0.001, **** p < 0.0001; ns, not significant.

    Journal: Advanced Science

    Article Title: Homoisoflavanone Delays Colorectal Cancer Progression via DNA Damage‐Induced Mitochondrial Apoptosis and Parthanatos‐Like Cell Death

    doi: 10.1002/advs.202511406

    Figure Lengend Snippet: HIF inhibits CRC cells growth. (A, B) Cell viability of HCT15 and HCT116 cells treated with trans‐coumaroyltyramine (trans‐Coum), N‐feruloyloctopamine (Feru), N‐trans‐feruloyltyramine (trans‐Feru), disporopsin (Disp), or HIF for 24 h. (C) IC50 values of HIF in HCT15 and HCT116 cells. (D) Cell migration analysis of HCT15 and HCT116 cells following HIF treatment. (E) Colony formation assessment of HCT15 and HCT116 cells after HIF treatment for 24 h. (F) Apoptosis analysis of HCT15 and HCT116 cells after 24 h of HIF treatment. (G, H) Cell cycle distribution analysis of HCT15 and HCT116 cells following HIF treatment for 24 h. (I) Expression levels of cell cycle proteins (Cyclin D1, Cyclin A2, CDK1, CDK2, CDK7) and apoptosis markers (c‐Caspase3, c‐Caspase8). (J, K) The impact of Z‐VAD‐FMK (Z‐VAD) co‐treatment on HIF‐induced cell death in HCT15 and HCT116 cells. ** p < 0.01, *** p < 0.001, **** p < 0.0001; ns, not significant.

    Article Snippet: The structures of N‐feruloyloctopamine, N‐p‐trans‐coumaroyltyramine, N‐trans‐feruloyltyramine, and Disporopsin were identified through comparison with authentic standards, including N‐feruloyloctopamine (66648‐44‐0, Targetmol), N‐p‐trans‐coumaroyltyramine (36417‐86‐4, Targetmol), N‐trans‐feruloyltyramine (66648‐43‐9, Targetmol), and Disporopsin (1430334‐05‐6, BioBioPha) and confirmed by LC‐MS analysis.

    Techniques: Migration, Expressing